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Journal Abstract Search

284 related items for PubMed ID: 10880455

  • 1. Possible association of BLM in decreasing DNA double strand breaks during DNA replication.
    Wang W, Seki M, Narita Y, Sonoda E, Takeda S, Yamada K, Masuko T, Katada T, Enomoto T.
    EMBO J; 2000 Jul 03; 19(13):3428-35. PubMed ID: 10880455
    [Abstract] [Full Text] [Related]

  • 2. Werner and Bloom helicases are involved in DNA repair in a complementary fashion.
    Imamura O, Fujita K, Itoh C, Takeda S, Furuichi Y, Matsumoto T.
    Oncogene; 2002 Jan 31; 21(6):954-63. PubMed ID: 11840341
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  • 3. Bloom helicase is involved in DNA surveillance in early S phase in vertebrate cells.
    Imamura O, Fujita K, Shimamoto A, Tanabe H, Takeda S, Furuichi Y, Matsumoto T.
    Oncogene; 2001 Mar 08; 20(10):1143-51. PubMed ID: 11313858
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  • 4. BLM helicase-dependent transport of p53 to sites of stalled DNA replication forks modulates homologous recombination.
    Sengupta S, Linke SP, Pedeux R, Yang Q, Farnsworth J, Garfield SH, Valerie K, Shay JW, Ellis NA, Wasylyk B, Harris CC.
    EMBO J; 2003 Mar 03; 22(5):1210-22. PubMed ID: 12606585
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  • 5. Role of the BLM helicase in replication fork management.
    Wu L.
    DNA Repair (Amst); 2007 Jul 01; 6(7):936-44. PubMed ID: 17363339
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  • 7. Bloom's syndrome protein is required for correct relocalization of RAD50/MRE11/NBS1 complex after replication fork arrest.
    Franchitto A, Pichierri P.
    J Cell Biol; 2002 Apr 01; 157(1):19-30. PubMed ID: 11916980
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  • 8. BLM has early and late functions in homologous recombination repair in mouse embryonic stem cells.
    Chu WK, Hanada K, Kanaar R, Hickson ID.
    Oncogene; 2010 Aug 19; 29(33):4705-14. PubMed ID: 20531307
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  • 9. Point mutations causing Bloom's syndrome abolish ATPase and DNA helicase activities of the BLM protein.
    Bahr A, De Graeve F, Kedinger C, Chatton B.
    Oncogene; 1998 Nov 19; 17(20):2565-71. PubMed ID: 9840919
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  • 12. BLM (the causative gene of Bloom syndrome) protein translocation into the nucleus by a nuclear localization signal.
    Kaneko H, Orii KO, Matsui E, Shimozawa N, Fukao T, Matsumoto T, Shimamoto A, Furuichi Y, Hayakawa S, Kasahara K, Kondo N.
    Biochem Biophys Res Commun; 1997 Nov 17; 240(2):348-53. PubMed ID: 9388480
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  • 14. The Bloom's syndrome helicase interacts directly with the human DNA mismatch repair protein hMSH6.
    Pedrazzi G, Bachrati CZ, Selak N, Studer I, Petkovic M, Hickson ID, Jiricny J, Stagljar I.
    Biol Chem; 2003 Aug 17; 384(8):1155-64. PubMed ID: 12974384
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  • 15. Functions of RecQ family helicases: possible involvement of Bloom's and Werner's syndrome gene products in guarding genome integrity during DNA replication.
    Enomoto T.
    J Biochem; 2001 Apr 17; 129(4):501-7. PubMed ID: 11275547
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  • 16. [Bloom syndrome].
    Kondo N.
    Nihon Rinsho; 2000 Jul 17; 58(7):1460-6. PubMed ID: 10921324
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  • 17. Bloom syndrome, genomic instability and cancer: the SOS-like hypothesis.
    Amor-Guéret M.
    Cancer Lett; 2006 May 08; 236(1):1-12. PubMed ID: 15950375
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  • 18. Cooperative roles of vertebrate Fbh1 and Blm DNA helicases in avoidance of crossovers during recombination initiated by replication fork collapse.
    Kohzaki M, Hatanaka A, Sonoda E, Yamazoe M, Kikuchi K, Vu Trung N, Szüts D, Sale JE, Shinagawa H, Watanabe M, Takeda S.
    Mol Cell Biol; 2007 Apr 08; 27(8):2812-20. PubMed ID: 17283053
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  • 19. Phosphorylation of BLM, dissociation from topoisomerase IIIalpha, and colocalization with gamma-H2AX after topoisomerase I-induced replication damage.
    Rao VA, Fan AM, Meng L, Doe CF, North PS, Hickson ID, Pommier Y.
    Mol Cell Biol; 2005 Oct 08; 25(20):8925-37. PubMed ID: 16199871
    [Abstract] [Full Text] [Related]

  • 20. Functional interactions between BLM and XRCC3 in the cell.
    Otsuki M, Seki M, Inoue E, Yoshimura A, Kato G, Yamanouchi S, Kawabe Y, Tada S, Shinohara A, Komura J, Ono T, Takeda S, Ishii Y, Enomoto T.
    J Cell Biol; 2007 Oct 08; 179(1):53-63. PubMed ID: 17923529
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