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Journal Abstract Search
364 related items for PubMed ID: 10531299
1. Correction of defective protein trafficking of a mutant HERG potassium channel in human long QT syndrome. Pharmacological and temperature effects. Zhou Z, Gong Q, January CT. J Biol Chem; 1999 Oct 29; 274(44):31123-6. PubMed ID: 10531299 [Abstract] [Full Text] [Related]
7. Defective protein trafficking in hERG-associated hereditary long QT syndrome (LQT2): molecular mechanisms and restoration of intracellular protein processing. Thomas D, Kiehn J, Katus HA, Karle CA. Cardiovasc Res; 2003 Nov 01; 60(2):235-41. PubMed ID: 14613852 [Abstract] [Full Text] [Related]
9. Novel characteristics of a misprocessed mutant HERG channel linked to hereditary long QT syndrome. Ficker E, Thomas D, Viswanathan PC, Dennis AT, Priori SG, Napolitano C, Memmi M, Wible BA, Kaufman ES, Iyengar S, Schwartz PJ, Rudy Y, Brown AM. Am J Physiol Heart Circ Physiol; 2000 Oct 01; 279(4):H1748-56. PubMed ID: 11009462 [Abstract] [Full Text] [Related]
14. A novel mutation (T65P) in the PAS domain of the human potassium channel HERG results in the long QT syndrome by trafficking deficiency. Paulussen A, Raes A, Matthijs G, Snyders DJ, Cohen N, Aerssens J. J Biol Chem; 2002 Dec 13; 277(50):48610-6. PubMed ID: 12354768 [Abstract] [Full Text] [Related]
16. A mechanistic link between an inherited and an acquired cardiac arrhythmia: HERG encodes the IKr potassium channel. Sanguinetti MC, Jiang C, Curran ME, Keating MT. Cell; 1995 Apr 21; 81(2):299-307. PubMed ID: 7736582 [Abstract] [Full Text] [Related]
17. A mechanism for the proarrhythmic effects of cisapride (Propulsid): high affinity blockade of the human cardiac potassium channel HERG. Rampe D, Roy ML, Dennis A, Brown AM. FEBS Lett; 1997 Nov 03; 417(1):28-32. PubMed ID: 9395068 [Abstract] [Full Text] [Related]
18. HERG channel dysfunction in human long QT syndrome. Intracellular transport and functional defects. Zhou Z, Gong Q, Epstein ML, January CT. J Biol Chem; 1998 Aug 14; 273(33):21061-6. PubMed ID: 9694858 [Abstract] [Full Text] [Related]
19. Retention in the endoplasmic reticulum as a mechanism of dominant-negative current suppression in human long QT syndrome. Ficker E, Dennis AT, Obejero-Paz CA, Castaldo P, Taglialatela M, Brown AM. J Mol Cell Cardiol; 2000 Dec 14; 32(12):2327-37. PubMed ID: 11113008 [Abstract] [Full Text] [Related]
20. Pharmacological correction of long QT-linked mutations in KCNH2 (hERG) increases the trafficking of Kv11.1 channels stored in the transitional endoplasmic reticulum. Smith JL, Reloj AR, Nataraj PS, Bartos DC, Schroder EA, Moss AJ, Ohno S, Horie M, Anderson CL, January CT, Delisle BP. Am J Physiol Cell Physiol; 2013 Nov 01; 305(9):C919-30. PubMed ID: 23864605 [Abstract] [Full Text] [Related] Page: [Next] [New Search]