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Journal Abstract Search

180 related items for PubMed ID: 10422292

  • 1. [Detection of t(3 ; 21) (q26 ; q22) with AML1/EVI1 mRNA during progression of myelodysplastic syndrome to acute myeloid leukemia].
    Choi I, Goto T, Nagano M, Muta K, Yufu Y, Uike N, Kozuru M, Abe Y, Nisimura J.
    Rinsho Ketsueki; 1999 Jun; 40(6):518-20. PubMed ID: 10422292
    [Abstract] [Full Text] [Related]

  • 2. t(3;21)(q26;q22) in myeloid leukemia: an aggressive syndrome of blast transformation associated with hydroxyurea or antimetabolite therapy.
    Yin CC, Cortes J, Barkoh B, Hayes K, Kantarjian H, Jones D.
    Cancer; 2006 Apr 15; 106(8):1730-8. PubMed ID: 16532439
    [Abstract] [Full Text] [Related]

  • 3. Acute promyelocytic leukemia relapsing as secondary acute myelogenous leukemia with translocation t(3;21)(q26;q22) and RUNX1-MDS1-EVI1 fusion transcript.
    Park TS, Choi JR, Yoon SH, Song J, Kim J, Kim SJ, Kwon O, Min YH.
    Cancer Genet Cytogenet; 2008 Dec 15; 187(2):61-73. PubMed ID: 19027486
    [Abstract] [Full Text] [Related]

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  • 5. Consistent intergenic splicing and production of multiple transcripts between AML1 at 21q22 and unrelated genes at 3q26 in (3;21)(q26;q22) translocations.
    Nucifora G, Begy CR, Kobayashi H, Roulston D, Claxton D, Pedersen-Bjergaard J, Parganas E, Ihle JN, Rowley JD.
    Proc Natl Acad Sci U S A; 1994 Apr 26; 91(9):4004-8. PubMed ID: 8171026
    [Abstract] [Full Text] [Related]

  • 6. [Study of genes involved in chronic myeloid leukemia with t (3; 21) (q26; q22) in blastic crisis].
    Liu XP, Zhang MR, Dai Y, Zhang L, Li R, Hao YS, Xiao ZJ.
    Zhonghua Xue Ye Xue Za Zhi; 2006 May 26; 27(5):310-3. PubMed ID: 16875579
    [Abstract] [Full Text] [Related]

  • 7. Rearrangements of the AML1/CBFA2 gene in myeloid leukemia with the 3;21 translocation: in vitro and in vivo studies.
    Zent C, Rowley JD, Nucifora G.
    Leukemia; 1997 Apr 26; 11 Suppl 3():273-8. PubMed ID: 9209363
    [Abstract] [Full Text] [Related]

  • 8. The chimeric genes AML1/MDS1 and AML1/EAP inhibit AML1B activation at the CSF1R promoter, but only AML1/MDS1 has tumor-promoter properties.
    Zent CS, Mathieu C, Claxton DF, Zhang DE, Tenen DG, Rowley JD, Nucifora G.
    Proc Natl Acad Sci U S A; 1996 Feb 06; 93(3):1044-8. PubMed ID: 8577711
    [Abstract] [Full Text] [Related]

  • 9. Both AML1 and EVI1 oncogenic components are required for the cooperation of AML1/MDS1/EVI1 with BCR/ABL in the induction of acute myelogenous leukemia in mice.
    Cuenco GM, Ren R.
    Oncogene; 2004 Jan 15; 23(2):569-79. PubMed ID: 14724585
    [Abstract] [Full Text] [Related]

  • 10. PRDX4, a member of the peroxiredoxin family, is fused to AML1 (RUNX1) in an acute myeloid leukemia patient with a t(X;21)(p22;q22).
    Zhang Y, Emmanuel N, Kamboj G, Chen J, Shurafa M, Van Dyke DL, Wiktor A, Rowley JD.
    Genes Chromosomes Cancer; 2004 Aug 15; 40(4):365-70. PubMed ID: 15188461
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  • 11. Cooperation of BCR-ABL and AML1/MDS1/EVI1 in blocking myeloid differentiation and rapid induction of an acute myelogenous leukemia.
    Cuenco GM, Ren R.
    Oncogene; 2001 Dec 13; 20(57):8236-48. PubMed ID: 11781838
    [Abstract] [Full Text] [Related]

  • 12. A novel chromosomal translocation t(3;7)(q26;q21) in myeloid leukemia resulting in overexpression of EVI1.
    Storlazzi CT, Anelli L, Albano F, Zagaria A, Ventura M, Rocchi M, Panagopoulos I, Pannunzio A, Ottaviani E, Liso V, Specchia G.
    Ann Hematol; 2004 Feb 13; 83(2):78-83. PubMed ID: 14551738
    [Abstract] [Full Text] [Related]

  • 13. The leukemia-associated transcription repressor AML1/MDS1/EVI1 requires CtBP to induce abnormal growth and differentiation of murine hematopoietic cells.
    Senyuk V, Chakraborty S, Mikhail FM, Zhao R, Chi Y, Nucifora G.
    Oncogene; 2002 May 09; 21(20):3232-40. PubMed ID: 12082639
    [Abstract] [Full Text] [Related]

  • 14. Fusion of ETV6 to MDS1/EVI1 as a result of t(3;12)(q26;p13) in myeloproliferative disorders.
    Peeters P, Wlodarska I, Baens M, Criel A, Selleslag D, Hagemeijer A, Van den Berghe H, Marynen P.
    Cancer Res; 1997 Feb 15; 57(4):564-9. PubMed ID: 9044825
    [Abstract] [Full Text] [Related]

  • 15. The AML1/Evi-1 fusion protein in the t(3;21) translocation exhibits transforming activity on Rat1 fibroblasts with dependence on the Evi-1 sequence.
    Kurokawa M, Ogawa S, Tanaka T, Mitani K, Yazaki Y, Witte ON, Hirai H.
    Oncogene; 1995 Sep 07; 11(5):833-40. PubMed ID: 7675444
    [Abstract] [Full Text] [Related]

  • 16. [Acute myelocytic leukemia with ins(21;8)(q22;q13q22) presenting with AML1/MTG8 chimeric mRNA].
    Yamazaki E, Ogawa K, Harano H, Kanamori H, Ishigatsubo Y.
    Rinsho Ketsueki; 2000 Sep 07; 41(9):745-9. PubMed ID: 11070937
    [Abstract] [Full Text] [Related]

  • 17. Molecular mechanism of blastic crisis in chronic myelocytic leukemia.
    Mitani K.
    Leukemia; 1997 Apr 07; 11 Suppl 3():503-5. PubMed ID: 9209439
    [Abstract] [Full Text] [Related]

  • 18. Rearrangement of the AML1/CBFA2 gene in myeloid leukemia with the 3;21 translocation: expression of co-existing multiple chimeric genes with similar functions as transcriptional repressors, but with opposite tumorigenic properties.
    Zent C, Kim N, Hiebert S, Zhang DE, Tenen DG, Rowley JD, Nucifora G.
    Curr Top Microbiol Immunol; 1996 Apr 07; 211():243-52. PubMed ID: 8585955
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