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  • Title: [Intravenous glucose tolerance and islet cell function following pancreatic duct occlusion in rats (author's transl)].
    Author: Neidhardt J, Schwille PO, Engelhardt W, Gebhardt C, Scholz D, Gall FP.
    Journal: Chir Forum Exp Klin Forsch; 1980; ():173-7. PubMed ID: 6993123.
    Abstract:
    Microsurgery, intraductal pancreatic occlusion by an Ethibloc bolus and intravenous glucose load in the rat enable investigations to be made into the feed-back relationships between occlusion-induced acinar atrophy and the response of the islet hormones insulin, glucagon, somatostatin, and glucose tolerance. Thirteen days after duct occlusion the latter is moderately delayed from 40-60 min, serum insulin is lowered, plasma somatostatin unchanged, and plasma glucagon moderately elevated, as compared with sham-operated control animals. It is suggested that the employed experimental conditions reveal an exo-endocrine pancreatic functional axis, but that the results obtained are not representative for states with an intact entero-insular axis.
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