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Journal Abstract Search

107 related items for PubMed ID: 1093185

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  • 3. Metabolic requirements for the development of hemadsorption activity and virus formation in BHK-21 cells infected with Newcastle disease virus.
    Nagai Y.
    J Virol; 1973 Apr; 11(4):479-86. PubMed ID: 4735590
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  • 5. Newcastle Disease Virus Establishes Persistent Infection in Tumor Cells In Vitro: Contribution of the Cleavage Site of Fusion Protein and Second Sialic Acid Binding Site of Hemagglutinin-Neuraminidase.
    Rangaswamy US, Wang W, Cheng X, McTamney P, Carroll D, Jin H.
    J Virol; 2017 Aug 15; 91(16):. PubMed ID: 28592535
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  • 6. Virus-receptor interactions of human parainfluenza viruses types 1, 2 and 3.
    Ah-Tye C, Schwartz S, Huberman K, Carlin E, Moscona A.
    Microb Pathog; 1999 Nov 15; 27(5):329-36. PubMed ID: 10545258
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  • 8. Roles of the highly conserved amino acids in the globular head and stalk region of the Newcastle disease virus HN protein in the membrane fusion process.
    Sun C, Wen H, Chen Y, Chu F, Lin B, Ren G, Song Y, Wang Z.
    Biosci Trends; 2015 Feb 15; 9(1):56-64. PubMed ID: 25787910
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  • 9. Avian cells expressing the Newcastle disease virus hemagglutinin-neuraminidase protein are resistant to Newcastle disease virus infection.
    Morrison TG, McGinnes LW.
    Virology; 1989 Jul 15; 171(1):10-7. PubMed ID: 2545025
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  • 12. Selective gene transfer to tumor cells by recombinant Newcastle Disease Virus via a bispecific fusion protein.
    Bian H, Fournier P, Moormann R, Peeters B, Schirrmacher V.
    Int J Oncol; 2005 Feb 15; 26(2):431-9. PubMed ID: 15645128
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  • 13. Modification of normal cell surface by smooth membrane preparations from BHK-21 cells infected with Newcastle disease virus.
    Nagai Y, Yoshida T, Yoshii S, Maeno K, Matsumoto T.
    Med Microbiol Immunol; 1975 Jul 02; 161(3):175-88. PubMed ID: 1101009
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  • 14. Differences in surface sialic acid and galactosyl residues of two autologous human melanoma cell lines.
    Reynier M, Aubery M, Lebec S, Vernay M, Aubert C.
    Med Oncol Tumor Pharmacother; 1984 Jul 02; 1(3):157-61. PubMed ID: 6544896
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  • 15. Neuraminidase is essential for fowl plague virus hemagglutinin to show hemagglutinating activity.
    Ohuchi M, Feldmann A, Ohuchi R, Klenk HD.
    Virology; 1995 Sep 10; 212(1):77-83. PubMed ID: 7676651
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  • 16. Sialic acid is cleaved from glycoconjugates at the cell surface when influenza virus neuraminidases are expressed from recombinant vaccinia viruses.
    Els MC, Laver WG, Air GM.
    Virology; 1989 May 10; 170(1):346-51. PubMed ID: 2718386
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  • 17. Surface modifications evoked by antidiuretic hormone in isolated epithelial cells: evidence from lectin probes.
    Pietras RJ, Naujokaitis PJ, Szego CM.
    J Supramol Struct; 1975 May 10; 3(5-6):391-400. PubMed ID: 813065
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  • 18. Inhibition of parainfluenza virus type 3 and Newcastle disease virus hemagglutinin-neuraminidase receptor binding: effect of receptor avidity and steric hindrance at the inhibitor binding sites.
    Porotto M, Murrell M, Greengard O, Lawrence MC, McKimm-Breschkin JL, Moscona A.
    J Virol; 2004 Dec 10; 78(24):13911-9. PubMed ID: 15564499
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  • 19. Second sialic acid binding site in Newcastle disease virus hemagglutinin-neuraminidase: implications for fusion.
    Zaitsev V, von Itzstein M, Groves D, Kiefel M, Takimoto T, Portner A, Taylor G.
    J Virol; 2004 Apr 10; 78(7):3733-41. PubMed ID: 15016893
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  • 20. Neuraminidase hemadsorption activity, conserved in avian influenza A viruses, does not influence viral replication in ducks.
    Kobasa D, Rodgers ME, Wells K, Kawaoka Y.
    J Virol; 1997 Sep 10; 71(9):6706-13. PubMed ID: 9261394
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