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674 related items for PubMed ID: 10980307

  • 1. Aggregates of mutant protein appear progressively in dendrites, in periaxonal processes of oligodendrocytes, and in neuronal and astrocytic perikarya of mice expressing the SOD1(G93A) mutation of familial amyotrophic lateral sclerosis.
    Stieber A, Gonatas JO, Gonatas NK.
    J Neurol Sci; 2000 Aug 15; 177(2):114-23. PubMed ID: 10980307
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  • 2. Aggregation of ubiquitin and a mutant ALS-linked SOD1 protein correlate with disease progression and fragmentation of the Golgi apparatus.
    Stieber A, Gonatas JO, Gonatas NK.
    J Neurol Sci; 2000 Feb 01; 173(1):53-62. PubMed ID: 10675580
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  • 3. The neuronal Golgi apparatus is fragmented in transgenic mice expressing a mutant human SOD1, but not in mice expressing the human NF-H gene.
    Stieber A, Gonatas JO, Collard J, Meier J, Julien J, Schweitzer P, Gonatas NK.
    J Neurol Sci; 2000 Feb 01; 173(1):63-72. PubMed ID: 10675581
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  • 4. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues.
    Watanabe M, Dykes-Hoberg M, Culotta VC, Price DL, Wong PC, Rothstein JD.
    Neurobiol Dis; 2001 Dec 01; 8(6):933-41. PubMed ID: 11741389
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  • 5. Galectin-1 deficiency improves axonal swelling of motor neurones in SOD1(G93A) transgenic mice.
    Kobayakawa Y, Sakumi K, Kajitani K, Kadoya T, Horie H, Kira J, Nakabeppu Y.
    Neuropathol Appl Neurobiol; 2015 Feb 01; 41(2):227-44. PubMed ID: 24707896
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  • 6. Neurofilaments and orthograde transport are reduced in ventral root axons of transgenic mice that express human SOD1 with a G93A mutation.
    Zhang B, Tu P, Abtahian F, Trojanowski JQ, Lee VM.
    J Cell Biol; 1997 Dec 01; 139(5):1307-15. PubMed ID: 9382875
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  • 12. Golgi apparatus of the motor neurons in patients with amyotrophic lateral sclerosis and in mice models of amyotrophic lateral sclerosis.
    Fujita Y, Okamoto K.
    Neuropathology; 2005 Dec 01; 25(4):388-94. PubMed ID: 16382790
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  • 15. Protein-bound crotonaldehyde accumulates in the spinal cord of superoxide dismutase-1 mutation-associated familial amyotrophic lateral sclerosis and its transgenic mouse model.
    Shibata N, Kawaguchi M, Uchida K, Kakita A, Takahashi H, Nakano R, Fujimura H, Sakoda S, Ihara Y, Nobukuni K, Takehisa Y, Kuroda S, Kokubo Y, Kuzuhara S, Honma T, Mochizuki Y, Mizutani T, Yamada S, Toi S, Sasaki S, Iwata M, Hirano A, Yamamoto T, Kato Y, Sawada T, Kobayashi M.
    Neuropathology; 2007 Feb 01; 27(1):49-61. PubMed ID: 17319283
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  • 17. Transgenic mice carrying a human mutant superoxide dismutase transgene develop neuronal cytoskeletal pathology resembling human amyotrophic lateral sclerosis lesions.
    Tu PH, Raju P, Robinson KA, Gurney ME, Trojanowski JQ, Lee VM.
    Proc Natl Acad Sci U S A; 1996 Apr 02; 93(7):3155-60. PubMed ID: 8610185
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  • 18. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.
    Neurobiol Dis; 2014 Apr 02; 64():48-59. PubMed ID: 24361555
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  • 19. Immunodetection of disease-associated conformers of mutant cu/zn superoxide dismutase 1 selectively expressed in degenerating neurons in amyotrophic lateral sclerosis.
    Sábado J, Casanovas A, Hernández S, Piedrafita L, Hereu M, Esquerda JE.
    J Neuropathol Exp Neurol; 2013 Jul 02; 72(7):646-61. PubMed ID: 23771221
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  • 20. Redox proteomics analysis of oxidatively modified proteins in G93A-SOD1 transgenic mice--a model of familial amyotrophic lateral sclerosis.
    Poon HF, Hensley K, Thongboonkerd V, Merchant ML, Lynn BC, Pierce WM, Klein JB, Calabrese V, Butterfield DA.
    Free Radic Biol Med; 2005 Aug 15; 39(4):453-62. PubMed ID: 16043017
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