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312 related items for PubMed ID: 10766872

  • 1. Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c.
    Shimizu S, Ide T, Yanagida T, Tsujimoto Y.
    J Biol Chem; 2000 Apr 21; 275(16):12321-5. PubMed ID: 10766872
    [Abstract] [Full Text] [Related]

  • 2. Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC.
    Shimizu S, Narita M, Tsujimoto Y.
    Nature; 1999 Jun 03; 399(6735):483-7. PubMed ID: 10365962
    [Abstract] [Full Text] [Related]

  • 3. Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells.
    Shimizu S, Matsuoka Y, Shinohara Y, Yoneda Y, Tsujimoto Y.
    J Cell Biol; 2001 Jan 22; 152(2):237-50. PubMed ID: 11266442
    [Abstract] [Full Text] [Related]

  • 4. Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator.
    Shimizu S, Shinohara Y, Tsujimoto Y.
    Oncogene; 2000 Sep 07; 19(38):4309-18. PubMed ID: 10980606
    [Abstract] [Full Text] [Related]

  • 5. BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death.
    Shimizu S, Konishi A, Kodama T, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 2000 Mar 28; 97(7):3100-5. PubMed ID: 10737788
    [Abstract] [Full Text] [Related]

  • 6. Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity.
    Shimizu S, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 2000 Jan 18; 97(2):577-82. PubMed ID: 10639121
    [Abstract] [Full Text] [Related]

  • 7. The voltage-dependent anion channel: an essential player in apoptosis.
    Tsujimoto Y, Shimizu S.
    Biochimie; 2002 Jan 18; 84(2-3):187-93. PubMed ID: 12022949
    [Abstract] [Full Text] [Related]

  • 8. Investigation of bax-induced release of cytochrome c from yeast mitochondria permeability of mitochondrial membranes, role of VDAC and ATP requirement.
    Priault M, Chaudhuri B, Clow A, Camougrand N, Manon S.
    Eur J Biochem; 1999 Mar 18; 260(3):684-91. PubMed ID: 10102996
    [Abstract] [Full Text] [Related]

  • 9. Bid, but not Bax, regulates VDAC channels.
    Rostovtseva TK, Antonsson B, Suzuki M, Youle RJ, Colombini M, Bezrukov SM.
    J Biol Chem; 2004 Apr 02; 279(14):13575-83. PubMed ID: 14729675
    [Abstract] [Full Text] [Related]

  • 10. Activation of mitochondrial voltage-dependent anion channel by apro-apoptotic BH3-only protein Bim.
    Sugiyama T, Shimizu S, Matsuoka Y, Yoneda Y, Tsujimoto Y.
    Oncogene; 2002 Jul 25; 21(32):4944-56. PubMed ID: 12118373
    [Abstract] [Full Text] [Related]

  • 11. VDAC regulation by the Bcl-2 family of proteins.
    Tsujimoto Y, Shimizu S.
    Cell Death Differ; 2000 Dec 25; 7(12):1174-81. PubMed ID: 11175254
    [Abstract] [Full Text] [Related]

  • 12. The voltage-dependent anion channel (VDAC): function in intracellular signalling, cell life and cell death.
    Shoshan-Barmatz V, Israelson A, Brdiczka D, Sheu SS.
    Curr Pharm Des; 2006 Dec 25; 12(18):2249-70. PubMed ID: 16787253
    [Abstract] [Full Text] [Related]

  • 13. Bcl-2 family of proteins: life-or-death switch in mitochondria.
    Tsujimoto Y.
    Biosci Rep; 2002 Feb 25; 22(1):47-58. PubMed ID: 12418550
    [Abstract] [Full Text] [Related]

  • 14. Mouse uterine epithelial apoptosis is associated with expression of mitochondrial voltage-dependent anion channels, release of cytochrome C from mitochondria, and the ratio of Bax to Bcl-2 or Bcl-X.
    Takagi-Morishita Y, Yamada N, Sugihara A, Iwasaki T, Tsujimura T, Terada N.
    Biol Reprod; 2003 Apr 25; 68(4):1178-84. PubMed ID: 12606449
    [Abstract] [Full Text] [Related]

  • 15. Bax increases the pore size of rat brain mitochondrial voltage-dependent anion channel in the presence of tBid.
    Banerjee J, Ghosh S.
    Biochem Biophys Res Commun; 2004 Oct 08; 323(1):310-4. PubMed ID: 15351738
    [Abstract] [Full Text] [Related]

  • 16. Identification of the protein-protein contact site and interaction mode of human VDAC1 with Bcl-2 family proteins.
    Shi Y, Chen J, Weng C, Chen R, Zheng Y, Chen Q, Tang H.
    Biochem Biophys Res Commun; 2003 Jun 13; 305(4):989-96. PubMed ID: 12767928
    [Abstract] [Full Text] [Related]

  • 17. Essential role of the voltage-dependent anion channel (VDAC) in mitochondrial permeability transition pore opening and cytochrome c release induced by arsenic trioxide.
    Zheng Y, Shi Y, Tian C, Jiang C, Jin H, Chen J, Almasan A, Tang H, Chen Q.
    Oncogene; 2004 Feb 12; 23(6):1239-47. PubMed ID: 14647451
    [Abstract] [Full Text] [Related]

  • 18. The function of complexes between the outer mitochondrial membrane pore (VDAC) and the adenine nucleotide translocase in regulation of energy metabolism and apoptosis.
    Vyssokikh MY, Brdiczka D.
    Acta Biochim Pol; 2003 Feb 12; 50(2):389-404. PubMed ID: 12833165
    [Abstract] [Full Text] [Related]

  • 19. VDAC-dependent permeabilization of the outer mitochondrial membrane by superoxide induces rapid and massive cytochrome c release.
    Madesh M, Hajnóczky G.
    J Cell Biol; 2001 Dec 10; 155(6):1003-15. PubMed ID: 11739410
    [Abstract] [Full Text] [Related]

  • 20. Bcl-xL promotes the open configuration of the voltage-dependent anion channel and metabolite passage through the outer mitochondrial membrane.
    Vander Heiden MG, Li XX, Gottleib E, Hill RB, Thompson CB, Colombini M.
    J Biol Chem; 2001 Jun 01; 276(22):19414-9. PubMed ID: 11259441
    [Abstract] [Full Text] [Related]

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