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407 related items for PubMed ID: 10320705

  • 1. Time dependence and role of N-methyl-D-aspartate glutamate receptors in the priming of D2-mediated rotational behavior and striatal Fos expression in 6-hydroxydopamine lesioned rats.
    Pollack AE, Strauss JB.
    Brain Res; 1999 May 08; 827(1-2):160-8. PubMed ID: 10320705
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  • 2. Prior D1 dopamine receptor stimulation is required to prime D2-mediated striatal Fos expression in 6-hydroxydopamine-lesioned rats.
    Pollack AE, Yates TM.
    Neuroscience; 1999 May 08; 94(2):505-14. PubMed ID: 10579212
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  • 3. Apomorphine priming alters the response of striatal outflow pathways to D2 agonist stimulation in 6-hydroxydopamine-lesioned rats.
    Pollack AE, Turgeon SM, Fink JS.
    Neuroscience; 1997 Jul 08; 79(1):79-93. PubMed ID: 9178866
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  • 6. D1 priming enhances both D1- and D2-mediated rotational behavior and striatal Fos expression in 6-hydroxydopamine lesioned rats.
    Pollack AE, Thomas LI.
    Pharmacol Biochem Behav; 2010 Jan 08; 94(3):346-51. PubMed ID: 19800912
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  • 8. Role of NMDA glutamate receptors in regulating D2 dopamine-dependent Fos induction in the rat striatopallidal pathway.
    Pollack AE, Bird JL, Lambert EB, Florin ZP, Castellar VL.
    Brain Res; 1999 Feb 13; 818(2):543-7. PubMed ID: 10082844
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  • 11. N-methyl-D-aspartate receptor blockade differentially modifies regional cerebral metabolic responses to D1 and D2 dopamine agonists in rats with a unilateral 6-hydroxydopamine lesion.
    Engber TM, Anderson JJ, Boldry RC, Kuo S, Chase TN.
    Neuroscience; 1993 Jun 13; 54(4):1051-61. PubMed ID: 8101981
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  • 13. Role of a 35 kDa fos-related antigen (FRA) in the long-term induction of striatal dynorphin expression in the 6-hydroxydopamine lesioned rat.
    Bronstein DM, Ye H, Pennypacker KR, Hudson PM, Hong JS.
    Brain Res Mol Brain Res; 1994 May 13; 23(3):191-203. PubMed ID: 7914658
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  • 17. Autoradiographic study of striatal dopamine re-uptake sites and dopamine D1 and D2 receptors in a 6-hydroxydopamine and quinolinic acid double-lesion rat model of striatonigral degeneration (multiple system atrophy) and effects of embryonic ventral mesencephalic, striatal or co-grafts.
    Puschban Z, Scherfler C, Granata R, Laboyrie P, Quinn NP, Jenner P, Poewe W, Wenning GK.
    Neuroscience; 2000 May 13; 95(2):377-88. PubMed ID: 10658617
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  • 18. Abnormal involuntary movement (AIM) expression following D2 dopamine agonist challenge is determined by the nature of prior dopamine receptor stimulation (priming) in 6-hydroxydopamine lesioned rats.
    Drake JD, Kibuuka LN, Dimitrov KD, Pollack AE.
    Pharmacol Biochem Behav; 2013 Apr 13; 105():26-33. PubMed ID: 23369985
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  • 19. A within-subjects microdialysis/behavioural study of the role of striatal acetylcholine in D1-dependent turning.
    Acquas E, Fenu S, Loddo P, Di Chiara G.
    Behav Brain Res; 1999 Sep 13; 103(2):219-28. PubMed ID: 10513590
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  • 20. EEG modifications in the cortex and striatum after dopaminergic priming in the 6-hydroxydopamine rat model of Parkinson's disease.
    Vorobyov VV, Schibaev NV, Morelli M, Carta AR.
    Brain Res; 2003 May 16; 972(1-2):177-85. PubMed ID: 12711091
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