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153 related items for PubMed ID: 11061030

  • 1. Lipopolysaccharide (LPS)-induced cell death of C3H mouse peritoneal macrophages in the presence of cycloheximide: different susceptibilities of C3H/HeN and C3H/HeJ mice macrophages.
    Karahashi H, Amano F.
    J Endotoxin Res; 2000; 6(1):33-9. PubMed ID: 11061030
    [Abstract] [Full Text] [Related]

  • 2. Lipopolysaccharide (LPS)-induced intra-uterine fetal death (IUFD) in mice is principally due to maternal cause but not fetal sensitivity to LPS.
    Kohmura Y, Kirikae T, Kirikae F, Nakano M, Sato I.
    Microbiol Immunol; 2000; 44(11):897-904. PubMed ID: 11145270
    [Abstract] [Full Text] [Related]

  • 3. Structure-activity relationships of lipopolysaccharide (LPS) in tumor necrosis factor-alpha (TNF-alpha) production and induction of macrophage cell death in the presence of cycloheximide (CHX) in a murine macrophage-like cell line, J774.1.
    Karahashi H, Amano F.
    Biol Pharm Bull; 1998 Oct; 21(10):1102-5. PubMed ID: 9821819
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  • 4. Endotoxin tolerance from lipopolysaccharide pretreatment induces nuclear factor-kappaB alterations not present in C3H/HeJ mice.
    West MA, Clair L, Kraatz J, Rodriguez JL.
    J Trauma; 2000 Aug; 49(2):298-305. PubMed ID: 10963543
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  • 5. Mechanisms involved in apoptosis of human macrophages induced by lipopolysaccharide from Actinobacillus actinomycetemcomitans in the presence of cycloheximide.
    Suzuki T, Kobayashi M, Isatsu K, Nishihara T, Aiuchi T, Nakaya K, Hasegawa K.
    Infect Immun; 2004 Apr; 72(4):1856-65. PubMed ID: 15039304
    [Abstract] [Full Text] [Related]

  • 6. Lipopolysaccharides (LPS) of oral black-pigmented bacteria induce tumor necrosis factor production by LPS-refractory C3H/HeJ macrophages in a way different from that of Salmonella LPS.
    Kirikae T, Nitta T, Kirikae F, Suda Y, Kusumoto S, Qureshi N, Nakano M.
    Infect Immun; 1999 Apr; 67(4):1736-42. PubMed ID: 10085012
    [Abstract] [Full Text] [Related]

  • 7. Alveolar macrophages from C3H/HeJ mice show sensitivity to endotoxin.
    Ryan LK, Vermeulen MW.
    Am J Respir Cell Mol Biol; 1995 May; 12(5):540-6. PubMed ID: 7742017
    [Abstract] [Full Text] [Related]

  • 8. Quantitative aspects of lipopolysaccharide and cytokine requirements to generate nitric oxide in macrophages from LPS-hyporesponsive (Lps(d)) C3H/HeJ mice.
    Kmonícková E, Zídek Z.
    Folia Microbiol (Praha); 2004 May; 49(6):737-44. PubMed ID: 15881412
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  • 10. Staphylococcal enterotoxin B induces hepatic injury and lethal shock in endotoxin-resistant C3H/HeJ mice despite a deficient macrophage response.
    Yasuda S, Nagaki M, Moriwaki H.
    J Endotoxin Res; 2002 May; 8(4):253-61. PubMed ID: 12230915
    [Abstract] [Full Text] [Related]

  • 11. Macrophages derived from C3H/HeJ (Lpsd) mice respond to bacterial lipopolysaccharide by activating NF-kappa B.
    Ding A, Hwang S, Lander HM, Xie QW.
    J Leukoc Biol; 1995 Jan; 57(1):174-9. PubMed ID: 7829969
    [Abstract] [Full Text] [Related]

  • 12. Paradoxical preservation of a lipopolysaccharide response in C3H/HeJ macrophages: induction of matrix metalloproteinase-9.
    Jin F, Nathan CF, Ding A.
    J Immunol; 1999 Mar 15; 162(6):3596-600. PubMed ID: 10092819
    [Abstract] [Full Text] [Related]

  • 13. LPS regulation of the immune response: separate mechanisms for murine B cell activation by lipid A (direct) and polysaccharide (macrophage-dependent) derived from Bacteroides LPS.
    Williamson SI, Wannemuehler MJ, Jirillo E, Pritchard DG, Michalek SM, McGhee JR.
    J Immunol; 1984 Nov 15; 133(5):2294-300. PubMed ID: 6332842
    [Abstract] [Full Text] [Related]

  • 14. Functional role of interleukin 1 in periodontal disease: induction of interleukin 1 production by Bacteroides gingivalis lipopolysaccharide in peritoneal macrophages from C3H/HeN and C3H/HeJ mice.
    Hanazawa S, Nakada K, Ohmori Y, Miyoshi T, Amano S, Kitano S.
    Infect Immun; 1985 Oct 15; 50(1):262-70. PubMed ID: 3876285
    [Abstract] [Full Text] [Related]

  • 15. The importance of a lipopolysaccharide-initiated, cytokine-mediated host defense mechanism in mice against extraintestinally invasive Escherichia coli.
    Cross A, Asher L, Seguin M, Yuan L, Kelly N, Hammack C, Sadoff J, Gemski P.
    J Clin Invest; 1995 Aug 15; 96(2):676-86. PubMed ID: 7635960
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  • 17. Tyrosine phosphorylation of mitogen-activated protein kinases is necessary for activation of murine macrophages by natural and synthetic bacterial products.
    Dong Z, Qi X, Fidler IJ.
    J Exp Med; 1993 Apr 01; 177(4):1071-7. PubMed ID: 8384652
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  • 18. Structural requirements of taxoids for nitric oxide and tumor necrosis factor production by murine macrophages.
    Kirikae T, Ojima I, Kirikae F, Ma Z, Kuduk SD, Slater JC, Takeuchi CS, Bounaud PY, Nakano M.
    Biochem Biophys Res Commun; 1996 Oct 03; 227(1):227-35. PubMed ID: 8858130
    [Abstract] [Full Text] [Related]

  • 19. In vitro macrophage endotoxin tolerance: defective in vitro macrophage map kinase signal transduction after LPS pretreatment is not present in macrophages from C3H/HeJ endotoxin resistant mice.
    Kraatz J, Clair L, Rodriguez JL, West MA.
    Shock; 1999 Jan 03; 11(1):58-63. PubMed ID: 9921718
    [Abstract] [Full Text] [Related]

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