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Journal Abstract Search

1474 related items for PubMed ID: 10579212

  • 1. Prior D1 dopamine receptor stimulation is required to prime D2-mediated striatal Fos expression in 6-hydroxydopamine-lesioned rats.
    Pollack AE, Yates TM.
    Neuroscience; 1999; 94(2):505-14. PubMed ID: 10579212
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  • 8. Repeated D1 dopamine receptor agonist administration prevents the development of both D1 and D2 striatal receptor supersensitivity following denervation.
    Hu XT, White FJ.
    Synapse; 1992 Mar; 10(3):206-16. PubMed ID: 1532677
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  • 10. D1-like and D2-like dopamine receptors synergistically activate rotation and c-fos expression in the dopamine-depleted striatum in a rat model of Parkinson's disease.
    Paul ML, Graybiel AM, David JC, Robertson HA.
    J Neurosci; 1992 Oct; 12(10):3729-42. PubMed ID: 1357113
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  • 12. Role of a 35 kDa fos-related antigen (FRA) in the long-term induction of striatal dynorphin expression in the 6-hydroxydopamine lesioned rat.
    Bronstein DM, Ye H, Pennypacker KR, Hudson PM, Hong JS.
    Brain Res Mol Brain Res; 1994 May; 23(3):191-203. PubMed ID: 7914658
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  • 13. Abnormal involuntary movement (AIM) expression following D2 dopamine agonist challenge is determined by the nature of prior dopamine receptor stimulation (priming) in 6-hydroxydopamine lesioned rats.
    Drake JD, Kibuuka LN, Dimitrov KD, Pollack AE.
    Pharmacol Biochem Behav; 2013 Apr; 105():26-33. PubMed ID: 23369985
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  • 14. Effects of intrasubthalamic injection of dopamine receptor agonists on subthalamic neurons in normal and 6-hydroxydopamine-lesioned rats: an electrophysiological and c-Fos study.
    Hassani OK, Féger J.
    Neuroscience; 1999 Apr; 92(2):533-43. PubMed ID: 10408602
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  • 15. Striatal Fos expression is indicative of dopamine D1/D2 synergism and receptor supersensitivity.
    LaHoste GJ, Yu J, Marshall JF.
    Proc Natl Acad Sci U S A; 1993 Aug 15; 90(16):7451-5. PubMed ID: 8102797
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  • 16. Ontogenetic quinpirole treatments fail to prime for D2 agonist-enhancement of locomotor activity in 6-hydroxydopamine-lesioned rats.
    Brus R, Kostrzewa RM, Nowak P, Perry KW, Kostrzewa JP.
    Neurotox Res; 2003 Aug 15; 5(5):329-38. PubMed ID: 14715452
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  • 17. Dopamine supersensitivity and D1/D2 synergism are unrelated to changes in striatal receptor density.
    LaHoste GJ, Marshall JF.
    Synapse; 1992 Sep 15; 12(1):14-26. PubMed ID: 1357762
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  • 18. The atypical dopamine D1 receptor agonist SKF 83959 induces striatal Fos expression in rats.
    Wirtshafter D, Osborn CV.
    Eur J Pharmacol; 2005 Dec 28; 528(1-3):88-94. PubMed ID: 16324697
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  • 19. Autoradiographic study of striatal dopamine re-uptake sites and dopamine D1 and D2 receptors in a 6-hydroxydopamine and quinolinic acid double-lesion rat model of striatonigral degeneration (multiple system atrophy) and effects of embryonic ventral mesencephalic, striatal or co-grafts.
    Puschban Z, Scherfler C, Granata R, Laboyrie P, Quinn NP, Jenner P, Poewe W, Wenning GK.
    Neuroscience; 2000 Dec 28; 95(2):377-88. PubMed ID: 10658617
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  • 20. NMDA glutamate receptor stimulation is required for the expression of D2 dopamine mediated responses in apomorphine primed 6-hydroxydopamine lesioned rats.
    Pollack AE, Haisley EC.
    Brain Res; 2001 Apr 06; 897(1-2):213-6. PubMed ID: 11282380
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