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Journal Abstract Search

138 related items for PubMed ID: 10720932

  • 1. Familial hyperinsulinism and pancreatic beta-cell ATP-sensitive potassium channels.
    Sharma N, Crane A, Gonzalez G, Bryan J, Aguilar-Bryan L.
    Kidney Int; 2000 Mar; 57(3):803-8. PubMed ID: 10720932
    [Abstract] [Full Text] [Related]

  • 2. Molecular biology of adenosine triphosphate-sensitive potassium channels.
    Aguilar-Bryan L, Bryan J.
    Endocr Rev; 1999 Apr; 20(2):101-35. PubMed ID: 10204114
    [Abstract] [Full Text] [Related]

  • 3. Hyperinsulinism of infancy: the regulated release of insulin by KATP channel-independent pathways.
    Straub SG, Cosgrove KE, Ammälä C, Shepherd RM, O'Brien RE, Barnes PD, Kuchinski N, Chapman JC, Schaeppi M, Glaser B, Lindley KJ, Sharp GW, Aynsley-Green A, Dunne MJ.
    Diabetes; 2001 Feb; 50(2):329-39. PubMed ID: 11272144
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  • 4. A point mutation inactivating the sulfonylurea receptor causes the severe form of persistent hyperinsulinemic hypoglycemia of infancy in Finland.
    Otonkoski T, Ammälä C, Huopio H, Cote GJ, Chapman J, Cosgrove K, Ashfield R, Huang E, Komulainen J, Ashcroft FM, Dunne MJ, Kere J, Thomas PM.
    Diabetes; 1999 Feb; 48(2):408-15. PubMed ID: 10334322
    [Abstract] [Full Text] [Related]

  • 5. The structure and function of the ATP-sensitive K+ channel in insulin-secreting pancreatic beta-cells.
    Miki T, Nagashima K, Seino S.
    J Mol Endocrinol; 1999 Apr; 22(2):113-23. PubMed ID: 10194514
    [Abstract] [Full Text] [Related]

  • 6. Defective trafficking and function of KATP channels caused by a sulfonylurea receptor 1 mutation associated with persistent hyperinsulinemic hypoglycemia of infancy.
    Cartier EA, Conti LR, Vandenberg CA, Shyng SL.
    Proc Natl Acad Sci U S A; 2001 Feb 27; 98(5):2882-7. PubMed ID: 11226335
    [Abstract] [Full Text] [Related]

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  • 8. Functional analyses of novel mutations in the sulfonylurea receptor 1 associated with persistent hyperinsulinemic hypoglycemia of infancy.
    Shyng SL, Ferrigni T, Shepard JB, Nestorowicz A, Glaser B, Permutt MA, Nichols CG.
    Diabetes; 1998 Jul 27; 47(7):1145-51. PubMed ID: 9648840
    [Abstract] [Full Text] [Related]

  • 9. Loss of functional KATP channels in pancreatic beta-cells causes persistent hyperinsulinemic hypoglycemia of infancy.
    Kane C, Shepherd RM, Squires PE, Johnson PR, James RF, Milla PJ, Aynsley-Green A, Lindley KJ, Dunne MJ.
    Nat Med; 1996 Dec 27; 2(12):1344-7. PubMed ID: 8946833
    [Abstract] [Full Text] [Related]

  • 10. Genetic analysis of Japanese patients with persistent hyperinsulinemic hypoglycemia of infancy: nucleotide-binding fold-2 mutation impairs cooperative binding of adenine nucleotides to sulfonylurea receptor 1.
    Tanizawa Y, Matsuda K, Matsuo M, Ohta Y, Ochi N, Adachi M, Koga M, Mizuno S, Kajita M, Tanaka Y, Tachibana K, Inoue H, Furukawa S, Amachi T, Ueda K, Oka Y.
    Diabetes; 2000 Jan 27; 49(1):114-20. PubMed ID: 10615958
    [Abstract] [Full Text] [Related]

  • 11. Mutation of the pancreatic islet inward rectifier Kir6.2 also leads to familial persistent hyperinsulinemic hypoglycemia of infancy.
    Thomas P, Ye Y, Lightner E.
    Hum Mol Genet; 1996 Nov 27; 5(11):1809-12. PubMed ID: 8923010
    [Abstract] [Full Text] [Related]

  • 12. ABCC8 (SUR1) and KCNJ11 (KIR6.2) mutations in persistent hyperinsulinemic hypoglycemia of infancy and evaluation of different therapeutic measures.
    Darendeliler F, Fournet JC, Baş F, Junien C, Gross MS, Bundak R, Saka N, Günöz H.
    J Pediatr Endocrinol Metab; 2002 Nov 27; 15(7):993-1000. PubMed ID: 12199344
    [Abstract] [Full Text] [Related]

  • 13. Congenital hyperinsulinism: molecular basis of a heterogeneous disease.
    Meissner T, Beinbrech B, Mayatepek E.
    Hum Mutat; 1999 Nov 27; 13(5):351-61. PubMed ID: 10338089
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  • 15. Diverse roles of K(ATP) channels learned from Kir6.2 genetically engineered mice.
    Seino S, Iwanaga T, Nagashima K, Miki T.
    Diabetes; 2000 Mar 27; 49(3):311-8. PubMed ID: 10868950
    [Abstract] [Full Text] [Related]

  • 16. Engineering a glucose-responsive human insulin-secreting cell line from islets of Langerhans isolated from a patient with persistent hyperinsulinemic hypoglycemia of infancy.
    MacFarlane WM, Chapman JC, Shepherd RM, Hashmi MN, Kamimura N, Cosgrove KE, O'Brien RE, Barnes PD, Hart AW, Docherty HM, Lindley KJ, Aynsley-Green A, James RF, Docherty K, Dunne MJ.
    J Biol Chem; 1999 Nov 26; 274(48):34059-66. PubMed ID: 10567373
    [Abstract] [Full Text] [Related]

  • 17. Identification and pharmacological correction of a membrane trafficking defect associated with a mutation in the sulfonylurea receptor causing familial hyperinsulinism.
    Partridge CJ, Beech DJ, Sivaprasadarao A.
    J Biol Chem; 2001 Sep 21; 276(38):35947-52. PubMed ID: 11457841
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  • 19. Diabetes and hypoglycaemia in young children and mutations in the Kir6.2 subunit of the potassium channel: therapeutic consequences.
    Flechtner I, de Lonlay P, Polak M.
    Diabetes Metab; 2006 Dec 21; 32(6):569-80. PubMed ID: 17296510
    [Abstract] [Full Text] [Related]

  • 20. Identification of a familial hyperinsulinism-causing mutation in the sulfonylurea receptor 1 that prevents normal trafficking and function of KATP channels.
    Taschenberger G, Mougey A, Shen S, Lester LB, LaFranchi S, Shyng SL.
    J Biol Chem; 2002 May 10; 277(19):17139-46. PubMed ID: 11867634
    [Abstract] [Full Text] [Related]

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