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Journal Abstract Search

186 related items for PubMed ID: 10197619

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  • 4. EFA (9-beta-D-erythrofuranosyladenine) is an effective salvage agent for methylthioadenosine phosphorylase-selective therapy of T-cell acute lymphoblastic leukemia with L-alanosine.
    Batova A, Cottam H, Yu J, Diccianni MB, Carrera CJ, Yu AL.
    Blood; 2006 Feb 01; 107(3):898-903. PubMed ID: 16234352
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  • 6. Lack of methylthioadenosine phosphorylase expression in mantle cell lymphoma is associated with shorter survival: implications for a potential targeted therapy.
    Marcé S, Balagué O, Colomo L, Martinez A, Höller S, Villamor N, Bosch F, Ott G, Rosenwald A, Leoni L, Esteller M, Fraga MF, Montserrat E, Colomer D, Campo E.
    Clin Cancer Res; 2006 Jun 15; 12(12):3754-61. PubMed ID: 16778103
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  • 8. Methylthioadenosine phosphorylase deficiency in Japanese osteosarcoma patients.
    Miyazaki S, Nishioka J, Shiraishi T, Matsumine A, Uchida A, Nobori T.
    Int J Oncol; 2007 Nov 15; 31(5):1069-76. PubMed ID: 17912432
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  • 10. Alanosine (UCSD).
    Yu J.
    Curr Opin Investig Drugs; 2001 Nov 15; 2(11):1623-30. PubMed ID: 11763167
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  • 11. Homozygous deletion of CDKN2A and codeletion of the methylthioadenosine phosphorylase gene in the majority of pleural mesotheliomas.
    Illei PB, Rusch VW, Zakowski MF, Ladanyi M.
    Clin Cancer Res; 2003 Jun 15; 9(6):2108-13. PubMed ID: 12796375
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  • 13. Methylthioadenosine phosphorylase gene deletions are common in osteosarcoma.
    García-Castellano JM, Villanueva A, Healey JH, Sowers R, Cordon-Cardo C, Huvos A, Bertino JR, Meyers P, Gorlick R.
    Clin Cancer Res; 2002 Mar 15; 8(3):782-7. PubMed ID: 11895909
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  • 16. Homozygous deletions of methylthioadenosine phosphorylase in human biliary tract cancers.
    Karikari CA, Mullendore M, Eshleman JR, Argani P, Leoni LM, Chattopadhyay S, Hidalgo M, Maitra A.
    Mol Cancer Ther; 2005 Dec 15; 4(12):1860-6. PubMed ID: 16373701
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  • 18. CDKN2A, CDKN2B, and MTAP gene dosage permits precise characterization of mono- and bi-allelic 9p21 deletions in childhood acute lymphoblastic leukemia.
    Bertin R, Acquaviva C, Mirebeau D, Guidal-Giroux C, Vilmer E, Cavé H.
    Genes Chromosomes Cancer; 2003 May 15; 37(1):44-57. PubMed ID: 12661005
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  • 19. Expression of methylthioadenosine phosphorylase cDNA in p16-, MTAP- malignant cells: restoration of methylthioadenosine phosphorylase-dependent salvage pathways and alterations of sensitivity to inhibitors of purine de novo synthesis.
    Chen ZH, Olopade OI, Savarese TM.
    Mol Pharmacol; 1997 Nov 15; 52(5):903-11. PubMed ID: 9351982
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  • 20. Methylthioadenosine phosphorylase gene deletions are frequently detected by fluorescence in situ hybridization in conventional chondrosarcomas.
    Chow WA, Bedell V, Gaytan P, Borden E, Goldblum J, Hicks D, Slovak ML.
    Cancer Genet Cytogenet; 2006 Apr 15; 166(2):95-100. PubMed ID: 16631464
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