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444 related items for PubMed ID: 10022341

  • 1. Stage-correlated distribution of type 1 and 2 dystrophic neurites in cortical and hippocampal plaques in Alzheimer's disease.
    Thal DR, Härtig W, Schober R.
    J Hirnforsch; 1998; 39(2):175-81. PubMed ID: 10022341
    [Abstract] [Full Text] [Related]

  • 2. Pathogenesis of Alzheimer-related neuritic plaques: AT8 immunoreactive dystrophic neurites precede argyrophilic neurites in plaques of the entorhinal region, hippocampal formation, and amygdala.
    Yilmazer-Hanke DM.
    Clin Neuropathol; 1998; 17(4):194-8. PubMed ID: 9707333
    [Abstract] [Full Text] [Related]

  • 3. Evidence that transmitter-containing dystrophic neurites precede paired helical filament and Alz-50 formation within senile plaques in the amygdala of nondemented elderly and patients with Alzheimer's disease.
    Benzing WC, Ikonomovic MD, Brady DR, Mufson EJ, Armstrong DM.
    J Comp Neurol; 1993 Aug 08; 334(2):176-91. PubMed ID: 7690048
    [Abstract] [Full Text] [Related]

  • 4. Exclusive association and simultaneous appearance of congophilic plaques and AT8-positive dystrophic neurites in Tg2576 mice suggest a mechanism of senile plaque formation and progression of neuritic dystrophy in Alzheimer's disease.
    Noda-Saita K, Terai K, Iwai A, Tsukamoto M, Shitaka Y, Kawabata S, Okada M, Yamaguchi T.
    Acta Neuropathol; 2004 Nov 08; 108(5):435-42. PubMed ID: 15372280
    [Abstract] [Full Text] [Related]

  • 5. Neuritic pathology and dementia in Alzheimer's disease.
    McKee AC, Kosik KS, Kowall NW.
    Ann Neurol; 1991 Aug 08; 30(2):156-65. PubMed ID: 1910274
    [Abstract] [Full Text] [Related]

  • 6. Amyloid cored plaques in Tg2576 transgenic mice are characterized by giant plaques, slightly activated microglia, and the lack of paired helical filament-typed, dystrophic neurites.
    Sasaki A, Shoji M, Harigaya Y, Kawarabayashi T, Ikeda M, Naito M, Matsubara E, Abe K, Nakazato Y.
    Virchows Arch; 2002 Oct 08; 441(4):358-67. PubMed ID: 12404061
    [Abstract] [Full Text] [Related]

  • 7. Selective PrP-like protein, doppel immunoreactivity in dystrophic neurites of senile plaques in Alzheimer's disease.
    Ferrer I, Freixas M, Blanco R, Carmona M, Puig B.
    Neuropathol Appl Neurobiol; 2004 Aug 08; 30(4):329-37. PubMed ID: 15305978
    [Abstract] [Full Text] [Related]

  • 8. Non-tau based neuronal degeneration in Alzheimer's disease -- an immunocytochemical and quantitative study in the supragranular layers of the middle temporal neocortex.
    van de Nes JA, Nafe R, Schlote W.
    Brain Res; 2008 Jun 05; 1213():152-65. PubMed ID: 18455153
    [Abstract] [Full Text] [Related]

  • 9. Regression stage senile plaques in the natural course of Alzheimer's disease.
    Oide T, Kinoshita T, Arima K.
    Neuropathol Appl Neurobiol; 2006 Oct 05; 32(5):539-56. PubMed ID: 16972888
    [Abstract] [Full Text] [Related]

  • 10. Alzheimer's disease. The density of amygdalar neuritic plaques is associated with the severity of neurofibrillary pathology and the degree of beta-amyloid protein deposition in the cerebral cortex.
    Yilmazer-Hanke DM.
    Acta Anat (Basel); 1998 Oct 05; 162(1):46-55. PubMed ID: 9789108
    [Abstract] [Full Text] [Related]

  • 11. Plaque-associated neuronal proteins: a recurrent motif in neuritic amyloid deposits throughout diverse cortical areas of the Alzheimer's disease brain.
    Schmidt ML, DiDario AG, Otvos L, Hoshi N, Kant JA, Lee VM, Trojanowski JQ.
    Exp Neurol; 1994 Dec 05; 130(2):311-22. PubMed ID: 7867760
    [Abstract] [Full Text] [Related]

  • 12. Tangle-associated neuritic clusters. A new lesion in Alzheimer's disease and aging suggests that aggregates of dystrophic neurites are not necessarily associated with beta/A4.
    Munoz DG, Wang D.
    Am J Pathol; 1992 May 05; 140(5):1167-78. PubMed ID: 1580329
    [Abstract] [Full Text] [Related]

  • 13. Biochemical and anatomical redistribution of tau protein in Alzheimer's disease.
    Mukaetova-Ladinska EB, Harrington CR, Roth M, Wischik CM.
    Am J Pathol; 1993 Aug 05; 143(2):565-78. PubMed ID: 8342603
    [Abstract] [Full Text] [Related]

  • 14. Cytoskeletal alterations differentiate presenilin-1 and sporadic Alzheimer's disease.
    Woodhouse A, Shepherd CE, Sokolova A, Carroll VL, King AE, Halliday GM, Dickson TC, Vickers JC.
    Acta Neuropathol; 2009 Jan 05; 117(1):19-29. PubMed ID: 19015863
    [Abstract] [Full Text] [Related]

  • 15. Neuropathology of mice carrying mutant APP(swe) and/or PS1(M146L) transgenes: alterations in the p75(NTR) cholinergic basal forebrain septohippocampal pathway.
    Jaffar S, Counts SE, Ma SY, Dadko E, Gordon MN, Morgan D, Mufson EJ.
    Exp Neurol; 2001 Aug 05; 170(2):227-43. PubMed ID: 11476589
    [Abstract] [Full Text] [Related]

  • 16. S100 beta protein expression in Alzheimer disease: potential role in the pathogenesis of neuritic plaques.
    Sheng JG, Mrak RE, Griffin WS.
    J Neurosci Res; 1994 Nov 01; 39(4):398-404. PubMed ID: 7884819
    [Abstract] [Full Text] [Related]

  • 17. Phosphorylated tau in neuritic plaques of APP(sw)/Tau (vlw) transgenic mice and Alzheimer disease.
    Pérez M, Morán MA, Ferrer I, Avila J, Gómez-Ramos P.
    Acta Neuropathol; 2008 Oct 01; 116(4):409-18. PubMed ID: 18679696
    [Abstract] [Full Text] [Related]

  • 18. Truncated tau protein as a new marker for Alzheimer's disease.
    Novák M.
    Acta Virol; 1994 Jun 01; 38(3):173-89. PubMed ID: 7817900
    [Abstract] [Full Text] [Related]

  • 19. Neuritic involvement within bFGF immunopositive plaques of Alzheimer's disease.
    Cummings BJ, Su JH, Cotman CW.
    Exp Neurol; 1993 Dec 01; 124(2):315-25. PubMed ID: 8287930
    [Abstract] [Full Text] [Related]

  • 20. The widespread alteration of neurites in Alzheimer's disease may be unrelated to amyloid deposition.
    Tabaton M, Mandybur TI, Perry G, Onorato M, Autilio-Gambetti L, Gambetti P.
    Ann Neurol; 1989 Dec 01; 26(6):771-8. PubMed ID: 2557796
    [Abstract] [Full Text] [Related]

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