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454 related items for PubMed ID: 10402466

  • 1. DAP-kinase participates in TNF-alpha- and Fas-induced apoptosis and its function requires the death domain.
    Cohen O, Inbal B, Kissil JL, Raveh T, Berissi H, Spivak-Kroizaman T, Feinstein E, Kimchi A.
    J Cell Biol; 1999 Jul 12; 146(1):141-8. PubMed ID: 10402466
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  • 2. Ultraviolet-irradiation-induced apoptosis is mediated via ligand independent activation of tumor necrosis factor receptor 1.
    Sheikh MS, Antinore MJ, Huang Y, Fornace AJ.
    Oncogene; 1998 Nov 19; 17(20):2555-63. PubMed ID: 9840918
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  • 3. Rapid B cell apoptosis induced by antigen receptor ligation does not require Fas (CD95/APO-1), the adaptor protein FADD/MORT1 or CrmA-sensitive caspases but is defective in both MRL-+/+ and MRL-lpr/lpr mice.
    Yoshida T, Higuchi T, Hagiyama H, Strasser A, Nishioka K, Tsubata T.
    Int Immunol; 2000 Apr 19; 12(4):517-26. PubMed ID: 10744653
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  • 4. Inhibition of death receptor-mediated gene induction by a cycloheximide-sensitive factor occurs at the level of or upstream of Fas-associated death domain protein (FADD).
    Wajant H, Haas E, Schwenzer R, Muhlenbeck F, Kreuz S, Schubert G, Grell M, Smith C, Scheurich P.
    J Biol Chem; 2000 Aug 11; 275(32):24357-66. PubMed ID: 10823821
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  • 5. Intracellular mechanisms of TRAIL: apoptosis through mitochondrial-dependent and -independent pathways.
    Suliman A, Lam A, Datta R, Srivastava RK.
    Oncogene; 2001 Apr 19; 20(17):2122-33. PubMed ID: 11360196
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  • 6. A functional genetic screen identifies regions at the C-terminal tail and death-domain of death-associated protein kinase that are critical for its proapoptotic activity.
    Raveh T, Berissi H, Eisenstein M, Spivak T, Kimchi A.
    Proc Natl Acad Sci U S A; 2000 Feb 15; 97(4):1572-7. PubMed ID: 10677501
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  • 7. Apoptosis induced by a chimeric Fas/FLICE receptor: lack of requirement for Fas- or FADD-binding proteins.
    Memon SA, Hou J, Moreno MB, Zacharchuk CM.
    J Immunol; 1998 Mar 01; 160(5):2046-9. PubMed ID: 9498739
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  • 8. FADD/MORT1 and caspase-8 are recruited to TRAIL receptors 1 and 2 and are essential for apoptosis mediated by TRAIL receptor 2.
    Sprick MR, Weigand MA, Rieser E, Rauch CT, Juo P, Blenis J, Krammer PH, Walczak H.
    Immunity; 2000 Jun 01; 12(6):599-609. PubMed ID: 10894160
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  • 15. Sensitization to death receptor cytotoxicity by inhibition of fas-associated death domain protein (FADD)/caspase signaling. Requirement of cell cycle progression.
    Lüschen S, Ussat S, Scherer G, Kabelitz D, Adam-Klages S.
    J Biol Chem; 2000 Aug 11; 275(32):24670-8. PubMed ID: 10827087
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  • 16. Fas- and tumor necrosis factor-mediated apoptosis uses the same binding surface of FADD to trigger signal transduction. A typical model for convergent signal transduction.
    Bang S, Jeong EJ, Kim IK, Jung YK, Kim KS.
    J Biol Chem; 2000 Nov 17; 275(46):36217-22. PubMed ID: 10952991
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  • 17. Expression of death-associated protein kinase and recruitment to the tumor necrosis factor signaling pathway following brief seizures.
    Henshall DC, Araki T, Schindler CK, Shinoda S, Lan JQ, Simon RP.
    J Neurochem; 2003 Sep 17; 86(5):1260-70. PubMed ID: 12911633
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  • 18. Casper is a FADD- and caspase-related inducer of apoptosis.
    Shu HB, Halpin DR, Goeddel DV.
    Immunity; 1997 Jun 17; 6(6):751-63. PubMed ID: 9208847
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  • 19. Casein kinase I attenuates tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis by regulating the recruitment of fas-associated death domain and procaspase-8 to the death-inducing signaling complex.
    Izeradjene K, Douglas L, Delaney AB, Houghton JA.
    Cancer Res; 2004 Nov 01; 64(21):8036-44. PubMed ID: 15520213
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  • 20. Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis.
    Lin Y, Devin A, Rodriguez Y, Liu ZG.
    Genes Dev; 1999 Oct 01; 13(19):2514-26. PubMed ID: 10521396
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