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539 related items for PubMed ID: 10360967

  • 1. Death signals from the B cell antigen receptor target mitochondria, activating necrotic and apoptotic death cascades in a murine B cell line, WEHI-231.
    Doi T, Motoyama N, Tokunaga A, Watanabe T.
    Int Immunol; 1999 Jun; 11(6):933-41. PubMed ID: 10360967
    [Abstract] [Full Text] [Related]

  • 2. B cell receptor-stimulated mitochondrial phospholipase A2 activation and resultant disruption of mitochondrial membrane potential correlate with the induction of apoptosis in WEHI-231 B cells.
    Katz E, Deehan MR, Seatter S, Lord C, Sturrock RD, Harnett MM.
    J Immunol; 2001 Jan 01; 166(1):137-47. PubMed ID: 11123286
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  • 3. The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death.
    Hirsch T, Marchetti P, Susin SA, Dallaporta B, Zamzami N, Marzo I, Geuskens M, Kroemer G.
    Oncogene; 1997 Sep 25; 15(13):1573-81. PubMed ID: 9380409
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  • 4. Mitochondria-dependent caspase-9 activation is necessary for antigen receptor-mediated effector caspase activation and apoptosis in WEHI 231 lymphoma cells.
    Herold MJ, Kuss AW, Kraus C, Berberich I.
    J Immunol; 2002 Apr 15; 168(8):3902-9. PubMed ID: 11937545
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  • 5. Antigen receptor-induced B lymphocyte apoptosis mediated via a protease of the caspase family.
    Andjelic S, Liou HC.
    Eur J Immunol; 1998 Feb 15; 28(2):570-81. PubMed ID: 9521067
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  • 6. Caspase-independent cell death induced by anti-CD2 or staurosporine in activated human peripheral T lymphocytes.
    Déas O, Dumont C, MacFarlane M, Rouleau M, Hebib C, Harper F, Hirsch F, Charpentier B, Cohen GM, Senik A.
    J Immunol; 1998 Oct 01; 161(7):3375-83. PubMed ID: 9759854
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  • 7. LPS-induced apoptosis is dependent upon mitochondrial dysfunction.
    Kuwabara T, Imajoh-Ohmi S.
    Apoptosis; 2004 Jul 01; 9(4):467-74. PubMed ID: 15192329
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  • 8. Ligation of CD40 rescues Ramos-Burkitt lymphoma B cells from calcium ionophore- and antigen receptor-triggered apoptosis by inhibiting activation of the cysteine protease CPP32/Yama and cleavage of its substrate PARP.
    An S, Knox KA.
    FEBS Lett; 1996 May 20; 386(2-3):115-22. PubMed ID: 8647264
    [Abstract] [Full Text] [Related]

  • 9. Doxorubicin treatment activates a Z-VAD-sensitive caspase, which causes deltapsim loss, caspase-9 activity, and apoptosis in Jurkat cells.
    Gamen S, Anel A, Pérez-Galán P, Lasierra P, Johnson D, Piñeiro A, Naval J.
    Exp Cell Res; 2000 Jul 10; 258(1):223-35. PubMed ID: 10912804
    [Abstract] [Full Text] [Related]

  • 10. Feedback regulation of mitochondria by caspase-9 in the B cell receptor-mediated apoptosis.
    Eeva J, Nuutinen U, Ropponen A, Mättö M, Eray M, Pellinen R, Wahlfors J, Pelkonen J.
    Scand J Immunol; 2009 Dec 10; 70(6):574-83. PubMed ID: 19906200
    [Abstract] [Full Text] [Related]

  • 11. Effector caspases are dispensable for the early nuclear morphological changes during chemical-induced apoptosis.
    Johnson VL, Ko SC, Holmstrom TH, Eriksson JE, Chow SC.
    J Cell Sci; 2000 Sep 10; 113 ( Pt 17)():2941-53. PubMed ID: 10934034
    [Abstract] [Full Text] [Related]

  • 12. LIGHT sensitizes IFN-gamma-mediated apoptosis of HT-29 human carcinoma cells through both death receptor and mitochondria pathways.
    Zhang MC, Liu HP, Demchik LL, Zhai YF, Yang DJ.
    Cell Res; 2004 Apr 10; 14(2):117-24. PubMed ID: 15115612
    [Abstract] [Full Text] [Related]

  • 13. Bcl-xL inhibits cytochrome c release but not mitochondrial depolarization during the activation of multiple death pathways by tumor necrosis factor-alpha.
    Johnson BW, Cepero E, Boise LH.
    J Biol Chem; 2000 Oct 06; 275(40):31546-53. PubMed ID: 10913120
    [Abstract] [Full Text] [Related]

  • 14. Spermine oxidation leads to necrosis with plasma membrane phosphatidylserine redistribution in mouse leukemia cells.
    Bonneau MJ, Poulin R.
    Exp Cell Res; 2000 Aug 25; 259(1):23-34. PubMed ID: 10942576
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  • 18. Apoptosis, in human monocytic THP.1 cells, results in the release of cytochrome c from mitochondria prior to their ultracondensation, formation of outer membrane discontinuities and reduction in inner membrane potential.
    Zhuang J, Dinsdale D, Cohen GM.
    Cell Death Differ; 1998 Nov 25; 5(11):953-62. PubMed ID: 9846182
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