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  • Title: Effect of oral contraceptives on vitamin B6 nutriture of young women.
    Author: Vir SC, Love AH.
    Journal: Int J Vitam Nutr Res; 1980; 50(1):29-34. PubMed ID: 7390712.
    Abstract:
    The effect of oral contraceptives (OC) on vitamin B6 status of young women was assessed by percentage stimulation of erythrocyte glutamic pyruvic transaminase (EGPT) activity. The subjects were studied under three groups--control (non OC users), cross-sectional study group (taking OC for over a period of 3 months) and follow up study group. No adverse effect of OC on mean basic EGPT, mean percentage stimulation level or in the incidence of deficiency was noted in cross-sectional study group as compared to controls. Similarly follow up of women before the initial of OC therapy and after a period of 3 months or 6 months usage also demonstrated no adverse effects on vitamin B6 nutriture. The vitamin B6 status showed no relationship with duration of OC use. Dietary vitamin B6 was below the recommended in majority of cases and showed no significant correlation with percentage stimulation of EGPT. 3 groups of young women were studied to determine any effect oral contraceptives (OCs) may have on vitamin B6 status; this effect was assessed by percentage of stimulation of erythrocyte glutamic pyruvic transaminase (EGPT) activity. The 3 groups were: 1) control (non-OC users); 2) a cross-sectional study group who took OCs for 3 months; and 3) a follow-up study group. When the cross-sectional group was compared with controls, no adverse effect of OC use on mean EGPT, mean percent stimulation levels, or mean incidence of deficiency was found. Follow-up of the cross-sectional, using data from before OC use and at 3-6 months after use, similarly demonstrated no adverse effects on vitamin B6 nutrition. Therefore, it is concluded that vitamin B6 status shows no relationship with duration of OC use either. In the majority of cases, dietary vitamin B6 was below recommended levels, but this showed no significant correlation with percent stimulation of EGPT.
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