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179 related items for PubMed ID: 10706885

  • 1.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 2. Loss of SHIP and CIS recruitment to the granulocyte colony-stimulating factor receptor contribute to hyperproliferative responses in severe congenital neutropenia/acute myelogenous leukemia.
    Hunter MG, Jacob A, O'donnell LC, Agler A, Druhan LJ, Coggeshall KM, Avalos BR.
    J Immunol; 2004 Oct 15; 173(8):5036-45. PubMed ID: 15470047
    [Abstract] [Full Text] [Related]

  • 3. A Truncated Granulocyte Colony-stimulating Factor Receptor (G-CSFR) Inhibits Apoptosis Induced by Neutrophil Elastase G185R Mutant: IMPLICATION FOR UNDERSTANDING CSF3R GENE MUTATIONS IN SEVERE CONGENITAL NEUTROPENIA.
    Qiu Y, Zhang Y, Hu N, Dong F.
    J Biol Chem; 2017 Feb 24; 292(8):3496-3505. PubMed ID: 28073911
    [Abstract] [Full Text] [Related]

  • 4. Deletion of a critical internalization domain in the G-CSFR in acute myelogenous leukemia preceded by severe congenital neutropenia.
    Hunter MG, Avalos BR.
    Blood; 1999 Jan 15; 93(2):440-6. PubMed ID: 9885205
    [Abstract] [Full Text] [Related]

  • 5. G-CSFR ubiquitination critically regulates myeloid cell survival and proliferation.
    Ai J, Druhan LJ, Loveland MJ, Avalos BR.
    PLoS One; 2008 Jan 15; 3(10):e3422. PubMed ID: 18923646
    [Abstract] [Full Text] [Related]

  • 6. Increased granulocyte colony-stimulating factor responsiveness but normal resting granulopoiesis in mice carrying a targeted granulocyte colony-stimulating factor receptor mutation derived from a patient with severe congenital neutropenia.
    McLemore ML, Poursine-Laurent J, Link DC.
    J Clin Invest; 1998 Aug 01; 102(3):483-92. PubMed ID: 9691084
    [Abstract] [Full Text] [Related]

  • 7. G-CSF-induced tyrosine phosphorylation of Gab2 is Lyn kinase dependent and associated with enhanced Akt and differentiative, not proliferative, responses.
    Zhu QS, Robinson LJ, Roginskaya V, Corey SJ.
    Blood; 2004 May 01; 103(9):3305-12. PubMed ID: 14656892
    [Abstract] [Full Text] [Related]

  • 8. Sustained receptor activation and hyperproliferation in response to granulocyte colony-stimulating factor (G-CSF) in mice with a severe congenital neutropenia/acute myeloid leukemia-derived mutation in the G-CSF receptor gene.
    Hermans MH, Antonissen C, Ward AC, Mayen AE, Ploemacher RE, Touw IP.
    J Exp Med; 1999 Feb 15; 189(4):683-92. PubMed ID: 9989983
    [Abstract] [Full Text] [Related]

  • 9. Activation of Akt kinase by granulocyte colony-stimulating factor (G-CSF): evidence for the role of a tyrosine kinase activity distinct from the Janus kinases.
    Dong F, Larner AC.
    Blood; 2000 Mar 01; 95(5):1656-62. PubMed ID: 10688821
    [Abstract] [Full Text] [Related]

  • 10. The granulocyte colony-stimulating factor receptor and its role in disorders of granulopoiesis.
    Avalos BR.
    Leuk Lymphoma; 1998 Jan 01; 28(3-4):265-73. PubMed ID: 9517498
    [Abstract] [Full Text] [Related]

  • 11. Novel mechanism of G-CSF refractoriness in patients with severe congenital neutropenia.
    Druhan LJ, Ai J, Massullo P, Kindwall-Keller T, Ranalli MA, Avalos BR.
    Blood; 2005 Jan 15; 105(2):584-91. PubMed ID: 15353486
    [Abstract] [Full Text] [Related]

  • 12. Defective internalization and sustained activation of truncated granulocyte colony-stimulating factor receptor found in severe congenital neutropenia/acute myeloid leukemia.
    Ward AC, van Aesch YM, Schelen AM, Touw IP.
    Blood; 1999 Jan 15; 93(2):447-58. PubMed ID: 9885206
    [Abstract] [Full Text] [Related]

  • 13. Mutations in the gene for the granulocyte colony-stimulating-factor receptor in patients with acute myeloid leukemia preceded by severe congenital neutropenia.
    Dong F, Brynes RK, Tidow N, Welte K, Löwenberg B, Touw IP.
    N Engl J Med; 1995 Aug 24; 333(8):487-93. PubMed ID: 7542747
    [Abstract] [Full Text] [Related]

  • 14. Impaired neutrophil maturation in truncated murine G-CSF receptor-transgenic mice.
    Mitsui T, Watanabe S, Taniguchi Y, Hanada S, Ebihara Y, Sato T, Heike T, Mitsuyama M, Nakahata T, Tsuji K.
    Blood; 2003 Apr 15; 101(8):2990-5. PubMed ID: 12672695
    [Abstract] [Full Text] [Related]

  • 15. Cellular and molecular abnormalities in severe congenital neutropenia predisposing to leukemia.
    Aprikyan AA, Kutyavin T, Stein S, Aprikian P, Rodger E, Liles WC, Boxer LA, Dale DC.
    Exp Hematol; 2003 May 15; 31(5):372-81. PubMed ID: 12763135
    [Abstract] [Full Text] [Related]

  • 16. The carboxyl terminus of the granulocyte colony-stimulating factor receptor, truncated in patients with severe congenital neutropenia/acute myeloid leukemia, is required for SH2-containing phosphatase-1 suppression of Stat activation.
    Dong F, Qiu Y, Yi T, Touw IP, Larner AC.
    J Immunol; 2001 Dec 01; 167(11):6447-52. PubMed ID: 11714811
    [Abstract] [Full Text] [Related]

  • 17. A truncation mutant of Csf3r cooperates with PML-RARα to induce acute myeloid leukemia in mice.
    Kunter G, Woloszynek JR, Link DC.
    Exp Hematol; 2011 Dec 01; 39(12):1136-43. PubMed ID: 21911095
    [Abstract] [Full Text] [Related]

  • 18. Mutations in the granulocyte colony-stimulating factor receptor gene in patients with severe congenital neutropenia.
    Dong F, Dale DC, Bonilla MA, Freedman M, Fasth A, Neijens HJ, Palmblad J, Briars GL, Carlsson G, Veerman AJ, Welte K, Löwenberg B, Touw IP.
    Leukemia; 1997 Jan 01; 11(1):120-5. PubMed ID: 9001427
    [Abstract] [Full Text] [Related]

  • 19. Severe congenital neutropenia terminating in acute myeloid leukemia: disease progression associated with mutations in the granulocyte-colony stimulating factor receptor gene.
    Touw IP, Dong F.
    Leuk Res; 1996 Aug 01; 20(8):629-31. PubMed ID: 8913314
    [Abstract] [Full Text] [Related]

  • 20. Point mutations in the conserved box 1 region inactivate the human granulocyte colony-stimulating factor receptor for growth signal transduction and tyrosine phosphorylation of p75c-rel.
    Avalos BR, Hunter MG, Parker JM, Ceselski SK, Druker BJ, Corey SJ, Mehta VB.
    Blood; 1995 Jun 01; 85(11):3117-26. PubMed ID: 7538818
    [Abstract] [Full Text] [Related]


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