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231 related items for PubMed ID: 10594817
1. Interaction between the protein InlB of Listeria monocytogenes and lipoteichoic acid: a novel mechanism of protein association at the surface of gram-positive bacteria. Jonquières R, Bierne H, Fiedler F, Gounon P, Cossart P. Mol Microbiol; 1999 Dec; 34(5):902-14. PubMed ID: 10594817 [Abstract] [Full Text] [Related]
2. Galactosylated wall teichoic acid, but not lipoteichoic acid, retains InlB on the surface of serovar 4b Listeria monocytogenes. Sumrall ET, Schefer CRE, Rismondo J, Schneider SR, Boulos S, Gründling A, Loessner MJ, Shen Y. Mol Microbiol; 2020 Mar; 113(3):638-649. PubMed ID: 32185836 [Abstract] [Full Text] [Related]
3. l-Rhamnosylation of wall teichoic acids promotes efficient surface association of Listeria monocytogenes virulence factors InlB and Ami through interaction with GW domains. Carvalho F, Sousa S, Cabanes D. Environ Microbiol; 2018 Nov; 20(11):3941-3951. PubMed ID: 29984543 [Abstract] [Full Text] [Related]
4. Glucose Decoration on Wall Teichoic Acid Is Required for Phage Adsorption and InlB-Mediated Virulence in Listeria ivanovii. Sumrall ET, Schneider SR, Boulos S, Loessner MJ, Shen Y. J Bacteriol; 2021 Jul 22; 203(16):e0013621. PubMed ID: 34096780 [Abstract] [Full Text] [Related]
5. InlB: an invasion protein of Listeria monocytogenes with a novel type of surface association. Braun L, Dramsi S, Dehoux P, Bierne H, Lindahl G, Cossart P. Mol Microbiol; 1997 Jul 22; 25(2):285-94. PubMed ID: 9282740 [Abstract] [Full Text] [Related]
6. Phage resistance at the cost of virulence: Listeria monocytogenes serovar 4b requires galactosylated teichoic acids for InlB-mediated invasion. Sumrall ET, Shen Y, Keller AP, Rismondo J, Pavlou M, Eugster MR, Boulos S, Disson O, Thouvenot P, Kilcher S, Wollscheid B, Cabanes D, Lecuit M, Gründling A, Loessner MJ. PLoS Pathog; 2019 Oct 22; 15(10):e1008032. PubMed ID: 31589660 [Abstract] [Full Text] [Related]
7. Identification of a Lipoteichoic Acid Glycosyltransferase Enzyme Reveals that GW-Domain-Containing Proteins Can Be Retained in the Cell Wall of Listeria monocytogenes in the Absence of Lipoteichoic Acid or Its Modifications. Percy MG, Karinou E, Webb AJ, Gründling A. J Bacteriol; 2016 Aug 01; 198(15):2029-42. PubMed ID: 27185829 [Abstract] [Full Text] [Related]
8. The 213-amino-acid leucine-rich repeat region of the listeria monocytogenes InlB protein is sufficient for entry into mammalian cells, stimulation of PI 3-kinase and membrane ruffling. Braun L, Nato F, Payrastre B, Mazié JC, Cossart P. Mol Microbiol; 1999 Oct 01; 34(1):10-23. PubMed ID: 10540282 [Abstract] [Full Text] [Related]
9. Discovery of genes required for lipoteichoic acid glycosylation predicts two distinct mechanisms for wall teichoic acid glycosylation. Rismondo J, Percy MG, Gründling A. J Biol Chem; 2018 Mar 02; 293(9):3293-3306. PubMed ID: 29343515 [Abstract] [Full Text] [Related]
10. gC1q-R/p32, a C1q-binding protein, is a receptor for the InlB invasion protein of Listeria monocytogenes. Braun L, Ghebrehiwet B, Cossart P. EMBO J; 2000 Apr 03; 19(7):1458-66. PubMed ID: 10747014 [Abstract] [Full Text] [Related]
11. The Host Scaffolding Protein Filamin A and the Exocyst Complex Control Exocytosis during InlB-Mediated Entry of Listeria monocytogenes. Bhalla M, Van Ngo H, Gyanwali GC, Ireton K. Infect Immun; 2019 Jan 03; 87(1):. PubMed ID: 30348826 [Abstract] [Full Text] [Related]
12. Cell-surface anchoring of Listeria adhesion protein on L. monocytogenes is fastened by internalin B for pathogenesis. Liu D, Bai X, Helmick HDB, Samaddar M, Amalaradjou MAR, Li X, Tenguria S, Gallina NLF, Xu L, Drolia R, Aryal UK, Moreira GMSG, Hust M, Seleem MN, Kokini JL, Ostafe R, Cox A, Bhunia AK. Cell Rep; 2023 May 30; 42(5):112515. PubMed ID: 37171960 [Abstract] [Full Text] [Related]
13. Internalin B is essential for adhesion and mediates the invasion of Listeria monocytogenes into human endothelial cells. Parida SK, Domann E, Rohde M, Müller S, Darji A, Hain T, Wehland J, Chakraborty T. Mol Microbiol; 1998 Apr 30; 28(1):81-93. PubMed ID: 9593298 [Abstract] [Full Text] [Related]
14. Formation of D-alanyl-lipoteichoic acid is required for adhesion and virulence of Listeria monocytogenes. Abachin E, Poyart C, Pellegrini E, Milohanic E, Fiedler F, Berche P, Trieu-Cuot P. Mol Microbiol; 2002 Jan 30; 43(1):1-14. PubMed ID: 11849532 [Abstract] [Full Text] [Related]
15. Streptococcus gordonii Type I Lipoteichoic Acid Contributes to Surface Protein Biogenesis. Lima BP, Kho K, Nairn BL, Davies JR, Svensäter G, Chen R, Steffes A, Vreeman GW, Meredith TC, Herzberg MC. mSphere; 2019 Dec 04; 4(6):. PubMed ID: 31801844 [Abstract] [Full Text] [Related]
16. Synergy between the N- and C-terminal domains of InlB for efficient invasion of non-phagocytic cells by Listeria monocytogenes. Jonquières R, Pizarro-Cerdá J, Cossart P. Mol Microbiol; 2001 Nov 04; 42(4):955-65. PubMed ID: 11737639 [Abstract] [Full Text] [Related]
17. Phylogenetically Defined Isoforms of Listeria monocytogenes Invasion Factor InlB Differently Activate Intracellular Signaling Pathways and Interact with the Receptor gC1q-R. Chalenko Y, Kalinin E, Marchenkov V, Sysolyatina E, Surin A, Sobyanin K, Ermolaeva S. Int J Mol Sci; 2019 Aug 24; 20(17):. PubMed ID: 31450632 [Abstract] [Full Text] [Related]