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268 related items for PubMed ID: 10697873

  • 1. Redox metabolism in glucose-6-phosphate dehydrogenase deficient erythrocytes and its relation to antimalarial chemotherapy.
    Ginsburg H, Golenser J.
    Parassitologia; 1999 Sep; 41(1-3):309-11. PubMed ID: 10697873
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  • 4. Regulation of intracellular glutathione levels in erythrocytes infected with chloroquine-sensitive and chloroquine-resistant Plasmodium falciparum.
    Meierjohann S, Walter RD, Müller S.
    Biochem J; 2002 Dec 15; 368(Pt 3):761-8. PubMed ID: 12225291
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  • 5. Inhibition of the intraerythrocytic development of Plasmodium falciparum in glucose-6-phosphate dehydrogenase deficient erythrocytes is enhanced by oxidants and by crisis form factor.
    Golenser J, Miller J, Spira DT, Kosower NS, Vande Waa JA, Jensen JB.
    Trop Med Parasitol; 1988 Dec 15; 39(4):273-6. PubMed ID: 3067318
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  • 7. Pathways for the reduction of oxidized glutathione in the Plasmodium falciparum-infected erythrocyte: can parasite enzymes replace host red cell glucose-6-phosphate dehydrogenase?
    Roth EF, Schulman S, Vanderberg J, Olson J.
    Blood; 1986 Mar 15; 67(3):827-30. PubMed ID: 3511989
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  • 18. Susceptibility to hydrogen peroxide of Plasmodium falciparum infecting glucose-6-phosphate dehydrogenase-deficient erythrocytes.
    Kamchonwongpaisan S, Bunyaratvej A, Wanachiwanawin W, Yuthavong Y.
    Parasitology; 1989 Oct 15; 99 Pt 2():171-4. PubMed ID: 2687776
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  • 19. The treatment of Plasmodium falciparum-infected erythrocytes with chloroquine leads to accumulation of ferriprotoporphyrin IX bound to particular parasite proteins and to the inhibition of the parasite's 6-phosphogluconate dehydrogenase.
    Famin O, Ginsburg H.
    Parasite; 2003 Mar 15; 10(1):39-50. PubMed ID: 12669348
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  • 20. The effect and mechanism of inhibiting glucose-6-phosphate dehydrogenase activity on the proliferation of Plasmodium falciparum.
    Zhang Z, Chen X, Jiang C, Fang Z, Feng Y, Jiang W.
    Biochim Biophys Acta Mol Cell Res; 2017 May 15; 1864(5):771-781. PubMed ID: 28214533
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