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  • Title: Ciliochoroidal effusion syndrome associated with posterior scleritis.
    Author: Ikeda N, Ikeda T, Nomura C, Mimura O.
    Journal: Jpn J Ophthalmol; 2007; 51(1):49-52. PubMed ID: 17295141.
    Abstract:
    BACKGROUND: To determine the cause of angle-closure glaucoma in a case of posterior scleritis. CASE: The patient was a 65-year-old woman with unilateral acute angle-closure glaucoma who did not respond to laser iridotomy. OBSERVATIONS: Slit-lamp examination demonstrated a shallow anterior chamber in the left eye. Intraocular pressure was 22 mmHg even after application of two antiglaucoma eye-drop preparations. B-scan ultrasonography demonstrated scleral thickening and choroidal detachment in the left eye. Ultrasound biomicroscopy showed a shallow anterior chamber with angle closure, annular ciliochoroidal effusion with ciliary body edema, and an anterior rotation of the ciliary body. After instillation of cycloplegics, the ciliary body and ciliary processes rotated posteriorly, resulting in the release of the pressure on the iris. These changes led to the opening of the angle and subsequent normalization of intraocular pressure. A diagnosis was made of ciliochoroidal effusion syndrome associated with posterior scleritis. CONCLUSIONS: Patients with posterior scleritis can develop ciliochoroidal effusion syndrome, which can lead to angle-closure glaucoma. The therapeutic strategy for acute angle-closure glaucoma induced by ciliochoroidal effusion syndrome differs completely from that for acute angle-closure glaucoma with pupillary block. In the case of ciliochoroidal effusion syndrome, it is important to relieve the compression of the angle by the iris by displacing the lens-iris diaphragm posteriorly by cycloplegics.
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