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Journal Abstract Search

185 related items for PubMed ID: 11108261

  • 1. A role for Akt in mediating the estrogenic functions of epidermal growth factor and insulin-like growth factor I.
    Martin MB, Franke TF, Stoica GE, Chambon P, Katzenellenbogen BS, Stoica BA, McLemore MS, Olivo SE, Stoica A.
    Endocrinology; 2000 Dec; 141(12):4503-11. PubMed ID: 11108261
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  • 2. Effect of estradiol on estrogen receptor-alpha gene expression and activity can be modulated by the ErbB2/PI 3-K/Akt pathway.
    Stoica GE, Franke TF, Moroni M, Mueller S, Morgan E, Iann MC, Winder AD, Reiter R, Wellstein A, Martin MB, Stoica A.
    Oncogene; 2003 Sep 11; 22(39):7998-8011. PubMed ID: 12970748
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  • 4. Heregulin-beta1 regulates the estrogen receptor-alpha gene expression and activity via the ErbB2/PI 3-K/Akt pathway.
    Stoica GE, Franke TF, Wellstein A, Morgan E, Czubayko F, List HJ, Reiter R, Martin MB, Stoica A.
    Oncogene; 2003 Apr 10; 22(14):2073-87. PubMed ID: 12687010
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  • 5. Insulin-like growth factor-I inhibits progesterone receptor expression in breast cancer cells via the phosphatidylinositol 3-kinase/Akt/mammalian target of rapamycin pathway: progesterone receptor as a potential indicator of growth factor activity in breast cancer.
    Cui X, Zhang P, Deng W, Oesterreich S, Lu Y, Mills GB, Lee AV.
    Mol Endocrinol; 2003 Apr 10; 17(4):575-88. PubMed ID: 12554765
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  • 7. Role of insulin-like growth factor-I in regulating estrogen receptor-alpha gene expression.
    Stoica A, Saceda M, Fakhro A, Joyner M, Martin MB.
    J Cell Biochem; 2000 Jan 10; 76(4):605-14. PubMed ID: 10653980
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  • 8. Antiestrogen ICI 182,780 decreases proliferation of insulin-like growth factor I (IGF-I)-treated MCF-7 cells without inhibiting IGF-I signaling.
    Varma H, Conrad SE.
    Cancer Res; 2002 Jul 15; 62(14):3985-91. PubMed ID: 12124331
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  • 11. Akt activation by estrogen in estrogen receptor-negative breast cancer cells.
    Tsai EM, Wang SC, Lee JN, Hung MC.
    Cancer Res; 2001 Dec 01; 61(23):8390-2. PubMed ID: 11731414
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  • 12. The role of growth factor receptor pathways in human breast cancer cells adapted to long-term estrogen deprivation.
    Sabnis GJ, Jelovac D, Long B, Brodie A.
    Cancer Res; 2005 May 01; 65(9):3903-10. PubMed ID: 15867390
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  • 13. Insulin growth factor-I and epidermal growth factor receptors recruit distinct upstream signaling molecules to enhance AKT activation in mammary epithelial cells.
    Fleming JM, Desury G, Polanco TA, Cohick WS.
    Endocrinology; 2006 Dec 01; 147(12):6027-35. PubMed ID: 16990343
    [Abstract] [Full Text] [Related]

  • 14. Phosphatidylinositol-3-OH Kinase (PI3K)/AKT2, activated in breast cancer, regulates and is induced by estrogen receptor alpha (ERalpha) via interaction between ERalpha and PI3K.
    Sun M, Paciga JE, Feldman RI, Yuan Z, Coppola D, Lu YY, Shelley SA, Nicosia SV, Cheng JQ.
    Cancer Res; 2001 Aug 15; 61(16):5985-91. PubMed ID: 11507039
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  • 15. Ligand-independent activation of steroid receptors.
    Flint AP, Sheldrick EL, Fisher PA.
    Domest Anim Endocrinol; 2002 Jul 15; 23(1-2):13-24. PubMed ID: 12142222
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  • 16. Follicle-stimulating hormone and insulin-like growth factor I synergistically induce up-regulation of cartilage link protein (Crtl1) via activation of phosphatidylinositol-dependent kinase/Akt in rat granulosa cells.
    Sun GW, Kobayashi H, Suzuki M, Kanayama N, Terao T.
    Endocrinology; 2003 Mar 15; 144(3):793-801. PubMed ID: 12586755
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  • 17. Phosphatidylinositol 3-kinase (PI-3K)/Akt but not PI-3K/p70 S6 kinase signaling mediates IGF-1-promoted lens epithelial cell survival.
    Chandrasekher G, Sailaja D.
    Invest Ophthalmol Vis Sci; 2004 Oct 15; 45(10):3577-88. PubMed ID: 15452065
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  • 18. Requirement of estrogen receptor-alpha in insulin-like growth factor-1 (IGF-1)-induced uterine responses and in vivo evidence for IGF-1/estrogen receptor cross-talk.
    Klotz DM, Hewitt SC, Ciana P, Raviscioni M, Lindzey JK, Foley J, Maggi A, DiAugustine RP, Korach KS.
    J Biol Chem; 2002 Mar 08; 277(10):8531-7. PubMed ID: 11751931
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  • 19. Epidermal growth factor induces WISP-2/CCN5 expression in estrogen receptor-alpha-positive breast tumor cells through multiple molecular cross-talks.
    Banerjee S, Sengupta K, Saxena NK, Dhar K, Banerjee SK.
    Mol Cancer Res; 2005 Mar 08; 3(3):151-62. PubMed ID: 15798095
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  • 20. Peptide growth factor cross-talk with the estrogen receptor requires the A/B domain and occurs independently of protein kinase C or estradiol.
    Ignar-Trowbridge DM, Pimentel M, Parker MG, McLachlan JA, Korach KS.
    Endocrinology; 1996 May 08; 137(5):1735-44. PubMed ID: 8612509
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