These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


PUBMED FOR HANDHELDS

Search MEDLINE/PubMed


  • Title: Arterial O2 saturation and maximum O2 consumption in moderate-altitude runners exposed to sea level and 3,050 m.
    Author: Tucker A, Stager JM, Cordain L.
    Journal: JAMA; ; 252(20):2867-71. PubMed ID: 6492368.
    Abstract:
    Twelve trained runners, who were moderate-altitude (1,520 m) residents, were acutely exposed to sea level and 3,050-m altitude in a hypo-hyperbaric chamber. At 1,520 m, maximum O2 consumption (VO2 max) was 67.7 +/- 0.9 mL/kg/min, minute ventilation (V) at maximum exercise was 169.8 +/- 4.6 L/min, and arterial O2 saturation (SaO2) decreased from 93.3% +/- 0.9% at rest, to 84.5% +/- 1.6% at maximum exercise. Exposure to 3,050 m produced a lower VO2max (56.5 +/- 1.5, -16.5%), a similar V at maximum exercise (169.9 +/- 6.0), and a similar fall in SaO2 (from 89.1% +/- 0.8% to 79.5% +/- 0.8%). Exposure to sea level increased VO2max to 72.4 +/- 1.4 (+6.9%), reduced V at maximum exercise to 158.8 +/- 6.5, and induced a smaller fall in SaO2 (from 96.9% +/- 0.4% to 92.1% +/- 1.0%). These changes are comparable with those reported previously in athletes at sea level exposed to similar altitudes, suggesting that residence at 1,520 m does not improve VO2max in highly trained athletes acutely exposed to lower or higher altitudes.
    [Abstract] [Full Text] [Related] [New Search]