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109 related items for PubMed ID: 10536672
1. Sodium influx via a non-selective pathway activated by the removal of extracellular divalent cations: possible role in the calcium paradox. Bosteels S, Matejovic P, Flameng W, Mubagwa K. Cardiovasc Res; 1999 Aug 01; 43(2):417-25. PubMed ID: 10536672 [Abstract] [Full Text] [Related]
2. Extracellular divalent cations block a cation non-selective conductance unrelated to calcium channels in rat cardiac muscle. Mubagwa K, Stengl M, Flameng W. J Physiol; 1997 Jul 15; 502 ( Pt 2)(Pt 2):235-47. PubMed ID: 9263906 [Abstract] [Full Text] [Related]
3. Modulation of the extracellular divalent cation-inhibited non-selective conductance in cardiac cells by metabolic inhibition and by oxidants. Macianskiene R, Matejovic P, Sipido K, Flameng W, Mubagwa K. J Mol Cell Cardiol; 2001 Jul 15; 33(7):1371-85. PubMed ID: 11437543 [Abstract] [Full Text] [Related]
4. Monovalent cation and L-type Ca2+ channels participate in calcium paradox-like phenomenon in rabbit aortic smooth muscle cells. Zakharov SI, Mongayt DA, Cohen RA, Bolotina VM. J Physiol; 1999 Jan 01; 514 ( Pt 1)(Pt 1):71-81. PubMed ID: 9831717 [Abstract] [Full Text] [Related]
10. Pathways for the movements of ions during calcium-free perfusion and the induction of the 'calcium paradox'. Tunstall J, Busselen P, Rodrigo GC, Chapman RA. J Mol Cell Cardiol; 1986 Mar 01; 18(3):241-54. PubMed ID: 2420998 [Abstract] [Full Text] [Related]
11. Mechanisms of glutamate-stimulated Mg2+ influx and subsequent Mg2+ efflux in rat forebrain neurones in culture. Stout AK, Li-Smerin Y, Johnson JW, Reynolds IJ. J Physiol; 1996 May 01; 492 ( Pt 3)(Pt 3):641-57. PubMed ID: 8734978 [Abstract] [Full Text] [Related]
12. A non-selective cation current activated via the multifunctional Ca(2+)-calmodulin-dependent protein kinase in human epithelial cells. Braun AP, Schulman H. J Physiol; 1995 Oct 01; 488 ( Pt 1)(Pt 1):37-55. PubMed ID: 8568664 [Abstract] [Full Text] [Related]
13. Manipulation of intracellular sodium by extracellular divalent cations: a 23Na and 31P NMR study on intact rat hearts. Van Echteld CJ, Van Emous JG, Jansen MA, Schreur JH, Ruigrok TJ. J Mol Cell Cardiol; 1998 Jan 01; 30(1):119-26. PubMed ID: 9500870 [Abstract] [Full Text] [Related]
14. Nifedipine blocks Ca2+ store refilling through a pathway not involving L-type Ca2+ channels in rabbit arteriolar smooth muscle. Curtis TM, Scholfield CN. J Physiol; 2001 May 01; 532(Pt 3):609-23. PubMed ID: 11313433 [Abstract] [Full Text] [Related]
15. The role of Na(+)-Ca2+ exchange in activation of excitation-contraction coupling in rat ventricular myocytes. Wasserstrom JA, Vites AM. J Physiol; 1996 Jun 01; 493 ( Pt 2)(Pt 2):529-42. PubMed ID: 8782114 [Abstract] [Full Text] [Related]
16. Na+ current and Ca2+ release from the sarcoplasmic reticulum during action potentials in guinea-pig ventricular myocytes. Sipido KR, Carmeliet E, Pappano A. J Physiol; 1995 Nov 15; 489 ( Pt 1)(Pt 1):1-17. PubMed ID: 8583394 [Abstract] [Full Text] [Related]
17. Functional characterization of ion permeation pathway in the N-type Ca2+ channel. Wakamori M, Strobeck M, Niidome T, Teramoto T, Imoto K, Mori Y. J Neurophysiol; 1998 Feb 15; 79(2):622-34. PubMed ID: 9463426 [Abstract] [Full Text] [Related]
18. Properties of the fluorescent sodium indicator "SBFI" in rat and rabbit cardiac myocytes. Levi AJ, Lee CO, Brooksby P. J Cardiovasc Electrophysiol; 1994 Mar 15; 5(3):241-57. PubMed ID: 7864922 [Abstract] [Full Text] [Related]