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101 related items for PubMed ID: 10486252

  • 1. Cyclic AMP mediates EDHF-type relaxations of rabbit jugular vein.
    Griffith TM, Taylor HJ.
    Biochem Biophys Res Commun; 1999 Sep 16; 263(1):52-7. PubMed ID: 10486252
    [Abstract] [Full Text] [Related]

  • 2. Gap junction-dependent increases in smooth muscle cAMP underpin the EDHF phenomenon in rabbit arteries.
    Taylor HJ, Chaytor AT, Edwards DH, Griffith TM.
    Biochem Biophys Res Commun; 2001 May 11; 283(3):583-9. PubMed ID: 11341764
    [Abstract] [Full Text] [Related]

  • 3. Comparison of glycyrrhetinic acid isoforms and carbenoxolone as inhibitors of EDHF-type relaxations mediated via gap junctions.
    Chaytor AT, Marsh WL, Hutcheson IR, Griffith TM.
    Endothelium; 2000 May 11; 7(4):265-78. PubMed ID: 11201524
    [Abstract] [Full Text] [Related]

  • 4. Inhibition of the gap junctional component of endothelium-dependent relaxations in rabbit iliac artery by 18-alpha glycyrrhetinic acid.
    Taylor HJ, Chaytor AT, Evans WH, Griffith TM.
    Br J Pharmacol; 1998 Sep 11; 125(1):1-3. PubMed ID: 9776336
    [Abstract] [Full Text] [Related]

  • 5. Nitric oxide-independent relaxations to acetylcholine and A23187 involve different routes of heterocellular communication. Role of Gap junctions and phospholipase A2.
    Hutcheson IR, Chaytor AT, Evans WH, Griffith TM.
    Circ Res; 1998 Sep 11; 84(1):53-63. PubMed ID: 9915774
    [Abstract] [Full Text] [Related]

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  • 7. Gap junction-dependent and -independent EDHF-type relaxations may involve smooth muscle cAMP accumulation.
    Chaytor AT, Taylor HJ, Griffith TM.
    Am J Physiol Heart Circ Physiol; 2002 Apr 11; 282(4):H1548-55. PubMed ID: 11893592
    [Abstract] [Full Text] [Related]

  • 8. Replacement of connexin 43 by connexin 32 in a knock-in mice model attenuates aortic endothelium-derived hyperpolarizing factor-mediated relaxation.
    López D, Rodríguez-Sinovas A, Agulló E, García A, Sánchez JA, García-Dorado D.
    Exp Physiol; 2009 Oct 11; 94(10):1088-97. PubMed ID: 19617266
    [Abstract] [Full Text] [Related]

  • 9. Mechanisms underlying the impaired EDHF-type relaxation response in mesenteric arteries from Otsuka Long-Evans Tokushima Fatty (OLETF) rats.
    Matsumoto T, Kobayashi T, Kamata K.
    Eur J Pharmacol; 2006 May 24; 538(1-3):132-40. PubMed ID: 16678154
    [Abstract] [Full Text] [Related]

  • 10. Role of voltage-dependent potassium channels and myo-endothelial gap junctions in 4-aminopyridine-induced inhibition of acetylcholine relaxation in rat carotid artery.
    Gupta PK, Subramani J, Leo MD, Sikarwar AS, Parida S, Prakash VR, Mishra SK.
    Eur J Pharmacol; 2008 Sep 04; 591(1-3):171-6. PubMed ID: 18577383
    [Abstract] [Full Text] [Related]

  • 11. Endothelium-derived relaxing factor-mediated vasodilation in mouse mesenteric vascular beds.
    Fujiwara H, Wake Y, Hashikawa-Hobara N, Makino K, Takatori S, Zamami Y, Kitamura Y, Kawasaki H.
    J Pharmacol Sci; 2012 Sep 04; 118(3):373-81. PubMed ID: 22450195
    [Abstract] [Full Text] [Related]

  • 12. Augmented endothelium-derived hyperpolarizing factor-mediated relaxations attenuate endothelial dysfunction in femoral and mesenteric, but not in carotid arteries from type I diabetic rats.
    Shi Y, Ku DD, Man RY, Vanhoutte PM.
    J Pharmacol Exp Ther; 2006 Jul 04; 318(1):276-81. PubMed ID: 16565165
    [Abstract] [Full Text] [Related]

  • 13. Mediation of EDHF-induced reduction of smooth muscle [Ca(2+)](i) and arteriolar dilation by K(+) channels, 5,6-EET, and gap junctions.
    Ungvari Z, Koller A.
    Microcirculation; 2001 Aug 04; 8(4):265-74. PubMed ID: 11528534
    [Abstract] [Full Text] [Related]

  • 14. Both endothelium and afferent nerve endings play a role in acetylcholine-induced renal vasodilation.
    Ay I, Tuncer M.
    Life Sci; 2006 Jul 24; 79(9):877-82. PubMed ID: 16616212
    [Abstract] [Full Text] [Related]

  • 15. Endothelial potassium channels, endothelium-dependent hyperpolarization and the regulation of vascular tone in health and disease.
    Coleman HA, Tare M, Parkington HC.
    Clin Exp Pharmacol Physiol; 2004 Sep 24; 31(9):641-9. PubMed ID: 15479173
    [Abstract] [Full Text] [Related]

  • 16. Inhibition of acetylcholine-induced EDHF response by elevated glucose in rat mesenteric artery.
    Ozkan MH, Uma S.
    Life Sci; 2005 Nov 19; 78(1):14-21. PubMed ID: 16125203
    [Abstract] [Full Text] [Related]

  • 17. Critical role of gap junctions in endothelium-dependent hyperpolarization in rat mesenteric arteries.
    Goto K, Fujii K, Kansui Y, Abe I, Iida M.
    Clin Exp Pharmacol Physiol; 2002 Jul 19; 29(7):595-602. PubMed ID: 12060103
    [Abstract] [Full Text] [Related]

  • 18. The endothelial component of cannabinoid-induced relaxation in rabbit mesenteric artery depends on gap junctional communication.
    Chaytor AT, Martin PE, Evans WH, Randall MD, Griffith TM.
    J Physiol; 1999 Oct 15; 520 Pt 2(Pt 2):539-50. PubMed ID: 10523421
    [Abstract] [Full Text] [Related]

  • 19. Characterization of nitric oxide- and prostaglandin-independent relaxation in response to acetylcholine in rabbit renal artery.
    Kagota S, Yamaguchi Y, Nakamura K, Kunitomo M.
    Clin Exp Pharmacol Physiol; 1999 Oct 15; 26(10):790-6. PubMed ID: 10549403
    [Abstract] [Full Text] [Related]

  • 20. Endothelium-derived hyperpolarizing factor-mediated renal vasodilatory response is impaired during acute and chronic hyperhomocysteinemia.
    De Vriese AS, Blom HJ, Heil SG, Mortier S, Kluijtmans LA, Van de Voorde J, Lameire NH.
    Circulation; 2004 May 18; 109(19):2331-6. PubMed ID: 15117854
    [Abstract] [Full Text] [Related]

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