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Journal Abstract Search

431 related items for PubMed ID: 10625696

  • 1. BNIP3 heterodimerizes with Bcl-2/Bcl-X(L) and induces cell death independent of a Bcl-2 homology 3 (BH3) domain at both mitochondrial and nonmitochondrial sites.
    Ray R, Chen G, Vande Velde C, Cizeau J, Park JH, Reed JC, Gietz RD, Greenberg AH.
    J Biol Chem; 2000 Jan 14; 275(2):1439-48. PubMed ID: 10625696
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  • 2. Adenovirus E1B-19K/BCL-2 interacting protein BNIP3 contains a BH3 domain and a mitochondrial targeting sequence.
    Yasuda M, Theodorakis P, Subramanian T, Chinnadurai G.
    J Biol Chem; 1998 May 15; 273(20):12415-21. PubMed ID: 9575197
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  • 3. The C. elegans orthologue ceBNIP3 interacts with CED-9 and CED-3 but kills through a BH3- and caspase-independent mechanism.
    Cizeau J, Ray R, Chen G, Gietz RD, Greenberg AH.
    Oncogene; 2000 Nov 16; 19(48):5453-63. PubMed ID: 11114722
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  • 4. Sequence-specific dimerization of the transmembrane domain of the "BH3-only" protein BNIP3 in membranes and detergent.
    Sulistijo ES, Jaszewski TM, MacKenzie KR.
    J Biol Chem; 2003 Dec 19; 278(51):51950-6. PubMed ID: 14532263
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  • 5. BNIP3alpha: a human homolog of mitochondrial proapoptotic protein BNIP3.
    Yasuda M, Han JW, Dionne CA, Boyd JM, Chinnadurai G.
    Cancer Res; 1999 Feb 01; 59(3):533-7. PubMed ID: 9973195
    [Abstract] [Full Text] [Related]

  • 6. Peptides derived from BH3 domains of Bcl-2 family members: a comparative analysis of inhibition of Bcl-2, Bcl-x(L) and Bax oligomerization, induction of cytochrome c release, and activation of cell death.
    Shangary S, Johnson DE.
    Biochemistry; 2002 Jul 30; 41(30):9485-95. PubMed ID: 12135371
    [Abstract] [Full Text] [Related]

  • 7. BNIP3 subfamily BH3-only proteins: mitochondrial stress sensors in normal and pathological functions.
    Chinnadurai G, Vijayalingam S, Gibson SB.
    Oncogene; 2008 Dec 30; 27 Suppl 1(Suppl 1):S114-27. PubMed ID: 19641497
    [Abstract] [Full Text] [Related]

  • 8. BH3 domain of BAD is required for heterodimerization with BCL-XL and pro-apoptotic activity.
    Zha J, Harada H, Osipov K, Jockel J, Waksman G, Korsmeyer SJ.
    J Biol Chem; 1997 Sep 26; 272(39):24101-4. PubMed ID: 9305851
    [Abstract] [Full Text] [Related]

  • 9. The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4.
    Huang DC, Adams JM, Cory S.
    EMBO J; 1998 Feb 16; 17(4):1029-39. PubMed ID: 9463381
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  • 11. Bcl-XL protects BimEL-induced Bax conformational change and cytochrome C release independent of interacting with Bax or BimEL.
    Yamaguchi H, Wang HG.
    J Biol Chem; 2002 Nov 01; 277(44):41604-12. PubMed ID: 12198137
    [Abstract] [Full Text] [Related]

  • 12. Acidosis regulates the stability, hydrophobicity, and activity of the BH3-only protein Bnip3.
    Frazier DP, Wilson A, Graham RM, Thompson JW, Bishopric NH, Webster KA.
    Antioxid Redox Signal; 2006 Nov 01; 8(9-10):1625-34. PubMed ID: 16987017
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  • 15. Bcl-2/E1B 19 kDa-interacting protein 3-like protein (Bnip3L) interacts with bcl-2/Bcl-xL and induces apoptosis by altering mitochondrial membrane permeability.
    Imazu T, Shimizu S, Tagami S, Matsushima M, Nakamura Y, Miki T, Okuyama A, Tsujimoto Y.
    Oncogene; 1999 Aug 12; 18(32):4523-9. PubMed ID: 10467396
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  • 18. Mutagenesis of the BH3 domain of BAX identifies residues critical for dimerization and killing.
    Wang K, Gross A, Waksman G, Korsmeyer SJ.
    Mol Cell Biol; 1998 Oct 12; 18(10):6083-9. PubMed ID: 9742125
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  • 20. Characterization of the anti-apoptotic mechanism of Bcl-B.
    Zhai D, Ke N, Zhang H, Ladror U, Joseph M, Eichinger A, Godzik A, Ng SC, Reed JC.
    Biochem J; 2003 Nov 15; 376(Pt 1):229-36. PubMed ID: 12921534
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