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252 related items for PubMed ID: 11073886

  • 1. Galpha(i2) but not Galpha(i3) is required for muscarinic inhibition of contractility and calcium currents in adult cardiomyocytes.
    Nagata K, Ye C, Jain M, Milstone DS, Liao R, Mortensen RM.
    Circ Res; 2000 Nov 10; 87(10):903-9. PubMed ID: 11073886
    [Abstract] [Full Text] [Related]

  • 2. Lack of muscarinic regulation of Ca(2+) channels in G(i2)alpha gene knockout mouse hearts.
    Chen F, Spicher K, Jiang M, Birnbaumer L, Wetzel GT.
    Am J Physiol Heart Circ Physiol; 2001 May 10; 280(5):H1989-95. PubMed ID: 11299198
    [Abstract] [Full Text] [Related]

  • 3. Galpha(i2), Galpha(i3)and Galpha(o) are all required for normal muscarinic inhibition of the cardiac calcium channels in nodal/atrial-like cultured cardiocytes.
    Ye C, Sowell MO, Vassilev PM, Milstone DS, Mortensen RM.
    J Mol Cell Cardiol; 1999 Sep 10; 31(9):1771-81. PubMed ID: 10471359
    [Abstract] [Full Text] [Related]

  • 4. Genetic disruption of G proteins, G(i2)alpha or G(o)alpha, does not abolish inotropic and chronotropic effects of stimulating muscarinic cholinoceptors in atrium.
    Boknik P, Grote-Wessels S, Barteska G, Jiang M, Müller FU, Schmitz W, Neumann J, Birnbaumer L.
    Br J Pharmacol; 2009 Nov 10; 158(6):1557-64. PubMed ID: 19906118
    [Abstract] [Full Text] [Related]

  • 5. Neuregulins regulate cardiac parasympathetic activity: muscarinic modulation of beta-adrenergic activity in myocytes from mice with neuregulin-1 gene deletion.
    Okoshi K, Nakayama M, Yan X, Okoshi MP, Schuldt AJ, Marchionni MA, Lorell BH.
    Circulation; 2004 Aug 10; 110(6):713-7. PubMed ID: 15289373
    [Abstract] [Full Text] [Related]

  • 6. Intracellular beta-blockade: overexpression of Galpha(i2) depresses the beta-adrenergic response in intact myocardium.
    Janssen PM, Schillinger W, Donahue JK, Zeitz O, Emami S, Lehnart SE, Weil J, Eschenhagen T, Hasenfuss G, Prestle J.
    Cardiovasc Res; 2002 Aug 01; 55(2):300-8. PubMed ID: 12123769
    [Abstract] [Full Text] [Related]

  • 7. Gαi2- and Gαi3-specific regulation of voltage-dependent L-type calcium channels in cardiomyocytes.
    Dizayee S, Kaestner S, Kuck F, Hein P, Klein C, Piekorz RP, Meszaros J, Matthes J, Bjrnbaumer L, Nürnberg B, Herzig S.
    PLoS One; 2011 Aug 01; 6(9):e24979. PubMed ID: 21966394
    [Abstract] [Full Text] [Related]

  • 8. Coupling of beta2-adrenoceptor to Gi proteins and its physiological relevance in murine cardiac myocytes.
    Xiao RP, Avdonin P, Zhou YY, Cheng H, Akhter SA, Eschenhagen T, Lefkowitz RJ, Koch WJ, Lakatta EG.
    Circ Res; 2011 Aug 01; 84(1):43-52. PubMed ID: 9915773
    [Abstract] [Full Text] [Related]

  • 9. Targeted inactivation of Galpha(i) does not alter cardiac function or beta-adrenergic sensitivity.
    Jain M, Lim CC, Nagata K, Davis VM, Milstone DS, Liao R, Mortensen RM.
    Am J Physiol Heart Circ Physiol; 2001 Feb 01; 280(2):H569-75. PubMed ID: 11158953
    [Abstract] [Full Text] [Related]

  • 10. Murine ventricular L-type Ca(2+) current is enhanced by zinterol via beta(1)-adrenoceptors, and is reduced in TG4 mice overexpressing the human beta(2)-adrenoceptor.
    Heubach JF, Graf EM, Molenaar P, Jäger A, Schröder F, Herzig S, Harding SE, Ravens U.
    Br J Pharmacol; 2001 May 01; 133(1):73-82. PubMed ID: 11325796
    [Abstract] [Full Text] [Related]

  • 11. Galphai1 and Galphai3 differentially interact with, and regulate, the G protein-activated K+ channel.
    Ivanina T, Varon D, Peleg S, Rishal I, Porozov Y, Dessauer CW, Keren-Raifman T, Dascal N.
    J Biol Chem; 2004 Apr 23; 279(17):17260-8. PubMed ID: 14963032
    [Abstract] [Full Text] [Related]

  • 12. Lack of effect of McN-A-343 on membrane current and contraction in guinea pig ventricular myocytes.
    Shen JB, Jiang B, Pappano AJ.
    J Pharmacol Exp Ther; 1999 Aug 23; 290(2):641-8. PubMed ID: 10411573
    [Abstract] [Full Text] [Related]

  • 13. Transregulation of adenylyl-cyclase-coupled inhibitory receptors in heart failure enhances anti-adrenergic effects on adult rat cardiomyocytes.
    Borst MM, Szalai P, Herzog N, Kübler W, Strasser RH.
    Cardiovasc Res; 1999 Oct 23; 44(1):113-20. PubMed ID: 10615395
    [Abstract] [Full Text] [Related]

  • 14. Overexpression of wild-type Galpha(i)-2 suppresses beta-adrenergic signaling in cardiac myocytes.
    Rau T, Nose M, Remmers U, Weil J, Weissmüller A, Davia K, Harding S, Peppel K, Koch WJ, Eschenhagen T.
    FASEB J; 2003 Mar 23; 17(3):523-5. PubMed ID: 12631586
    [Abstract] [Full Text] [Related]

  • 15. Altered beta-adrenergic signal transduction in nonfailing hypertrophied myocytes from Dahl salt-sensitive rats.
    Nagata K, Communal C, Lim CC, Jain M, Suter TM, Eberli FR, Satoh N, Colucci WS, Apstein CS, Liao R.
    Am J Physiol Heart Circ Physiol; 2000 Nov 23; 279(5):H2502-8. PubMed ID: 11045988
    [Abstract] [Full Text] [Related]

  • 16. The regulator of G protein signaling RGS4 selectively enhances alpha 2A-adreoreceptor stimulation of the GTPase activity of Go1alpha and Gi2alpha.
    Cavalli A, Druey KM, Milligan G.
    J Biol Chem; 2000 Aug 04; 275(31):23693-9. PubMed ID: 10807934
    [Abstract] [Full Text] [Related]

  • 17. The functional activity of inhibitory G protein (G(i)) is not increased in failing heart ventricle.
    Hussain RI, Aronsen JM, Afzal F, Sjaastad I, Osnes JB, Skomedal T, Levy FO, Krobert KA.
    J Mol Cell Cardiol; 2013 Mar 04; 56():129-38. PubMed ID: 23220156
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  • 19. Dopamine-D2S receptor inhibition of calcium influx, adenylyl cyclase, and mitogen-activated protein kinase in pituitary cells: distinct Galpha and Gbetagamma requirements.
    Banihashemi B, Albert PR.
    Mol Endocrinol; 2002 Oct 04; 16(10):2393-404. PubMed ID: 12351703
    [Abstract] [Full Text] [Related]

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