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Journal Abstract Search

520 related items for PubMed ID: 11006360

  • 21. In vitro re-expression of the aryl hydrocarbon receptor (Ahr) in cultured Ahr-deficient mouse antral follicles partially restores the phenotype to that of cultured wild-type mouse follicles.
    Ziv-Gal A, Gao L, Karman BN, Flaws JA.
    Toxicol In Vitro; 2015 Mar; 29(2):329-36. PubMed ID: 25500125
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  • 22. Female Sprague-Dawley rats exposed to a single oral dose of 2,3,7,8-tetrachlorodibenzo-p-dioxin exhibit sustained depletion of aryl hydrocarbon receptor protein in liver, spleen, thymus, and lung.
    Pollenz RS, Santostefano MJ, Klett E, Richardson VM, Necela B, Birnbaum LS.
    Toxicol Sci; 1998 Apr; 42(2):117-28. PubMed ID: 9579024
    [Abstract] [Full Text] [Related]

  • 23. T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4+ and CD8+ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD.
    Kerkvliet NI, Shepherd DM, Baecher-Steppan L.
    Toxicol Appl Pharmacol; 2002 Dec 01; 185(2):146-52. PubMed ID: 12490139
    [Abstract] [Full Text] [Related]

  • 24. Stromal estrogen receptors mediate mitogenic effects of estradiol on uterine epithelium.
    Cooke PS, Buchanan DL, Young P, Setiawan T, Brody J, Korach KS, Taylor J, Lubahn DB, Cunha GR.
    Proc Natl Acad Sci U S A; 1997 Jun 10; 94(12):6535-40. PubMed ID: 9177253
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  • 25. Functional activation of arylhydrocarbon receptor (AhR) in primary T cells by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
    Doi H, Baba T, Tohyama C, Nohara K.
    Chemosphere; 2003 Jul 10; 52(4):655-62. PubMed ID: 12738279
    [Abstract] [Full Text] [Related]

  • 26. The constitutively active Ah receptor (CA-AhR) mouse as a model for dioxin exposure - effects in reproductive organs.
    Brunnberg S, Andersson P, Poellinger L, Hanberg A.
    Chemosphere; 2011 Dec 10; 85(11):1701-6. PubMed ID: 22014662
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  • 27. A chimeric aryl hydrocarbon receptor knockout mouse model indicates that aryl hydrocarbon receptor activation in hematopoietic cells contributes to the hepatic lesions induced by 2,3,7, 8-tetrachlorodibenzo-p-dioxin.
    Thurmond TS, Silverstone AE, Baggs RB, Quimby FW, Staples JE, Gasiewicz TA.
    Toxicol Appl Pharmacol; 1999 Jul 01; 158(1):33-40. PubMed ID: 10387930
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  • 28. Altered thyroxin and retinoid metabolic response to 2,3,7,8-tetrachlorodibenzo-p-dioxin in aryl hydrocarbon receptor-null mice.
    Nishimura N, Yonemoto J, Miyabara Y, Fujii-Kuriyama Y, Tohyama C.
    Arch Toxicol; 2005 May 01; 79(5):260-7. PubMed ID: 15902423
    [Abstract] [Full Text] [Related]

  • 29. Prolonged depletion of AH receptor without alteration of receptor mRNA levels after treatment of cells in culture with 2,3,7,8-tetrachlorodibenzo-p-dioxin.
    Giannone JV, Li W, Probst M, Okey AB.
    Biochem Pharmacol; 1998 Feb 15; 55(4):489-97. PubMed ID: 9514084
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  • 31. Aryl-hydrocarbon receptor-dependent pathway and toxic effects of TCDD in humans: a population-based study in Seveso, Italy.
    Baccarelli A, Pesatori AC, Masten SA, Patterson DG, Needham LL, Mocarelli P, Caporaso NE, Consonni D, Grassman JA, Bertazzi PA, Landi MT.
    Toxicol Lett; 2004 Apr 01; 149(1-3):287-93. PubMed ID: 15093275
    [Abstract] [Full Text] [Related]

  • 32. The aryl hydrocarbon receptor and aryl hydrocarbon receptor nuclear translocator protein show distinct subcellular localizations in Hepa 1c1c7 cells by immunofluorescence microscopy.
    Pollenz RS, Sattler CA, Poland A.
    Mol Pharmacol; 1994 Mar 01; 45(3):428-38. PubMed ID: 8145729
    [Abstract] [Full Text] [Related]

  • 33. TCDD-induced expression of Ah receptor responsive genes in the pituitary and brain of cellular retinol-binding protein (CRBP-I) knockout mice.
    Huang P, Ceccatelli S, Hoegberg P, Sten Shi TJ, Håkansson H, Rannug A.
    Toxicol Appl Pharmacol; 2003 Nov 01; 192(3):262-74. PubMed ID: 14575644
    [Abstract] [Full Text] [Related]

  • 34. Aryl hydrocarbon receptor (AHR) and AHR nuclear translocator (ARNT) expression in Baikal seal (Pusa sibirica) and association with 2,3,7,8-TCDD toxic equivalents and CYP1 expression levels.
    Kim EY, Iwata H, Suda T, Tanabe S, Amano M, Miyazaki N, Petrov EA.
    Comp Biochem Physiol C Toxicol Pharmacol; 2005 Jul 01; 141(3):281-91. PubMed ID: 16111922
    [Abstract] [Full Text] [Related]

  • 35. Disruption of clock gene expression alters responses of the aryl hydrocarbon receptor signaling pathway in the mouse mammary gland.
    Qu X, Metz RP, Porter WW, Cassone VM, Earnest DJ.
    Mol Pharmacol; 2007 Nov 01; 72(5):1349-58. PubMed ID: 17715397
    [Abstract] [Full Text] [Related]

  • 36. Expression of the arylhydrocarbon receptor and the arylhydrocarbon receptor nuclear translocator during early gestation in the rabbit uterus.
    Tscheudschilsuren G, Hombach-Klonisch S, Küchenhoff A, Fischer B, Klonisch T.
    Toxicol Appl Pharmacol; 1999 Nov 01; 160(3):231-7. PubMed ID: 10544057
    [Abstract] [Full Text] [Related]

  • 37. Physicochemical differences in the AH receptors of the most TCDD-susceptible and the most TCDD-resistant rat strains.
    Pohjanvirta R, Viluksela M, Tuomisto JT, Unkila M, Karasinska J, Franc MA, Holowenko M, Giannone JV, Harper PA, Tuomisto J, Okey AB.
    Toxicol Appl Pharmacol; 1999 Feb 15; 155(1):82-95. PubMed ID: 10036221
    [Abstract] [Full Text] [Related]

  • 38. Effects of TCDD on Ah receptor, ARNT, EGF, and TGF-alpha expression in embryonic mouse urinary tract.
    Bryant PL, Clark GC, Probst MR, Abbott BD.
    Teratology; 1997 May 15; 55(5):326-37. PubMed ID: 9261927
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