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179 related items for PubMed ID: 10455158

  • 1. Extracellular signal-regulated protein kinase activation is required for the anti-hypertrophic effect of atrial natriuretic factor in neonatal rat ventricular myocytes.
    Silberbach M, Gorenc T, Hershberger RE, Stork PJ, Steyger PS, Roberts CT.
    J Biol Chem; 1999 Aug 27; 274(35):24858-64. PubMed ID: 10455158
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  • 2. Specific role of the extracellular signal-regulated kinase pathway in angiotensin II-induced cardiac hypertrophy in vitro.
    Aoki H, Richmond M, Izumo S, Sadoshima J.
    Biochem J; 2000 Apr 01; 347 Pt 1(Pt 1):275-84. PubMed ID: 10727428
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  • 3. Dissociation of p44 and p42 mitogen-activated protein kinase activation from receptor-induced hypertrophy in neonatal rat ventricular myocytes.
    Post GR, Goldstein D, Thuerauf DJ, Glembotski CC, Brown JH.
    J Biol Chem; 1996 Apr 05; 271(14):8452-7. PubMed ID: 8626545
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  • 4. Opposing effects of Jun kinase and p38 mitogen-activated protein kinases on cardiomyocyte hypertrophy.
    Nemoto S, Sheng Z, Lin A.
    Mol Cell Biol; 1998 Jun 05; 18(6):3518-26. PubMed ID: 9584192
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  • 7. c-Jun N-terminal kinase-interacting protein 1 inhibits gene expression in response to hypertrophic agonists in neonatal rat ventricular myocytes.
    Finn SG, Dickens M, Fuller SJ.
    Biochem J; 2001 Sep 01; 358(Pt 2):489-95. PubMed ID: 11513749
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  • 8. The mitogen-activated protein kinase kinase MEK1 stimulates a pattern of gene expression typical of the hypertrophic phenotype in rat ventricular cardiomyocytes.
    Gillespie-Brown J, Fuller SJ, Bogoyevitch MA, Cowley S, Sugden PH.
    J Biol Chem; 1995 Nov 24; 270(47):28092-6. PubMed ID: 7499296
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  • 10. Raf-1 kinase activity is necessary and sufficient for gene expression changes but not sufficient for cellular morphology changes associated with cardiac myocyte hypertrophy.
    Thorburn J, McMahon M, Thorburn A.
    J Biol Chem; 1994 Dec 02; 269(48):30580-6. PubMed ID: 7982977
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  • 14. Extracellular ATP inhibits adrenergic agonist-induced hypertrophy of neonatal cardiac myocytes.
    Zheng JS, Boluyt MO, Long X, O'Neill L, Lakatta EG, Crow MT.
    Circ Res; 1996 Apr 02; 78(4):525-35. PubMed ID: 8635209
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  • 15. Ras and rho are required for galphaq-induced hypertrophic gene expression in neonatal rat cardiac myocytes.
    Hines WA, Thorburn A.
    J Mol Cell Cardiol; 1998 Mar 02; 30(3):485-94. PubMed ID: 9515026
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  • 16. Activation of mitogen-activated protein kinases (p38-MAPKs, SAPKs/JNKs and ERKs) by the G-protein-coupled receptor agonist phenylephrine in the perfused rat heart.
    Lazou A, Sugden PH, Clerk A.
    Biochem J; 1998 Jun 01; 332 ( Pt 2)(Pt 2):459-65. PubMed ID: 9601075
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  • 17. Cross-talk between the ERK and p70 S6 kinase (S6K) signaling pathways. MEK-dependent activation of S6K2 in cardiomyocytes.
    Wang L, Gout I, Proud CG.
    J Biol Chem; 2001 Aug 31; 276(35):32670-7. PubMed ID: 11431469
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  • 18. Stimulation of the p38 mitogen-activated protein kinase pathway in neonatal rat ventricular myocytes by the G protein-coupled receptor agonists, endothelin-1 and phenylephrine: a role in cardiac myocyte hypertrophy?
    Clerk A, Michael A, Sugden PH.
    J Cell Biol; 1998 Jul 27; 142(2):523-35. PubMed ID: 9679149
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  • 19. Activation of protein synthesis in cardiomyocytes by the hypertrophic agent phenylephrine requires the activation of ERK and involves phosphorylation of tuberous sclerosis complex 2 (TSC2).
    Rolfe M, McLeod LE, Pratt PF, Proud CG.
    Biochem J; 2005 Jun 15; 388(Pt 3):973-84. PubMed ID: 15757502
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  • 20. Requirement of activation of the extracellular signal-regulated kinase cascade in myocardial cell hypertrophy.
    Ueyama T, Kawashima S, Sakoda T, Rikitake Y, Ishida T, Kawai M, Yamashita T, Ishido S, Hotta H, Yokoyama M.
    J Mol Cell Cardiol; 2000 Jun 15; 32(6):947-60. PubMed ID: 10888249
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