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207 related items for PubMed ID: 11045671

  • 1. Advanced glycation endproduct-modified superoxide dismutase-1 (SOD1)-positive inclusions are common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutations and transgenic mice expressing human SOD1 with a G85R mutation.
    Kato S, Horiuchi S, Liu J, Cleveland DW, Shibata N, Nakashima K, Nagai R, Hirano A, Takikawa M, Kato M, Nakano I, Ohama E.
    Acta Neuropathol; 2000 Nov; 100(5):490-505. PubMed ID: 11045671
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  • 2. Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutation.
    Kato S, Nakashima K, Horiuchi S, Nagai R, Cleveland DW, Liu J, Hirano A, Takikawa M, Kato M, Nakano I, Sakoda S, Asayama K, Ohama E.
    Neuropathology; 2001 Mar; 21(1):67-81. PubMed ID: 11304045
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  • 3. Astrocytic hyaline inclusions contain advanced glycation endproducts in familial amyotrophic lateral sclerosis with superoxide dismutase 1 gene mutation: immunohistochemical and immunoelectron microscopical analyses.
    Kato S, Horiuchi S, Nakashima K, Hirano A, Shibata N, Nakano I, Saito M, Kato M, Asayama K, Ohama E.
    Acta Neuropathol; 1999 Mar; 97(3):260-6. PubMed ID: 10090673
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  • 4. Advanced glycation endproducts are deposited in neuronal hyaline inclusions: a study on familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutation.
    Shibata N, Hirano A, Kato S, Nagai R, Horiuchi S, Komori T, Umahara T, Asayama K, Kobayashi M.
    Acta Neuropathol; 1999 Mar; 97(3):240-6. PubMed ID: 10090670
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  • 5. New consensus research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 (SOD1) gene mutations: inclusions containing SOD1 in neurons and astrocytes.
    Kato S, Takikawa M, Nakashima K, Hirano A, Cleveland DW, Kusaka H, Shibata N, Kato M, Nakano I, Ohama E.
    Amyotroph Lateral Scler Other Motor Neuron Disord; 2000 Jun; 1(3):163-84. PubMed ID: 11464950
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  • 12. TDP-43 immunoreactivity in neuronal inclusions in familial amyotrophic lateral sclerosis with or without SOD1 gene mutation.
    Tan CF, Eguchi H, Tagawa A, Onodera O, Iwasaki T, Tsujino A, Nishizawa M, Kakita A, Takahashi H.
    Acta Neuropathol; 2007 May; 113(5):535-42. PubMed ID: 17333220
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  • 13. HtrA2/Omi-immunoreactive intraneuronal inclusions in the anterior horn of patients with sporadic and Cu/Zn superoxide dismutase (SOD1) mutant amyotrophic lateral sclerosis.
    Kawamoto Y, Ito H, Kobayashi Y, Suzuki Y, Akiguchi I, Fujimura H, Sakoda S, Kusaka H, Hirano A, Takahashi R.
    Neuropathol Appl Neurobiol; 2010 Jun; 36(4):331-44. PubMed ID: 20202124
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  • 15. Familial amyotrophic lateral sclerosis with a two base pair deletion in superoxide dismutase 1: gene multisystem degeneration with intracytoplasmic hyaline inclusions in astrocytes.
    Kato S, Shimoda M, Watanabe Y, Nakashima K, Takahashi K, Ohama E.
    J Neuropathol Exp Neurol; 1996 Oct; 55(10):1089-101. PubMed ID: 8858006
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  • 16. Aggregates of mutant protein appear progressively in dendrites, in periaxonal processes of oligodendrocytes, and in neuronal and astrocytic perikarya of mice expressing the SOD1(G93A) mutation of familial amyotrophic lateral sclerosis.
    Stieber A, Gonatas JO, Gonatas NK.
    J Neurol Sci; 2000 Aug 15; 177(2):114-23. PubMed ID: 10980307
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  • 18. An in vitro model for Lewy body-like hyaline inclusion/astrocytic hyaline inclusion: induction by ER stress with an ALS-linked SOD1 mutation.
    Yamagishi S, Koyama Y, Katayama T, Taniguchi M, Hitomi J, Kato M, Aoki M, Itoyama Y, Kato S, Tohyama M.
    PLoS One; 2007 Oct 10; 2(10):e1030. PubMed ID: 17925878
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  • 19. Stabilization of mutant Cu/Zn superoxide dismutase (SOD1) protein by coexpressed wild SOD1 protein accelerates the disease progression in familial amyotrophic lateral sclerosis mice.
    Fukada K, Nagano S, Satoh M, Tohyama C, Nakanishi T, Shimizu A, Yanagihara T, Sakoda S.
    Eur J Neurosci; 2001 Dec 10; 14(12):2032-6. PubMed ID: 11860498
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  • 20. Transgenic mice carrying a human mutant superoxide dismutase transgene develop neuronal cytoskeletal pathology resembling human amyotrophic lateral sclerosis lesions.
    Tu PH, Raju P, Robinson KA, Gurney ME, Trojanowski JQ, Lee VM.
    Proc Natl Acad Sci U S A; 1996 Apr 02; 93(7):3155-60. PubMed ID: 8610185
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