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649 related items for PubMed ID: 10338314

  • 1. Lack of involvement of neuronal nitric oxide synthase in the pathogenesis of a transgenic mouse model of familial amyotrophic lateral sclerosis.
    Facchinetti F, Sasaki M, Cutting FB, Zhai P, MacDonald JE, Reif D, Beal MF, Huang PL, Dawson TM, Gurney ME, Dawson VL.
    Neuroscience; 1999; 90(4):1483-92. PubMed ID: 10338314
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  • 3. Neuronal nitric oxide synthase inhibitor, 7-nitroindazole, delays motor dysfunction and spinal motoneuron degeneration in the wobbler mouse.
    Ikeda K, Iwasaki Y, Kinoshita M.
    J Neurol Sci; 1998 Sep 18; 160(1):9-15. PubMed ID: 9804111
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  • 5. Cu/Zn superoxide dismutase (SOD1) mutations associated with familial amyotrophic lateral sclerosis (ALS) affect cellular free radical release in the presence of oxidative stress.
    Cookson MR, Menzies FM, Manning P, Eggett CJ, Figlewicz DA, McNeil CJ, Shaw PJ.
    Amyotroph Lateral Scler Other Motor Neuron Disord; 2002 Jun 18; 3(2):75-85. PubMed ID: 12215229
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  • 7. Absence of p53: no effect in a transgenic mouse model of familial amyotrophic lateral sclerosis.
    Kuntz C, Kinoshita Y, Beal MF, Donehower LA, Morrison RS.
    Exp Neurol; 2000 Sep 18; 165(1):184-90. PubMed ID: 10964497
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  • 8. S-nitrosylated protein disulfide isomerase contributes to mutant SOD1 aggregates in amyotrophic lateral sclerosis.
    Chen X, Zhang X, Li C, Guan T, Shang H, Cui L, Li XM, Kong J.
    J Neurochem; 2013 Jan 18; 124(1):45-58. PubMed ID: 23043510
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  • 20. Alteration in intracellular calcium homeostasis reduces motor neuronal viability expressing mutated Cu/Zn superoxide dismutase through a nitric oxide/guanylyl cyclase cGMP cascade.
    Kim HJ, Kim M, Kim SH, Sung JJ, Lee KW.
    Neuroreport; 2002 Jul 02; 13(9):1131-5. PubMed ID: 12151755
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