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  • Title: Membrane lipid environment of carp brain and liver mitochondrial monoamine oxidase.
    Author: Kinemuchi H, Sunami Y, Sudo M, Suh YH, Arai Y, Kamijo K.
    Journal: Comp Biochem Physiol C Comp Pharmacol Toxicol; 1985; 80(2):245-52. PubMed ID: 2861003.
    Abstract:
    Delipidation of carp liver mitochondria by treatment with methyl ethyl ketone (MEK) or Triton X-100 and then perchlorate greatly reduced MAO activities. Treatment with only Triton X-100 resulted in less reduction in activity. The Km values of the remaining activities were similar regardless of these treatments. The sensitivities towards clorgyline and l-deprenyl of the remaining activity in the Triton X-100-treated residue and the phospholipase C-treated carp brain mitochondria were found to be unchanged, but those of the activity remaining in the MEK-treated residue were similarly decreased. No evidence was obtained suggesting conversion of carp MAO to either MAO-A or MAO-B by the modification of the mitochondrial lipid environment by the treatments employed.
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