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152 related items for PubMed ID: 10686508

  • 1. Sodium salicylate-induced apoptosis of human peripheral blood eosinophils is independent of the activation of c-Jun N-terminal kinase and p38 mitogen-activated protein kinase.
    Wong CK, Zhang JP, Lam CW, Ho CY, Hjelm NM.
    Int Arch Allergy Immunol; 2000 Jan; 121(1):44-52. PubMed ID: 10686508
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  • 3. Opposing effects of sodium salicylate and haematopoietic cytokines IL-3, IL-5 and GM-CSF on mitogen-activated protein kinases and apoptosis of EoL-1 cells.
    Wong CK, Zhang JP, Lam CW, Ho CY, Hjelm NM.
    Immunol Invest; 1999 Jan; 28(5-6):365-79. PubMed ID: 10574634
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  • 5. Sodium salicylate induces apoptosis via p38 mitogen-activated protein kinase but inhibits tumor necrosis factor-induced c-Jun N-terminal kinase/stress-activated protein kinase activation.
    Schwenger P, Bellosta P, Vietor I, Basilico C, Skolnik EY, Vilcek J.
    Proc Natl Acad Sci U S A; 1997 Apr 01; 94(7):2869-73. PubMed ID: 9096313
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  • 9. Sodium salicylate inhibits macrophage TNF-alpha production and alters MAPK activation.
    Vittimberga FJ, McDade TP, Perugini RA, Callery MP.
    J Surg Res; 1999 Jun 15; 84(2):143-9. PubMed ID: 10357911
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  • 12. Activation of the p38 and p42/p44 mitogen-activated protein kinase families by the histamine H(1) receptor in DDT(1)MF-2 cells.
    Robinson AJ, Dickenson JM.
    Br J Pharmacol; 2001 Aug 15; 133(8):1378-86. PubMed ID: 11498525
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  • 13. Role of mitogen-activated protein kinases in S-nitrosoglutathione-induced macrophage apoptosis.
    Callsen D, Brüne B.
    Biochemistry; 1999 Feb 23; 38(8):2279-86. PubMed ID: 10029520
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  • 14. Tumour necrosis factor-induced activation of c-Jun N-terminal kinase is sensitive to caspase-dependent modulation while activation of mitogen-activated protein kinase (MAPK) or p38 MAPK is not.
    Mohamed AA, Jupp OJ, Anderson HM, Littlejohn AF, Vandenabeele P, MacEwan DJ.
    Biochem J; 2002 Aug 15; 366(Pt 1):145-55. PubMed ID: 11996667
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  • 15. Hyperosmolarity-induced interleukin-8 expression in human bronchial epithelial cells through p38 mitogen-activated protein kinase.
    Hashimoto S, Matsumoto K, Gon Y, Nakayama T, Takeshita I, Horie T.
    Am J Respir Crit Care Med; 1999 Feb 15; 159(2):634-40. PubMed ID: 9927384
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  • 16. Activation of extracellular signal-regulated kinase and c-Jun-NH(2)-terminal kinase but not p38 mitogen-activated protein kinases is required for RRR-alpha-tocopheryl succinate-induced apoptosis of human breast cancer cells.
    Yu W, Liao QY, Hantash FM, Sanders BG, Kline K.
    Cancer Res; 2001 Sep 01; 61(17):6569-76. PubMed ID: 11522656
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  • 17. Interleukin-25-induced chemokines and interleukin-6 release from eosinophils is mediated by p38 mitogen-activated protein kinase, c-Jun N-terminal kinase, and nuclear factor-kappaB.
    Wong CK, Cheung PF, Ip WK, Lam CW.
    Am J Respir Cell Mol Biol; 2005 Aug 01; 33(2):186-94. PubMed ID: 15860795
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  • 18. Arachidonic acid directly activates members of the mitogen-activated protein kinase superfamily in rabbit proximal tubule cells.
    Alexander LD, Cui XL, Falck JR, Douglas JG.
    Kidney Int; 2001 Jun 01; 59(6):2039-53. PubMed ID: 11380805
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  • 19. An inhibitor of p38 and JNK MAP kinases prevents activation of caspase and apoptosis of cultured cerebellar granule neurons.
    Harada J, Sugimoto M.
    Jpn J Pharmacol; 1999 Mar 01; 79(3):369-78. PubMed ID: 10230866
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  • 20. JNK (c-Jun N-terminal kinase) and p38 activation in receptor-mediated and chemically-induced apoptosis of T-cells: differential requirements for caspase activation.
    MacFarlane M, Cohen GM, Dickens M.
    Biochem J; 2000 May 15; 348 Pt 1(Pt 1):93-101. PubMed ID: 10794718
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