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Journal Abstract Search

275 related items for PubMed ID: 10648821

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  • 2. Functional rescue of the nephrogenic diabetes insipidus-causing vasopressin V2 receptor mutants G185C and R202C by a second site suppressor mutation.
    Schülein R, Zühlke K, Krause G, Rosenthal W.
    J Biol Chem; 2001 Mar 16; 276(11):8384-92. PubMed ID: 11116139
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  • 3. Expression studies of two vasopressin V2 receptor gene mutations, R202C and 804insG, in nephrogenic diabetes insipidus.
    Tsukaguchi H, Matsubara H, Inada M.
    Kidney Int; 1995 Aug 16; 48(2):554-62. PubMed ID: 7564126
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  • 10. Molecular aspects of vasopressin receptor function.
    Schöneberg T, Kostenis E, Liu J, Gudermann T, Wess J.
    Adv Exp Med Biol; 1998 Aug 16; 449():347-58. PubMed ID: 10026824
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  • 11. A dileucine sequence and an upstream glutamate residue in the intracellular carboxyl terminus of the vasopressin V2 receptor are essential for cell surface transport in COS.M6 cells.
    Schülein R, Hermosilla R, Oksche A, Dehe M, Wiesner B, Krause G, Rosenthal W.
    Mol Pharmacol; 1998 Sep 16; 54(3):525-35. PubMed ID: 9730911
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  • 12. Site-directed mutagenesis of cysteinyl and serine residues of human thromboxane A2 receptor in insect cells.
    Chiang N, Kan WM, Tai HH.
    Arch Biochem Biophys; 1996 Oct 01; 334(1):9-17. PubMed ID: 8837733
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  • 13. A disulfide bonding interaction role for cysteines in the extracellular domain of the thyrotropin-releasing hormone receptor.
    Cook JV, McGregor A, Lee T, Milligan G, Eidne KA.
    Endocrinology; 1996 Jul 01; 137(7):2851-8. PubMed ID: 8770906
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  • 14. Polarized expression of the vasopressin V2 receptor in Madin-Darby canine kidney cells.
    Andersen-Beckh B, Dehe M, Schülein R, Wiesner B, Rutz C, Liebenhoff U, Rosenthal W, Oksche A.
    Kidney Int; 1999 Aug 01; 56(2):517-27. PubMed ID: 10432391
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  • 15. Vasopressin V2 receptor mutants that cause X-linked nephrogenic diabetes insipidus: analysis of expression, processing, and function.
    Oksche A, Schülein R, Rutz C, Liebenhoff U, Dickson J, Müller H, Birnbaumer M, Rosenthal W.
    Mol Pharmacol; 1996 Oct 01; 50(4):820-8. PubMed ID: 8863826
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  • 16. Misfolded vasopressin V2 receptors caused by extracellular point mutations entail congential nephrogenic diabetes insipidus.
    Postina R, Ufer E, Pfeiffer R, Knoers NV, Fahrenholz F.
    Mol Cell Endocrinol; 2000 Jun 01; 164(1-2):31-9. PubMed ID: 11026555
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  • 17. Binding-, intracellular transport-, and biosynthesis-defective mutants of vasopressin type 2 receptor in patients with X-linked nephrogenic diabetes insipidus.
    Tsukaguchi H, Matsubara H, Taketani S, Mori Y, Seido T, Inada M.
    J Clin Invest; 1995 Oct 01; 96(4):2043-50. PubMed ID: 7560098
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  • 18. Conserved extracellular cysteine pair in the M3 muscarinic acetylcholine receptor is essential for proper receptor cell surface localization but not for G protein coupling.
    Zeng FY, Soldner A, Schöneberg T, Wess J.
    J Neurochem; 1999 Jun 01; 72(6):2404-14. PubMed ID: 10349850
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  • 19. An intramolecular disulfide bond between conserved extracellular cysteines in the gonadotropin-releasing hormone receptor is essential for binding and activation.
    Cook JV, Eidne KA.
    Endocrinology; 1997 Jul 01; 138(7):2800-6. PubMed ID: 9202220
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  • 20. Diverse vasopressin V2 receptor functionality underlying partial congenital nephrogenic diabetes insipidus.
    Faerch M, Christensen JH, Rittig S, Johansson JO, Gregersen N, de Zegher F, Corydon TJ.
    Am J Physiol Renal Physiol; 2009 Dec 01; 297(6):F1518-25. PubMed ID: 19812297
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