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242 related items for PubMed ID: 10958676

  • 1. Role of the LXCXE binding site in Rb function.
    Dahiya A, Gavin MR, Luo RX, Dean DC.
    Mol Cell Biol; 2000 Sep; 20(18):6799-805. PubMed ID: 10958676
    [Abstract] [Full Text] [Related]

  • 2. Mutagenesis of the pRB pocket reveals that cell cycle arrest functions are separable from binding to viral oncoproteins.
    Dick FA, Sailhamer E, Dyson NJ.
    Mol Cell Biol; 2000 May; 20(10):3715-27. PubMed ID: 10779361
    [Abstract] [Full Text] [Related]

  • 3. Establishment of irreversible growth arrest in myogenic differentiation requires the RB LXCXE-binding function.
    Chen TT, Wang JY.
    Mol Cell Biol; 2000 Aug; 20(15):5571-80. PubMed ID: 10891495
    [Abstract] [Full Text] [Related]

  • 4. Retinoblastoma protein recruits histone deacetylase to repress transcription.
    Brehm A, Miska EA, McCance DJ, Reid JL, Bannister AJ, Kouzarides T.
    Nature; 1998 Feb 05; 391(6667):597-601. PubMed ID: 9468139
    [Abstract] [Full Text] [Related]

  • 5. Growth suppression by an E2F-binding-defective retinoblastoma protein (RB): contribution from the RB C pocket.
    Whitaker LL, Su H, Baskaran R, Knudsen ES, Wang JY.
    Mol Cell Biol; 1998 Jul 05; 18(7):4032-42. PubMed ID: 9632788
    [Abstract] [Full Text] [Related]

  • 6. E2F-Rb complexes assemble and inhibit cdc25A transcription in cervical carcinoma cells following repression of human papillomavirus oncogene expression.
    Wu L, Goodwin EC, Naeger LK, Vigo E, Galaktionov K, Helin K, DiMaio D.
    Mol Cell Biol; 2000 Oct 05; 20(19):7059-67. PubMed ID: 10982822
    [Abstract] [Full Text] [Related]

  • 7. Inhibition of DNA synthesis by RB: effects on G1/S transition and S-phase progression.
    Knudsen ES, Buckmaster C, Chen TT, Feramisco JR, Wang JY.
    Genes Dev; 1998 Aug 01; 12(15):2278-92. PubMed ID: 9694794
    [Abstract] [Full Text] [Related]

  • 8. The three members of the pocket proteins family share the ability to repress E2F activity through recruitment of a histone deacetylase.
    Ferreira R, Magnaghi-Jaulin L, Robin P, Harel-Bellan A, Trouche D.
    Proc Natl Acad Sci U S A; 1998 Sep 01; 95(18):10493-8. PubMed ID: 9724731
    [Abstract] [Full Text] [Related]

  • 9. Retinoblastoma protein represses transcription by recruiting a histone deacetylase.
    Magnaghi-Jaulin L, Groisman R, Naguibneva I, Robin P, Lorain S, Le Villain JP, Troalen F, Trouche D, Harel-Bellan A.
    Nature; 1998 Feb 05; 391(6667):601-5. PubMed ID: 9468140
    [Abstract] [Full Text] [Related]

  • 10. Destabilization of the retinoblastoma tumor suppressor by human papillomavirus type 16 E7 is not sufficient to overcome cell cycle arrest in human keratinocytes.
    Helt AM, Galloway DA.
    J Virol; 2001 Aug 05; 75(15):6737-47. PubMed ID: 11435552
    [Abstract] [Full Text] [Related]

  • 11. Role of LXCXE motif-dependent interactions in the activity of the retinoblastoma protein.
    Chan HM, Smith L, La Thangue NB.
    Oncogene; 2001 Sep 27; 20(43):6152-63. PubMed ID: 11593423
    [Abstract] [Full Text] [Related]

  • 12. Human SWI-SNF component BRG1 represses transcription of the c-fos gene.
    Murphy DJ, Hardy S, Engel DA.
    Mol Cell Biol; 1999 Apr 27; 19(4):2724-33. PubMed ID: 10082538
    [Abstract] [Full Text] [Related]

  • 13. Retinoblastoma-repression of E2F-dependent transcription depends on the ability of the retinoblastoma protein to interact with E2F and is abrogated by the adenovirus E1A oncoprotein.
    Arroyo M, Raychaudhuri P.
    Nucleic Acids Res; 1992 Nov 25; 20(22):5947-54. PubMed ID: 1461728
    [Abstract] [Full Text] [Related]

  • 14. The large subunit of replication factor C promotes cell survival after DNA damage in an LxCxE motif- and Rb-dependent manner.
    Pennaneach V, Salles-Passador I, Munshi A, Brickner H, Regazzoni K, Dick F, Dyson N, Chen TT, Wang JY, Fotedar R, Fotedar A.
    Mol Cell; 2001 Apr 25; 7(4):715-27. PubMed ID: 11336696
    [Abstract] [Full Text] [Related]

  • 15. E2F4-RB and E2F4-p107 complexes suppress gene expression by transforming growth factor beta through E2F binding sites.
    Li JM, Hu PP, Shen X, Yu Y, Wang XF.
    Proc Natl Acad Sci U S A; 1997 May 13; 94(10):4948-53. PubMed ID: 9144170
    [Abstract] [Full Text] [Related]

  • 16. E1A 12S and 13S of the transformation-defective adenovirus type 12 strain CS-1 inactivate proteins of the RB family, permitting transactivation of the E2F-dependent promoter.
    Pützer BM, Rumpf H, Rega S, Brockmann D, Esche H.
    J Virol; 1997 Dec 13; 71(12):9538-48. PubMed ID: 9371617
    [Abstract] [Full Text] [Related]

  • 17. Che-1 affects cell growth by interfering with the recruitment of HDAC1 by Rb.
    Bruno T, De Angelis R, De Nicola F, Barbato C, Di Padova M, Corbi N, Libri V, Benassi B, Mattei E, Chersi A, Soddu S, Floridi A, Passananti C, Fanciulli M.
    Cancer Cell; 2002 Nov 13; 2(5):387-99. PubMed ID: 12450794
    [Abstract] [Full Text] [Related]

  • 18. The histone deacetylase HDAC3 targets RbAp48 to the retinoblastoma protein.
    Nicolas E, Ait-Si-Ali S, Trouche D.
    Nucleic Acids Res; 2001 Aug 01; 29(15):3131-6. PubMed ID: 11470869
    [Abstract] [Full Text] [Related]

  • 19. RbAp48 belongs to the histone deacetylase complex that associates with the retinoblastoma protein.
    Nicolas E, Morales V, Magnaghi-Jaulin L, Harel-Bellan A, Richard-Foy H, Trouche D.
    J Biol Chem; 2000 Mar 31; 275(13):9797-804. PubMed ID: 10734134
    [Abstract] [Full Text] [Related]

  • 20. Exit from G1 and S phase of the cell cycle is regulated by repressor complexes containing HDAC-Rb-hSWI/SNF and Rb-hSWI/SNF.
    Zhang HS, Gavin M, Dahiya A, Postigo AA, Ma D, Luo RX, Harbour JW, Dean DC.
    Cell; 2000 Mar 31; 101(1):79-89. PubMed ID: 10778858
    [Abstract] [Full Text] [Related]

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