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479 related items for PubMed ID: 10639121

  • 1. Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity.
    Shimizu S, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 2000 Jan 18; 97(2):577-82. PubMed ID: 10639121
    [Abstract] [Full Text] [Related]

  • 2. Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells.
    Shimizu S, Matsuoka Y, Shinohara Y, Yoneda Y, Tsujimoto Y.
    J Cell Biol; 2001 Jan 22; 152(2):237-50. PubMed ID: 11266442
    [Abstract] [Full Text] [Related]

  • 3. Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC.
    Shimizu S, Narita M, Tsujimoto Y.
    Nature; 1999 Jun 03; 399(6735):483-7. PubMed ID: 10365962
    [Abstract] [Full Text] [Related]

  • 4. Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release.
    Kandasamy K, Srinivasula SM, Alnemri ES, Thompson CB, Korsmeyer SJ, Bryant JL, Srivastava RK.
    Cancer Res; 2003 Apr 01; 63(7):1712-21. PubMed ID: 12670926
    [Abstract] [Full Text] [Related]

  • 5. Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator.
    Shimizu S, Shinohara Y, Tsujimoto Y.
    Oncogene; 2000 Sep 07; 19(38):4309-18. PubMed ID: 10980606
    [Abstract] [Full Text] [Related]

  • 6. Activation of mitochondrial voltage-dependent anion channel by apro-apoptotic BH3-only protein Bim.
    Sugiyama T, Shimizu S, Matsuoka Y, Yoneda Y, Tsujimoto Y.
    Oncogene; 2002 Jul 25; 21(32):4944-56. PubMed ID: 12118373
    [Abstract] [Full Text] [Related]

  • 7. Direct addition of BimL to mitochondria does not lead to cytochrome c release.
    Terradillos O, Montessuit S, Huang DC, Martinou JC.
    FEBS Lett; 2002 Jul 03; 522(1-3):29-34. PubMed ID: 12095614
    [Abstract] [Full Text] [Related]

  • 8. Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria.
    Narita M, Shimizu S, Ito T, Chittenden T, Lutz RJ, Matsuda H, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 1998 Dec 08; 95(25):14681-6. PubMed ID: 9843949
    [Abstract] [Full Text] [Related]

  • 9. Bid-induced cytochrome c release is mediated by a pathway independent of mitochondrial permeability transition pore and Bax.
    Kim TH, Zhao Y, Barber MJ, Kuharsky DK, Yin XM.
    J Biol Chem; 2000 Dec 15; 275(50):39474-81. PubMed ID: 10982793
    [Abstract] [Full Text] [Related]

  • 10. Two pathways for tBID-induced cytochrome c release from rat brain mitochondria: BAK- versus BAX-dependence.
    Brustovetsky N, Dubinsky JM, Antonsson B, Jemmerson R.
    J Neurochem; 2003 Jan 15; 84(1):196-207. PubMed ID: 12485416
    [Abstract] [Full Text] [Related]

  • 11. Bid, but not Bax, regulates VDAC channels.
    Rostovtseva TK, Antonsson B, Suzuki M, Youle RJ, Colombini M, Bezrukov SM.
    J Biol Chem; 2004 Apr 02; 279(14):13575-83. PubMed ID: 14729675
    [Abstract] [Full Text] [Related]

  • 12. Control of mitochondrial permeability by Bcl-2 family members.
    Sharpe JC, Arnoult D, Youle RJ.
    Biochim Biophys Acta; 2004 Mar 01; 1644(2-3):107-13. PubMed ID: 14996495
    [Abstract] [Full Text] [Related]

  • 13. Inhibition of Bax-induced cytochrome c release from neural cell and brain mitochondria by dibucaine and propranolol.
    Polster BM, Basañez G, Young M, Suzuki M, Fiskum G.
    J Neurosci; 2003 Apr 01; 23(7):2735-43. PubMed ID: 12684459
    [Abstract] [Full Text] [Related]

  • 14. VDAC2 inhibits BAK activation and mitochondrial apoptosis.
    Cheng EH, Sheiko TV, Fisher JK, Craigen WJ, Korsmeyer SJ.
    Science; 2003 Jul 25; 301(5632):513-7. PubMed ID: 12881569
    [Abstract] [Full Text] [Related]

  • 15. Mouse uterine epithelial apoptosis is associated with expression of mitochondrial voltage-dependent anion channels, release of cytochrome C from mitochondria, and the ratio of Bax to Bcl-2 or Bcl-X.
    Takagi-Morishita Y, Yamada N, Sugihara A, Iwasaki T, Tsujimura T, Terada N.
    Biol Reprod; 2003 Apr 25; 68(4):1178-84. PubMed ID: 12606449
    [Abstract] [Full Text] [Related]

  • 16. Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c.
    Shimizu S, Ide T, Yanagida T, Tsujimoto Y.
    J Biol Chem; 2000 Apr 21; 275(16):12321-5. PubMed ID: 10766872
    [Abstract] [Full Text] [Related]

  • 17. Bid induces cytochrome c-impermeable Bax channels in liposomes.
    Roucou X, Rostovtseva T, Montessuit S, Martinou JC, Antonsson B.
    Biochem J; 2002 May 01; 363(Pt 3):547-52. PubMed ID: 11964155
    [Abstract] [Full Text] [Related]

  • 18. Granzyme B can cause mitochondrial depolarization and cell death in the absence of BID, BAX, and BAK.
    Thomas DA, Scorrano L, Putcha GV, Korsmeyer SJ, Ley TJ.
    Proc Natl Acad Sci U S A; 2001 Dec 18; 98(26):14985-90. PubMed ID: 11752447
    [Abstract] [Full Text] [Related]

  • 19. BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death.
    Shimizu S, Konishi A, Kodama T, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 2000 Mar 28; 97(7):3100-5. PubMed ID: 10737788
    [Abstract] [Full Text] [Related]

  • 20. Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics.
    Letai A, Bassik MC, Walensky LD, Sorcinelli MD, Weiler S, Korsmeyer SJ.
    Cancer Cell; 2002 Sep 28; 2(3):183-92. PubMed ID: 12242151
    [Abstract] [Full Text] [Related]

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