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175 related items for PubMed ID: 11009479

  • 1. Vasodilator mechanisms in the coronary circulation of endothelial nitric oxide synthase-deficient mice.
    Lamping KG, Nuno DW, Shesely EG, Maeda N, Faraci FM.
    Am J Physiol Heart Circ Physiol; 2000 Oct; 279(4):H1906-12. PubMed ID: 11009479
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  • 2. Acetylcholine-induced relaxation in blood vessels from endothelial nitric oxide synthase knockout mice.
    Chataigneau T, Félétou M, Huang PL, Fishman MC, Duhault J, Vanhoutte PM.
    Br J Pharmacol; 1999 Jan; 126(1):219-26. PubMed ID: 10051139
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  • 3. Contribution of nitric oxide synthase isoforms to cholinergic vasodilation in murine retinal arterioles.
    Gericke A, Goloborodko E, Sniatecki JJ, Steege A, Wojnowski L, Pfeiffer N.
    Exp Eye Res; 2013 Apr; 109():60-6. PubMed ID: 23434456
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  • 4. Role of nitric oxide synthase isoforms for ophthalmic artery reactivity in mice.
    Laspas P, Goloborodko E, Sniatecki JJ, Kordasz ML, Manicam C, Wojnowski L, Li H, Patzak A, Pfeiffer N, Gericke A.
    Exp Eye Res; 2014 Oct; 127():1-8. PubMed ID: 25017185
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  • 5. Regulation of nitric oxide-dependent vasodilation in coronary arteries of estrogen receptor-alpha-deficient mice.
    Muller-Delp JM, Lubahn DB, Nichol KE, Philips BJ, Price EM, Curran EM, Laughlin MH.
    Am J Physiol Heart Circ Physiol; 2003 Nov; 285(5):H2150-7. PubMed ID: 12881205
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  • 6. Neuronal nitric oxide synthase-derived hydrogen peroxide is a major endothelium-dependent relaxing factor.
    Capettini LS, Cortes SF, Gomes MA, Silva GA, Pesquero JL, Lopes MJ, Teixeira MM, Lemos VS.
    Am J Physiol Heart Circ Physiol; 2008 Dec; 295(6):H2503-11. PubMed ID: 18952716
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  • 7. An in vitro investigation of aorta and corpus cavernosum from eNOS and nNOS gene-deficient mice.
    Nangle MR, Cotter MA, Cameron NE.
    Pflugers Arch; 2004 May; 448(2):139-45. PubMed ID: 14722775
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  • 8. Neuronal NOS-dependent dilation to flow in coronary arteries of male eNOS-KO mice.
    Huang A, Sun D, Shesely EG, Levee EM, Koller A, Kaley G.
    Am J Physiol Heart Circ Physiol; 2002 Feb; 282(2):H429-36. PubMed ID: 11788389
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  • 10. Enhanced release of prostaglandins contributes to flow-induced arteriolar dilation in eNOS knockout mice.
    Sun D, Huang A, Smith CJ, Stackpole CJ, Connetta JA, Shesely EG, Koller A, Kaley G.
    Circ Res; 1999 Aug 06; 85(3):288-93. PubMed ID: 10436172
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  • 11. Acetylcholine-induced relaxation of peripheral arteries isolated from mice lacking endothelial nitric oxide synthase.
    Waldron GJ, Ding H, Lovren F, Kubes P, Triggle CR.
    Br J Pharmacol; 1999 Oct 06; 128(3):653-8. PubMed ID: 10516645
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  • 12. In eNOS knockout mice skeletal muscle arteriolar dilation to acetylcholine is mediated by EDHF.
    Huang A, Sun D, Smith CJ, Connetta JA, Shesely EG, Koller A, Kaley G.
    Am J Physiol Heart Circ Physiol; 2000 Mar 06; 278(3):H762-8. PubMed ID: 10710344
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  • 16. Endothelial nitric oxide synthase activation leads to dilatory H2O2 production in mouse cerebral arteries.
    Drouin A, Thorin-Trescases N, Hamel E, Falck JR, Thorin E.
    Cardiovasc Res; 2007 Jan 01; 73(1):73-81. PubMed ID: 17113574
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  • 17. A change in the redox environment and thromboxane A2 production precede endothelial dysfunction in mice.
    Gendron ME, Thorin E.
    Am J Physiol Heart Circ Physiol; 2007 Oct 01; 293(4):H2508-15. PubMed ID: 17644574
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  • 18. Responses of cerebral arterioles to ADP: eNOS-dependent and eNOS-independent mechanisms.
    Faraci FM, Lynch C, Lamping KG.
    Am J Physiol Heart Circ Physiol; 2004 Dec 01; 287(6):H2871-6. PubMed ID: 15548728
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