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78 related items for PubMed ID: 10036254

  • 1. Role of mitochondrial dysfunction in the Ca2+-induced decline of transmitter release at K+-depolarized motor neuron terminals.
    Calupca MA, Hendricks GM, Hardwick JC, Parsons RL.
    J Neurophysiol; 1999 Feb; 81(2):498-506. PubMed ID: 10036254
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  • 2. Presynaptic function is altered in snake K+-depolarized motor nerve terminals containing compromised mitochondria.
    Calupca MA, Prior C, Merriam LA, Hendricks GM, Parsons RL.
    J Physiol; 2001 Apr 01; 532(Pt 1):217-27. PubMed ID: 11283236
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  • 3. Transmitter release differs at snake twitch and tonic endplates during potassium-induced nerve terminal depolarization.
    Connor EA, Dunaevsky A, Griffiths DJ, Hardwick JC, Parsons RL.
    J Neurophysiol; 1997 Feb 01; 77(2):749-60. PubMed ID: 9065847
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  • 4. Empty synaptic vesicles recycle and undergo exocytosis at vesamicol-treated motor nerve terminals.
    Parsons RL, Calupca MA, Merriam LA, Prior C.
    J Neurophysiol; 1999 Jun 01; 81(6):2696-700. PubMed ID: 10368389
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  • 5. Quantal transmitter release at snake twitch and tonic muscle fibres during prolonged nerve terminal depolarization.
    Coniglio LM, Hardwick JC, Parsons RL.
    J Physiol; 1993 Jul 01; 466():383-403. PubMed ID: 8410698
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  • 6. Inhibition of mitochondrial Ca2+ uptake affects phasic release from motor terminals differently depending on external [Ca2+].
    Talbot JD, David G, Barrett EF.
    J Neurophysiol; 2003 Jul 01; 90(1):491-502. PubMed ID: 12672777
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  • 8. Mitochondrial Ca2+ uptake prevents desynchronization of quantal release and minimizes depletion during repetitive stimulation of mouse motor nerve terminals.
    David G, Barrett EF.
    J Physiol; 2003 Apr 15; 548(Pt 2):425-38. PubMed ID: 12588898
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  • 9. Sodium-dependent increase in quantal secretion induced by brevetoxin-3 in Ca2+-free medium is associated with depletion of synaptic vesicles and swelling of motor nerve terminals in situ.
    Meunier FA, Colasante C, Molgo J.
    Neuroscience; 1997 Jun 15; 78(3):883-93. PubMed ID: 9153666
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  • 14. Presynaptic mitochondria in functionally different motor neurons exhibit similar affinities for Ca2+ but exert little influence as Ca2+ buffers at nerve firing rates in situ.
    Chouhan AK, Zhang J, Zinsmaier KE, Macleod GT.
    J Neurosci; 2010 Feb 03; 30(5):1869-81. PubMed ID: 20130196
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  • 18. Ouabain evokes exocytosis dependent on ryanodine and mitochondrial calcium stores that is not followed by compensatory endocytosis at the neuromuscular junction.
    Amaral E, Leite LF, Gomez MV, Prado MA, Guatimosim C.
    Neurochem Int; 2009 Nov 03; 55(6):406-13. PubMed ID: 19406178
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  • 19. Synchronous and asynchronous exocytosis induced by subthreshold high K+ at Cs(+)-loaded terminals of rat hippocampal neurons.
    Lu FM, Kuba K.
    J Neurophysiol; 2002 Mar 03; 87(3):1222-33. PubMed ID: 11877496
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  • 20. Activation of the protein phosphatase calcineurin during carbachol exposure decreases the extent of recovery from end-plate desensitization.
    Hardwick JC, Parsons RL.
    J Neurophysiol; 1996 Dec 03; 76(6):3609-16. PubMed ID: 8985861
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