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71 related items for PubMed ID: 11013131

  • 1. Functional suppression of sodium channels by beta(1)-subunits as a molecular mechanism of idiopathic ventricular fibrillation.
    Wan X, Wang Q, Kirsch GE.
    J Mol Cell Cardiol; 2000 Oct; 32(10):1873-84. PubMed ID: 11013131
    [Abstract] [Full Text] [Related]

  • 2. Expression and intracellular localization of an SCN5A double mutant R1232W/T1620M implicated in Brugada syndrome.
    Baroudi G, Acharfi S, Larouche C, Chahine M.
    Circ Res; 2002 Jan 11; 90(1):E11-6. PubMed ID: 11786529
    [Abstract] [Full Text] [Related]

  • 3. Accelerated inactivation in a mutant Na(+) channel associated with idiopathic ventricular fibrillation.
    Wan X, Chen S, Sadeghpour A, Wang Q, Kirsch GE.
    Am J Physiol Heart Circ Physiol; 2001 Jan 11; 280(1):H354-60. PubMed ID: 11123251
    [Abstract] [Full Text] [Related]

  • 4. Electrocardiographic characteristics and SCN5A mutations in idiopathic ventricular fibrillation associated with early repolarization.
    Watanabe H, Nogami A, Ohkubo K, Kawata H, Hayashi Y, Ishikawa T, Makiyama T, Nagao S, Yagihara N, Takehara N, Kawamura Y, Sato A, Okamura K, Hosaka Y, Sato M, Fukae S, Chinushi M, Oda H, Okabe M, Kimura A, Maemura K, Watanabe I, Kamakura S, Horie M, Aizawa Y, Shimizu W, Makita N.
    Circ Arrhythm Electrophysiol; 2011 Dec 11; 4(6):874-81. PubMed ID: 22028457
    [Abstract] [Full Text] [Related]

  • 5. A novel mechanism associated with idiopathic ventricular fibrillation (IVF) mutations R1232W and T1620M in human cardiac sodium channels.
    Vilin YY, Fujimoto E, Ruben PC.
    Pflugers Arch; 2001 May 11; 442(2):204-11. PubMed ID: 11417215
    [Abstract] [Full Text] [Related]

  • 6. Clinical and electrophysiological characteristics of Brugada syndrome caused by a missense mutation in the S5-pore site of SCN5A.
    Itoh H, Shimizu M, Mabuchi H, Imoto K.
    J Cardiovasc Electrophysiol; 2005 Apr 11; 16(4):378-83. PubMed ID: 15828879
    [Abstract] [Full Text] [Related]

  • 7. A mutation causing pseudohypoaldosteronism type 1 identifies a conserved glycine that is involved in the gating of the epithelial sodium channel.
    Gründer S, Firsov D, Chang SS, Jaeger NF, Gautschi I, Schild L, Lifton RP, Rossier BC.
    EMBO J; 1997 Mar 03; 16(5):899-907. PubMed ID: 9118951
    [Abstract] [Full Text] [Related]

  • 8. Furin cleavage activates the epithelial Na+ channel by relieving Na+ self-inhibition.
    Sheng S, Carattino MD, Bruns JB, Hughey RP, Kleyman TR.
    Am J Physiol Renal Physiol; 2006 Jun 03; 290(6):F1488-96. PubMed ID: 16449353
    [Abstract] [Full Text] [Related]

  • 9. A novel SCN5A mutation, F1344S, identified in a patient with Brugada syndrome and fever-induced ventricular fibrillation.
    Keller DI, Huang H, Zhao J, Frank R, Suarez V, Delacrétaz E, Brink M, Osswald S, Schwick N, Chahine M.
    Cardiovasc Res; 2006 Jun 01; 70(3):521-9. PubMed ID: 16616735
    [Abstract] [Full Text] [Related]

  • 10. Novel Brugada syndrome-causing mutation in ion-conducting pore of cardiac Na+ channel does not affect ion selectivity properties.
    Amin AS, Verkerk AO, Bhuiyan ZA, Wilde AA, Tan HL.
    Acta Physiol Scand; 2005 Dec 01; 185(4):291-301. PubMed ID: 16266370
    [Abstract] [Full Text] [Related]

  • 11. Molecular properties of brain sodium channels: an important target for anticonvulsant drugs.
    Catterall WA.
    Adv Neurol; 1999 Dec 01; 79():441-56. PubMed ID: 10514834
    [Abstract] [Full Text] [Related]

  • 12. Novel mechanism for Brugada syndrome: defective surface localization of an SCN5A mutant (R1432G).
    Baroudi G, Pouliot V, Denjoy I, Guicheney P, Shrier A, Chahine M.
    Circ Res; 2001 Jun 22; 88(12):E78-83. PubMed ID: 11420310
    [Abstract] [Full Text] [Related]

  • 13. Cardiac Na(+) channel dysfunction in Brugada syndrome is aggravated by beta(1)-subunit.
    Makita N, Shirai N, Wang DW, Sasaki K, George AL, Kanno M, Kitabatake A.
    Circulation; 2001 Jun 22; 101(1):54-60. PubMed ID: 10618304
    [Abstract] [Full Text] [Related]

  • 14. Genetic basis and molecular mechanism for idiopathic ventricular fibrillation.
    Chen Q, Kirsch GE, Zhang D, Brugada R, Brugada J, Brugada P, Potenza D, Moya A, Borggrefe M, Breithardt G, Ortiz-Lopez R, Wang Z, Antzelevitch C, O'Brien RE, Schulze-Bahr E, Keating MT, Towbin JA, Wang Q.
    Nature; 1998 Mar 19; 392(6673):293-6. PubMed ID: 9521325
    [Abstract] [Full Text] [Related]

  • 15. Generalized epilepsy with febrile seizures plus-associated sodium channel beta1 subunit mutations severely reduce beta subunit-mediated modulation of sodium channel function.
    Xu R, Thomas EA, Gazina EV, Richards KL, Quick M, Wallace RH, Harkin LA, Heron SE, Berkovic SF, Scheffer IE, Mulley JC, Petrou S.
    Neuroscience; 2007 Aug 10; 148(1):164-74. PubMed ID: 17629415
    [Abstract] [Full Text] [Related]

  • 16. A novel missense mutation in the SCN5A gene associated with Brugada syndrome bidirectionally affecting blocking actions of antiarrhythmic drugs.
    Itoh H, Shimizu M, Takata S, Mabuchi H, Imoto K.
    J Cardiovasc Electrophysiol; 2005 May 10; 16(5):486-93. PubMed ID: 15877619
    [Abstract] [Full Text] [Related]

  • 17. The heterotetrameric architecture of the epithelial sodium channel (ENaC).
    Firsov D, Gautschi I, Merillat AM, Rossier BC, Schild L.
    EMBO J; 1998 Jan 15; 17(2):344-52. PubMed ID: 9430626
    [Abstract] [Full Text] [Related]

  • 18. Loss-of-function mutation of the SCN3B-encoded sodium channel {beta}3 subunit associated with a case of idiopathic ventricular fibrillation.
    Valdivia CR, Medeiros-Domingo A, Ye B, Shen WK, Algiers TJ, Ackerman MJ, Makielski JC.
    Cardiovasc Res; 2010 Jun 01; 86(3):392-400. PubMed ID: 20042427
    [Abstract] [Full Text] [Related]

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  • 20. Functional protein expression of multiple sodium channel alpha- and beta-subunit isoforms in neonatal cardiomyocytes.
    Kaufmann SG, Westenbroek RE, Zechner C, Maass AH, Bischoff S, Muck J, Wischmeyer E, Scheuer T, Maier SK.
    J Mol Cell Cardiol; 2010 Jan 01; 48(1):261-9. PubMed ID: 19426735
    [Abstract] [Full Text] [Related]

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