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119 related items for PubMed ID: 10851065

  • 1. Oncogenic transformation by ras and fos is mediated by c-Jun N-terminal phosphorylation.
    Behrens A, Jochum W, Sibilia M, Wagner EF.
    Oncogene; 2000 May 18; 19(22):2657-63. PubMed ID: 10851065
    [Abstract] [Full Text] [Related]

  • 2. Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation.
    Behrens A, Sibilia M, Wagner EF.
    Nat Genet; 1999 Mar 18; 21(3):326-9. PubMed ID: 10080190
    [Abstract] [Full Text] [Related]

  • 3. Suppression of Ras-stimulated transformation by the JNK signal transduction pathway.
    Kennedy NJ, Sluss HK, Jones SN, Bar-Sagi D, Flavell RA, Davis RJ.
    Genes Dev; 2003 Mar 01; 17(5):629-37. PubMed ID: 12629045
    [Abstract] [Full Text] [Related]

  • 4. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.
    Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD.
    Oncogene; 2000 Sep 28; 19(41):4685-94. PubMed ID: 11032018
    [Abstract] [Full Text] [Related]

  • 5. A peptide fragment of ependymin neurotrophic factor uses protein kinase C and the mitogen-activated protein kinase pathway to activate c-Jun N-terminal kinase and a functional AP-1 containing c-Jun and c-Fos proteins in mouse NB2a cells.
    Adams DS, Hasson B, Boyer-Boiteau A, El-Khishin A, Shashoua VE.
    J Neurosci Res; 2003 May 01; 72(3):405-16. PubMed ID: 12692907
    [Abstract] [Full Text] [Related]

  • 6. Influence of the glucocorticoid receptor on c-fos inducibility in activated ras-containing mouse lung cells.
    Johnson TA, Li J, Beer DG.
    Mol Carcinog; 1996 Oct 01; 17(2):70-7. PubMed ID: 8890955
    [Abstract] [Full Text] [Related]

  • 7. Suppression of malignant growth potentials of v-Src-transformed human gallbladder epithelial cells by adenovirus-mediated dominant negative H-Ras.
    Tokumitsu Y, Nakano S, Ueno H, Niho Y.
    J Cell Physiol; 2000 May 01; 183(2):221-7. PubMed ID: 10737897
    [Abstract] [Full Text] [Related]

  • 8. Oncogenic and transcriptional cooperation with Ha-Ras requires phosphorylation of c-Jun on serines 63 and 73.
    Smeal T, Binetruy B, Mercola DA, Birrer M, Karin M.
    Nature; 1991 Dec 12; 354(6353):494-6. PubMed ID: 1749429
    [Abstract] [Full Text] [Related]

  • 9. Downregulation of c-fos gene transcription in cells transformed by E1A and cHa-ras oncogenes: a role of sustained activation of MAP/ERK kinase cascade and of inactive chromatin structure at c-fos promoter.
    Kukushkin AN, Abramova MV, Svetlikova SB, Darieva ZA, Pospelova TV, Pospelov VA.
    Oncogene; 2002 Jan 24; 21(5):719-30. PubMed ID: 11850800
    [Abstract] [Full Text] [Related]

  • 10. High levels of phosphorylated c-Jun, Fra-1, Fra-2 and ATF-2 proteins correlate with malignant phenotypes in the multistage mouse skin carcinogenesis model.
    Zoumpourlis V, Papassava P, Linardopoulos S, Gillespie D, Balmain A, Pintzas A.
    Oncogene; 2000 Aug 17; 19(35):4011-21. PubMed ID: 10962557
    [Abstract] [Full Text] [Related]

  • 11. Transformation suppressor activity of a Jun transcription factor lacking its activation domain.
    Lloyd A, Yancheva N, Wasylyk B.
    Nature; 1991 Aug 15; 352(6336):635-8. PubMed ID: 1907719
    [Abstract] [Full Text] [Related]

  • 12. AP-1 mediated retinal photoreceptor apoptosis is independent of N-terminal phosphorylation of c-Jun.
    Grimm C, Wenzel A, Behrens A, Hafezi F, Wagner EF, Remé CE.
    Cell Death Differ; 2001 Aug 15; 8(8):859-67. PubMed ID: 11526439
    [Abstract] [Full Text] [Related]

  • 13. c-Jun NH2-terminal kinase 2 is required for Ras transformation independently of activator protein 1.
    Nielsen C, Thastrup J, Bøttzauw T, Jäättelä M, Kallunki T.
    Cancer Res; 2007 Jan 01; 67(1):178-85. PubMed ID: 17210697
    [Abstract] [Full Text] [Related]

  • 14. Endothelin-1-induced cardiac hypertrophy is inhibited by activation of peroxisome proliferator-activated receptor-alpha partly via blockade of c-Jun NH2-terminal kinase pathway.
    Irukayama-Tomobe Y, Miyauchi T, Sakai S, Kasuya Y, Ogata T, Takanashi M, Iemitsu M, Sudo T, Goto K, Yamaguchi I.
    Circulation; 2004 Feb 24; 109(7):904-10. PubMed ID: 14967736
    [Abstract] [Full Text] [Related]

  • 15. Amino-terminal phosphorylation of c-Jun regulates apoptosis in the retinal ganglion cells by optic nerve transection.
    Yoshida K, Behrens A, Le-Niculescu H, Wagner EF, Harada T, Imaki J, Ohno S, Karin M.
    Invest Ophthalmol Vis Sci; 2002 May 24; 43(5):1631-5. PubMed ID: 11980884
    [Abstract] [Full Text] [Related]

  • 16. Inhibition of Fos- and Ras-induced transformation by mutant Fos proteins with structural alterations in functionally different domains.
    Wick M, Lucibello FC, Müller R.
    Oncogene; 1992 May 24; 7(5):859-67. PubMed ID: 1570155
    [Abstract] [Full Text] [Related]

  • 17. Anisomycin abrogates repression of protooncogene c-fos transcription in E1A + cHa-ras-transformed cells through activation of MEK/ERK kinase cascade.
    Kukushkin AN, Svetlikova SB, Amanzholov RA, Pospelov VA.
    J Cell Biochem; 2008 Feb 15; 103(3):1005-12. PubMed ID: 17647273
    [Abstract] [Full Text] [Related]

  • 18. Interaction of phosphorylated c-Jun with TCF4 regulates intestinal cancer development.
    Nateri AS, Spencer-Dene B, Behrens A.
    Nature; 2005 Sep 08; 437(7056):281-5. PubMed ID: 16007074
    [Abstract] [Full Text] [Related]

  • 19. The c-Jun NH(2)-terminal protein kinase/AP-1 pathway is required for efficient apoptosis induced by vinblastine.
    Fan M, Goodwin ME, Birrer MJ, Chambers TC.
    Cancer Res; 2001 Jun 01; 61(11):4450-8. PubMed ID: 11389075
    [Abstract] [Full Text] [Related]

  • 20. The defective transforming phenotype of c-Jun Ala(63/73) is rescued by mutation of the C-terminal phosphorylation site.
    Bost F, Caron L, Vial E, Montreau N, Marchetti I, Dejong V, Defize L, Castellazzi M, Binétruy B.
    Oncogene; 2001 Nov 01; 20(50):7425-9. PubMed ID: 11704873
    [Abstract] [Full Text] [Related]

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