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191 related items for PubMed ID: 1083555

  • 1. Genetic control of B-cell responses. II. Identification of the spleen B-cell defect in C3H/HeJ mice.
    Coutinho A.
    Scand J Immunol; 1976; 5(1-2):129-40. PubMed ID: 1083555
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  • 2. Immunologic properties of bacterial lipopolysaccharide (LPS). IV. Cellular basis of the unresponsiveness of C3H/HeJ mouse spleen cells to LPS-induced mitogenesis.
    Skidmore BJ, Chiller JM, Weigle WO.
    J Immunol; 1977 Jan; 118(1):274-81. PubMed ID: 299760
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  • 3. Genetic control of lymphocyte activation: lack of response to low doses of concanavalin A in lipopolysaccharide-nonresponder mice.
    Bick PH, Persson U, Smith E, Möller E, Hammarström L.
    J Exp Med; 1977 Oct 01; 146(4):1146-51. PubMed ID: 330792
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  • 4. LPS regulation of the immune response: Bacteroides endotoxin induces mitogenic, polyclonal, and antibody responses in classical LPS responsive but not C3H/HeJ mice.
    Wannemuehler MJ, Michalek SM, Jirillo E, Williamson SI, Hirasawa M, McGhee JR.
    J Immunol; 1984 Jul 01; 133(1):299-305. PubMed ID: 6202784
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  • 5. LPS regulation of the immune response: separate mechanisms for murine B cell activation by lipid A (direct) and polysaccharide (macrophage-dependent) derived from Bacteroides LPS.
    Williamson SI, Wannemuehler MJ, Jirillo E, Pritchard DG, Michalek SM, McGhee JR.
    J Immunol; 1984 Nov 01; 133(5):2294-300. PubMed ID: 6332842
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  • 6. Partial restoration of the lipopolysaccharide-induced proliferative response in splenic B cells from C3H/HeJ mice.
    Kuus-Reichel K, Ulevitch RJ.
    J Immunol; 1986 Jul 15; 137(2):472-7. PubMed ID: 3487572
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  • 7. Immunologic responsiveness of the C3H/HeJ mouse: differential ability of butanol-extracted lipopolysaccharide (LPS) to evoke LPS-mediated effects.
    Goodman MG, Parks DE, Weigle WO.
    J Exp Med; 1978 Mar 01; 147(3):800-13. PubMed ID: 75941
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  • 8. Functional nuclei of LPS-nonresponder C3H/HeJ mice after transfer into LPS-responder C3H/HeN cells by cell fusion.
    Watanabe T, Ohara J.
    Nature; 1981 Mar 05; 290(5801):58-9. PubMed ID: 7207585
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  • 13. Genetic control of responses to bacterial lipopolysaccharides in mice. II. A gene that influences a membrane component involved in the activation of bone marrow-derived lymphocytes by lipipolysaccharides.
    Watson J, Riblet R.
    J Immunol; 1975 May 05; 114(5):1462-8. PubMed ID: 123543
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  • 14. Electrokinetic properties of splenic lymphocytes from the low-lipopolysaccharide responder C3H/Hej mice.
    Dumont F, Barrois R.
    Folia Biol (Praha); 1976 May 05; 12(3):145-50. PubMed ID: 1086804
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  • 16. Genetic control of responses to bacterial lipopolysaccharides in mice. I. Evidence for a single gene that influences mitogenic and immunogenic respones to lipopolysaccharides.
    Watson J, Riblet R.
    J Exp Med; 1974 Nov 01; 140(5):1147-61. PubMed ID: 4138849
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  • 17. Lipopolysaccharide nonresponder cells: the C3H/HeJ defect.
    Sultzer BM, Castagna R, Bandekar J, Wong P.
    Immunobiology; 1993 Apr 01; 187(3-5):257-71. PubMed ID: 8330899
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  • 18. Polyclonal activation of murine B lymphocytes by Fc fragments. III. Characterization of the defect in the ability of the C3H/HeJ mouse to respond to Fc fragments.
    Morgan EL, Weigle WO.
    J Immunol; 1980 Dec 01; 125(6):2467-72. PubMed ID: 7000901
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  • 19. Genetical control of B-cell responses. IV. Inheritance of the unresponsiveness to lipopolysaccharides.
    Coutinho A, Moller G, Gronowicz E.
    J Exp Med; 1975 Jul 01; 142(1):253-8. PubMed ID: 1097575
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  • 20. Restoration of LPS responsiveness of C3H/HeJ mouse lymphocytes by microinjection of cytoplasmic factor(s) from LPS-stimulated normal lymphocytes.
    Eda Y, Ohara J, Watanabe T.
    J Immunol; 1983 Sep 01; 131(3):1294-9. PubMed ID: 6604094
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