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258 related items for PubMed ID: 10963543

  • 1. Endotoxin tolerance from lipopolysaccharide pretreatment induces nuclear factor-kappaB alterations not present in C3H/HeJ mice.
    West MA, Clair L, Kraatz J, Rodriguez JL.
    J Trauma; 2000 Aug; 49(2):298-305. PubMed ID: 10963543
    [Abstract] [Full Text] [Related]

  • 2. In vitro macrophage endotoxin tolerance: defective in vitro macrophage map kinase signal transduction after LPS pretreatment is not present in macrophages from C3H/HeJ endotoxin resistant mice.
    Kraatz J, Clair L, Rodriguez JL, West MA.
    Shock; 1999 Jan; 11(1):58-63. PubMed ID: 9921718
    [Abstract] [Full Text] [Related]

  • 3. Nuclear factor translocation and acute anterior uveitis.
    Wang J, Lu H, Hu X, Chen W, Xu Z, Li S, Xu Y.
    Mol Vis; 2011 Jan 18; 17():170-6. PubMed ID: 21264236
    [Abstract] [Full Text] [Related]

  • 4. Lipopolysaccharide tolerance in murine peritoneal macrophages induces downregulation of the lipopolysaccharide signal transduction pathway through mitogen-activated protein kinase and nuclear factor-kappaB cascades, but not lipopolysaccharide-incorporation steps.
    Tominaga K, Saito S, Matsuura M, Nakano M.
    Biochim Biophys Acta; 1999 Jun 08; 1450(2):130-44. PubMed ID: 10354505
    [Abstract] [Full Text] [Related]

  • 5. Macrophages derived from C3H/HeJ (Lpsd) mice respond to bacterial lipopolysaccharide by activating NF-kappa B.
    Ding A, Hwang S, Lander HM, Xie QW.
    J Leukoc Biol; 1995 Jan 08; 57(1):174-9. PubMed ID: 7829969
    [Abstract] [Full Text] [Related]

  • 6. Quantitative aspects of lipopolysaccharide and cytokine requirements to generate nitric oxide in macrophages from LPS-hyporesponsive (Lps(d)) C3H/HeJ mice.
    Kmonícková E, Zídek Z.
    Folia Microbiol (Praha); 2004 Jan 08; 49(6):737-44. PubMed ID: 15881412
    [Abstract] [Full Text] [Related]

  • 7. Lipopolysaccharide pretreatment produces macrophage endotoxin tolerance via a serum-independent pathway.
    Kraatz J, Clair L, Bellingham J, Wahlstrom K, Rodriguez JL, West MA.
    J Trauma; 1998 Oct 08; 45(4):684-91. PubMed ID: 9783605
    [Abstract] [Full Text] [Related]

  • 8. Staphylococcal enterotoxin B induces hepatic injury and lethal shock in endotoxin-resistant C3H/HeJ mice despite a deficient macrophage response.
    Yasuda S, Nagaki M, Moriwaki H.
    J Endotoxin Res; 2002 Oct 08; 8(4):253-61. PubMed ID: 12230915
    [Abstract] [Full Text] [Related]

  • 9. Lipopolysaccharide (LPS)-induced intra-uterine fetal death (IUFD) in mice is principally due to maternal cause but not fetal sensitivity to LPS.
    Kohmura Y, Kirikae T, Kirikae F, Nakano M, Sato I.
    Microbiol Immunol; 2000 Oct 08; 44(11):897-904. PubMed ID: 11145270
    [Abstract] [Full Text] [Related]

  • 10. LPS regulation of the immune response: Bacteroides endotoxin induces mitogenic, polyclonal, and antibody responses in classical LPS responsive but not C3H/HeJ mice.
    Wannemuehler MJ, Michalek SM, Jirillo E, Williamson SI, Hirasawa M, McGhee JR.
    J Immunol; 1984 Jul 08; 133(1):299-305. PubMed ID: 6202784
    [Abstract] [Full Text] [Related]

  • 11. Alveolar macrophages from C3H/HeJ mice show sensitivity to endotoxin.
    Ryan LK, Vermeulen MW.
    Am J Respir Cell Mol Biol; 1995 May 08; 12(5):540-6. PubMed ID: 7742017
    [Abstract] [Full Text] [Related]

  • 12. Lipopolysaccharides (LPS) of oral black-pigmented bacteria induce tumor necrosis factor production by LPS-refractory C3H/HeJ macrophages in a way different from that of Salmonella LPS.
    Kirikae T, Nitta T, Kirikae F, Suda Y, Kusumoto S, Qureshi N, Nakano M.
    Infect Immun; 1999 Apr 08; 67(4):1736-42. PubMed ID: 10085012
    [Abstract] [Full Text] [Related]

  • 13. Inhibitory kappaBalpha control of nuclear factor-kappaB is dysregulated in endotoxin tolerant macrophages.
    Wahlstrom K, Bellingham J, Rodriguez JL, West MA.
    Shock; 1999 Apr 08; 11(4):242-7. PubMed ID: 10220299
    [Abstract] [Full Text] [Related]

  • 14. LPS regulation of the immune response: separate mechanisms for murine B cell activation by lipid A (direct) and polysaccharide (macrophage-dependent) derived from Bacteroides LPS.
    Williamson SI, Wannemuehler MJ, Jirillo E, Pritchard DG, Michalek SM, McGhee JR.
    J Immunol; 1984 Nov 08; 133(5):2294-300. PubMed ID: 6332842
    [Abstract] [Full Text] [Related]

  • 15. Berberine ameliorates TNBS-induced colitis by inhibiting lipid peroxidation, enterobacterial growth and NF-κB activation.
    Lee IA, Hyun YJ, Kim DH.
    Eur J Pharmacol; 2010 Dec 01; 648(1-3):162-70. PubMed ID: 20828550
    [Abstract] [Full Text] [Related]

  • 16. Tyrosine phosphorylation of mitogen-activated protein kinases is necessary for activation of murine macrophages by natural and synthetic bacterial products.
    Dong Z, Qi X, Fidler IJ.
    J Exp Med; 1993 Apr 01; 177(4):1071-7. PubMed ID: 8384652
    [Abstract] [Full Text] [Related]

  • 17. The activation of tumoricidal properties in macrophages of endotoxin responder and nonresponder mice by liposome-encapsulated immunomodulators.
    Fogler WE, Talmadge JE, Fidler IJ.
    J Reticuloendothel Soc; 1983 Mar 01; 33(3):165-74. PubMed ID: 6834360
    [Abstract] [Full Text] [Related]

  • 18. [K. pneumoniea endotoxin induced mice beta-defensin-4 mRNA expression and its signaling transduction].
    Cai S, Du J, Chen X, Huang N, Wang L, Ma X, Kiyoyuki K, Fumihiko N, Wang B, Takaiki H.
    Hua Xi Yi Ke Da Xue Xue Bao; 2001 Jun 01; 32(2):157-62. PubMed ID: 12600073
    [Abstract] [Full Text] [Related]

  • 19. Lipopolysaccharide (LPS)-induced cell death of C3H mouse peritoneal macrophages in the presence of cycloheximide: different susceptibilities of C3H/HeN and C3H/HeJ mice macrophages.
    Karahashi H, Amano F.
    J Endotoxin Res; 2000 Jun 01; 6(1):33-9. PubMed ID: 11061030
    [Abstract] [Full Text] [Related]

  • 20. The expression of prostaglandin E receptors EP2 and EP4 and their different regulation by lipopolysaccharide in C3H/HeN peritoneal macrophages.
    Ikegami R, Sugimoto Y, Segi E, Katsuyama M, Karahashi H, Amano F, Maruyama T, Yamane H, Tsuchiya S, Ichikawa A.
    J Immunol; 2001 Apr 01; 166(7):4689-96. PubMed ID: 11254729
    [Abstract] [Full Text] [Related]


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