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Journal Abstract Search

255 related items for PubMed ID: 10970056

  • 21. Mutations in neurofilament genes are not a significant primary cause of non-SOD1-mediated amyotrophic lateral sclerosis.
    Garcia ML, Singleton AB, Hernandez D, Ward CM, Evey C, Sapp PA, Hardy J, Brown RH, Cleveland DW.
    Neurobiol Dis; 2006 Jan; 21(1):102-9. PubMed ID: 16084104
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  • 26. [Superoxide dismutase-1 (SOD-1) gene mutation-dependent mechanisms of neural degeneration in amyotrophic lateral sclerosis].
    Iłzecka J.
    Neurol Neurochir Pol; 2001 Jan; 35(3):461-9. PubMed ID: 11732268
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  • 28. A novel SOD1 mutation in a young amyotrophic lateral sclerosis patient with a very slowly progressive clinical course.
    Georgoulopoulou E, Gellera C, Bragato C, Sola P, Chiari A, Bernabei C, Mandrioli J.
    Muscle Nerve; 2010 Oct; 42(4):596-7. PubMed ID: 20740631
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  • 30. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
    Mackenzie IR, Bigio EH, Ince PG, Geser F, Neumann M, Cairns NJ, Kwong LK, Forman MS, Ravits J, Stewart H, Eisen A, McClusky L, Kretzschmar HA, Monoranu CM, Highley JR, Kirby J, Siddique T, Shaw PJ, Lee VM, Trojanowski JQ.
    Ann Neurol; 2007 May; 61(5):427-34. PubMed ID: 17469116
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  • 31. Oxidative stress in amyotrophic lateral sclerosis.
    Robberecht W.
    J Neurol; 2000 Mar; 247 Suppl 1():I1-6. PubMed ID: 10795881
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  • 32. Recent advances in research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 gene mutations: neuronal Lewy body-like hyaline inclusions and astrocytic hyaline inclusions.
    Kato S, Saito M, Hirano A, Ohama E.
    Histol Histopathol; 1999 Jul; 14(3):973-89. PubMed ID: 10425565
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  • 33. Combined fulminant frontotemporal dementia and amyotrophic lateral sclerosis associated with an I113T SOD1 mutation.
    Katz JS, Katzberg HD, Woolley SC, Marklund SL, Andersen PM.
    Amyotroph Lateral Scler; 2012 Oct; 13(6):567-9. PubMed ID: 22670877
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  • 34. Increased mitochondrial antioxidative activity or decreased oxygen free radical propagation prevent mutant SOD1-mediated motor neuron cell death and increase amyotrophic lateral sclerosis-like transgenic mouse survival.
    Liu R, Li B, Flanagan SW, Oberley LW, Gozal D, Qiu M.
    J Neurochem; 2002 Feb; 80(3):488-500. PubMed ID: 11905995
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  • 35. An RNAi strategy for treatment of amyotrophic lateral sclerosis caused by mutant Cu,Zn superoxide dismutase.
    Xia XG, Zhou H, Zhou S, Yu Y, Wu R, Xu Z.
    J Neurochem; 2005 Jan; 92(2):362-7. PubMed ID: 15663483
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  • 36. Expression of the copper-zinc superoxide dismutase gene in amyotrophic lateral sclerosis.
    Nishiyama K, Murayama S, Kwak S, Kanazawa I.
    Ann Neurol; 1997 Apr; 41(4):551-6. PubMed ID: 9124814
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  • 37. Mutant SOD1 causes motor neuron disease independent of copper chaperone-mediated copper loading.
    Subramaniam JR, Lyons WE, Liu J, Bartnikas TB, Rothstein J, Price DL, Cleveland DW, Gitlin JD, Wong PC.
    Nat Neurosci; 2002 Apr; 5(4):301-7. PubMed ID: 11889469
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  • 38. Differential expression of inflammation- and apoptosis-related genes in spinal cords of a mutant SOD1 transgenic mouse model of familial amyotrophic lateral sclerosis.
    Yoshihara T, Ishigaki S, Yamamoto M, Liang Y, Niwa J, Takeuchi H, Doyu M, Sobue G.
    J Neurochem; 2002 Jan; 80(1):158-67. PubMed ID: 11796754
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  • 39. Progranulin does not affect motor neuron degeneration in mutant SOD1 mice and rats.
    Herdewyn S, De Muynck L, Van Den Bosch L, Robberecht W, Van Damme P.
    Neurobiol Aging; 2013 Oct; 34(10):2302-3. PubMed ID: 23608112
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  • 40. Amyotrophic lateral sclerosis (motor neuron disease): proposed mechanisms and pathways to treatment.
    Goodall EF, Morrison KE.
    Expert Rev Mol Med; 2006 May 24; 8(11):1-22. PubMed ID: 16723044
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