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Journal Abstract Search

73 related items for PubMed ID: 10903887

  • 1. Suppression of IkappaBalpha expression is necessary for c-Jun N-terminal kinase-mediated enhancement of Fas cytotoxicity.
    Chang NS, Schultz L, Heath J.
    Biochem Biophys Res Commun; 2000 Jul 21; 274(1):4-10. PubMed ID: 10903887
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  • 2. TGF-beta-induced matrix proteins inhibit p42/44 MAPK and JNK activation and suppress TNF-mediated IkappaBalpha degradation and NF-kappaB nuclear translocation in L929 fibroblasts.
    Chang NS.
    Biochem Biophys Res Commun; 2000 Jan 07; 267(1):194-200. PubMed ID: 10623598
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  • 3. IkappaBalpha is essential for maintaining basal c-Jun N-terminal kinase (JNK) activation and regulating JNK-mediated resistance to tumor necrosis factor cytotoxicity in L929 cells.
    Chang NS.
    Biochem Biophys Res Commun; 1999 Sep 16; 263(1):107-12. PubMed ID: 10486261
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  • 4. Arsenic inhibits NF-kappaB-mediated gene transcription by blocking IkappaB kinase activity and IkappaBalpha phosphorylation and degradation.
    Roussel RR, Barchowsky A.
    Arch Biochem Biophys; 2000 May 01; 377(1):204-12. PubMed ID: 10775461
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  • 5. Non-apoptotic signaling pathways activated by soluble Fas ligand in serum-starved human fibroblasts. Mitogen-activated protein kinases and NF-kappaB-dependent gene expression.
    Ahn JH, Park SM, Cho HS, Lee MS, Yoon JB, Vilcek J, Lee TH.
    J Biol Chem; 2001 Dec 14; 276(50):47100-6. PubMed ID: 11600497
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  • 6. Genetic deletion of PKR abrogates TNF-induced activation of IkappaBalpha kinase, JNK, Akt and cell proliferation but potentiates p44/p42 MAPK and p38 MAPK activation.
    Takada Y, Ichikawa H, Pataer A, Swisher S, Aggarwal BB.
    Oncogene; 2007 Feb 22; 26(8):1201-12. PubMed ID: 16924232
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  • 7. Metabolic inhibitors sensitize for CD95 (APO-1/Fas)-induced apoptosis by down-regulating Fas-associated death domain-like interleukin 1-converting enzyme inhibitory protein expression.
    Fulda S, Meyer E, Debatin KM.
    Cancer Res; 2000 Jul 15; 60(14):3947-56. PubMed ID: 10919673
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  • 9. Oleandrin suppresses activation of nuclear transcription factor-kappaB, activator protein-1, and c-Jun NH2-terminal kinase.
    Manna SK, Sah NK, Newman RA, Cisneros A, Aggarwal BB.
    Cancer Res; 2000 Jul 15; 60(14):3838-47. PubMed ID: 10919658
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  • 11. Protein tyrosine kinase inhibitors block tumor necrosis factor-induced activation of nuclear factor-kappaB, degradation of IkappaBalpha, nuclear translocation of p65, and subsequent gene expression.
    Natarajan K, Manna SK, Chaturvedi MM, Aggarwal BB.
    Arch Biochem Biophys; 1998 Apr 01; 352(1):59-70. PubMed ID: 9521814
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  • 13. Pyrrolidine dithiocarbamate-induced neuronal cell death is mediated by Akt, casein kinase 2, c-Jun N-terminal kinase, and IkappaB kinase in embryonic hippocampal progenitor cells.
    Min YK, Park JH, Chong SA, Kim YS, Ahn YS, Seo JT, Bae YS, Chung KC.
    J Neurosci Res; 2003 Mar 01; 71(5):689-700. PubMed ID: 12584727
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  • 15. Involvement of p42/p44 MAPK, JNK, and NF-kappaB in IL-1beta-induced ICAM-1 expression in human pulmonary epithelial cells.
    Lin FS, Lin CC, Chien CS, Luo SF, Yang CM.
    J Cell Physiol; 2005 Feb 01; 202(2):464-73. PubMed ID: 15389584
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  • 17. MAP kinase-dependent, NF-kappaB-independent regulation of inhibitor of apoptosis protein genes by TNF-alpha.
    Furusu A, Nakayama K, Xu Q, Konta T, Kitamura M.
    J Cell Physiol; 2007 Mar 01; 210(3):703-10. PubMed ID: 17133355
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