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Journal Abstract Search

167 related items for PubMed ID: 10445668

  • 1. Okadaic acid and anisomycin are protective and stimulate the SAPK/JNK pathway.
    Barancik M, Htun P, Schaper W.
    J Cardiovasc Pharmacol; 1999 Aug; 34(2):182-90. PubMed ID: 10445668
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  • 5. Inhibition of p38 MAP kinase increases okadaic acid mediated AP-1 expression and DNA binding but has no effect on TRE dependent transcription.
    Rosenberger SF, Gupta A, Bowden GT.
    Oncogene; 1999 Jun 17; 18(24):3626-32. PubMed ID: 10380884
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  • 6. Stimulation of multiple mitogen-activated protein kinase sub-families by oxidative stress and phosphorylation of the small heat shock protein, HSP25/27, in neonatal ventricular myocytes.
    Clerk A, Michael A, Sugden PH.
    Biochem J; 1998 Aug 01; 333 ( Pt 3)(Pt 3):581-9. PubMed ID: 9677316
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  • 7. Inhibition of the cardiac p38-MAPK pathway by SB203580 delays ischemic cell death.
    Barancik M, Htun P, Strohm C, Kilian S, Schaper W.
    J Cardiovasc Pharmacol; 2000 Mar 01; 35(3):474-83. PubMed ID: 10710135
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  • 8. Neither ERK nor JNK/SAPK MAP kinase subtypes are essential for histone H3/HMG-14 phosphorylation or c-fos and c-jun induction.
    Cano E, Hazzalin CA, Kardalinou E, Buckle RS, Mahadevan LC.
    J Cell Sci; 1995 Nov 01; 108 ( Pt 11)():3599-609. PubMed ID: 8586671
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  • 9. Early modifications in the expression of mitogen-activated protein kinase (MAPK/ERK), stress-activated kinases SAPK/JNK and p38, and their phosphorylated substrates following focal cerebral ischemia.
    Ferrer I, Friguls B, Dalfó E, Planas AM.
    Acta Neuropathol; 2003 May 01; 105(5):425-37. PubMed ID: 12677442
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  • 15. Ribotoxic stress response: activation of the stress-activated protein kinase JNK1 by inhibitors of the peptidyl transferase reaction and by sequence-specific RNA damage to the alpha-sarcin/ricin loop in the 28S rRNA.
    Iordanov MS, Pribnow D, Magun JL, Dinh TH, Pearson JA, Chen SL, Magun BE.
    Mol Cell Biol; 1997 Jun 01; 17(6):3373-81. PubMed ID: 9154836
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  • 16. Distinct regulation of osmoprotective genes in yeast and mammals. Aldose reductase osmotic response element is induced independent of p38 and stress-activated protein kinase/Jun N-terminal kinase in rabbit kidney cells.
    Kültz D, Garcia-Perez A, Ferraris JD, Burg MB.
    J Biol Chem; 1997 May 16; 272(20):13165-70. PubMed ID: 9148932
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  • 17. MST/MLK2, a member of the mixed lineage kinase family, directly phosphorylates and activates SEK1, an activator of c-Jun N-terminal kinase/stress-activated protein kinase.
    Hirai Si, Katoh M, Terada M, Kyriakis JM, Zon LI, Rana A, Avruch J, Ohno S.
    J Biol Chem; 1997 Jun 13; 272(24):15167-73. PubMed ID: 9182538
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  • 18. Activation of mitogen-activated protein kinases (p38-MAPKs, SAPKs/JNKs and ERKs) by the G-protein-coupled receptor agonist phenylephrine in the perfused rat heart.
    Lazou A, Sugden PH, Clerk A.
    Biochem J; 1998 Jun 01; 332 ( Pt 2)(Pt 2):459-65. PubMed ID: 9601075
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  • 19. Activation of c-Jun N-terminal kinases and p38-mitogen-activated protein kinases in human heart failure secondary to ischaemic heart disease.
    Cook SA, Sugden PH, Clerk A.
    J Mol Cell Cardiol; 1999 Aug 01; 31(8):1429-34. PubMed ID: 10423341
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  • 20. Stimulation of the stress-activated mitogen-activated protein kinase subfamilies in perfused heart. p38/RK mitogen-activated protein kinases and c-Jun N-terminal kinases are activated by ischemia/reperfusion.
    Bogoyevitch MA, Gillespie-Brown J, Ketterman AJ, Fuller SJ, Ben-Levy R, Ashworth A, Marshall CJ, Sugden PH.
    Circ Res; 1996 Aug 01; 79(2):162-73. PubMed ID: 8755992
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