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Journal Abstract Search

424 related items for PubMed ID: 11126361

  • 1. A methylthioadenosine phosphorylase (MTAP) fusion transcript identifies a new gene on chromosome 9p21 that is frequently deleted in cancer.
    Schmid M, Sen M, Rosenbach MD, Carrera CJ, Friedman H, Carson DA.
    Oncogene; 2000 Nov 23; 19(50):5747-54. PubMed ID: 11126361
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  • 4. Methylthioadenosine phosphorylase gene deletions are common in osteosarcoma.
    García-Castellano JM, Villanueva A, Healey JH, Sowers R, Cordon-Cardo C, Huvos A, Bertino JR, Meyers P, Gorlick R.
    Clin Cancer Res; 2002 Mar 23; 8(3):782-7. PubMed ID: 11895909
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  • 5. CDKN2A, CDKN2B, and MTAP gene dosage permits precise characterization of mono- and bi-allelic 9p21 deletions in childhood acute lymphoblastic leukemia.
    Bertin R, Acquaviva C, Mirebeau D, Guidal-Giroux C, Vilmer E, Cavé H.
    Genes Chromosomes Cancer; 2003 May 23; 37(1):44-57. PubMed ID: 12661005
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  • 6. Hypermethylation-associated inactivation indicates a tumor suppressor role for p15INK4B.
    Herman JG, Jen J, Merlo A, Baylin SB.
    Cancer Res; 1996 Feb 15; 56(4):722-7. PubMed ID: 8631003
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  • 7. Homozygous deletions at chromosome 9p21 and mutation analysis of p16 and p15 in microdissected primary non-small cell lung cancers.
    Packenham JP, Taylor JA, White CM, Anna CH, Barrett JC, Devereux TR.
    Clin Cancer Res; 1995 Jul 15; 1(7):687-90. PubMed ID: 9816033
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  • 8. Association of rat p15INK4B/p16INK4 deletions with monosomy 5 in kidney epithelial cell lines but not primary renal tumors.
    Knapek DF, Serrano M, Beach D, Trono D, Walker CL.
    Cancer Res; 1995 Apr 15; 55(8):1607-12. PubMed ID: 7712460
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  • 9. Homozygous codeletion and differential decreased expression of p15INK4b, p16INK4a-alpha and p16INK4a-beta in mouse lung tumor cells.
    Herzog CR, Soloff EV, McDoniels AL, Tyson FL, Malkinson AM, Haugen-Strano A, Wiseman RW, Anderson MW, You M.
    Oncogene; 1996 Nov 07; 13(9):1885-91. PubMed ID: 8934534
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  • 10. Mechanism of inactivation of CDKN2 and MTS2 in non-small cell lung cancer and association with advanced stage.
    Nakagawa K, Conrad NK, Williams JP, Johnson BE, Kelley MJ.
    Oncogene; 1995 Nov 02; 11(9):1843-51. PubMed ID: 7478613
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  • 11. Genomic organization and mutation analysis of Hel-N1 in lung cancers with chromosome 9p21 deletions.
    Cairns P, Okami K, King P, Bonacum J, Ahrendt S, Wu L, Mao L, Jen J, Sidransky D.
    Cancer Res; 1997 Dec 01; 57(23):5356-9. PubMed ID: 9393760
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  • 12. Cloning and characterization of murine p16INK4a and p15INK4b genes.
    Quelle DE, Ashmun RA, Hannon GJ, Rehberger PA, Trono D, Richter KH, Walker C, Beach D, Sherr CJ, Serrano M.
    Oncogene; 1995 Aug 17; 11(4):635-45. PubMed ID: 7651726
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  • 13. Gene deletion chemoselectivity: codeletion of the genes for p16(INK4), methylthioadenosine phosphorylase, and the alpha- and beta-interferons in human pancreatic cell carcinoma lines and its implications for chemotherapy.
    Chen ZH, Zhang H, Savarese TM.
    Cancer Res; 1996 Mar 01; 56(5):1083-90. PubMed ID: 8640765
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  • 14. Codeletion of p15 and p16 genes in primary non-small cell lung carcinoma.
    Xiao S, Li D, Corson JM, Vijg J, Fletcher JA.
    Cancer Res; 1995 Jul 15; 55(14):2968-71. PubMed ID: 7606711
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  • 16. Loss of the p16INK4a and p15INK4b genes, as well as neighboring 9p21 markers, in sporadic melanoma.
    Flores JF, Walker GJ, Glendening JM, Haluska FG, Castresana JS, Rubio MP, Pastorfide GC, Boyer LA, Kao WH, Bulyk ML, Barnhill RL, Hayward NK, Housman DE, Fountain JW.
    Cancer Res; 1996 Nov 01; 56(21):5023-32. PubMed ID: 8895759
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  • 19. Analysis of CDKN2A, CDKN2B, CDKN2C, and cyclin Ds gene status in hepatoblastoma.
    Iolascon A, Giordani L, Moretti A, Basso G, Borriello A, Della Ragione F.
    Hepatology; 1998 Apr 01; 27(4):989-95. PubMed ID: 9537438
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  • 20. Codeletion of the genes for p16INK4, methylthioadenosine phosphorylase, interferon-alpha1, interferon-beta1, and other 9p21 markers in human malignant cell lines.
    Zhang H, Chen ZH, Savarese TM.
    Cancer Genet Cytogenet; 1996 Jan 01; 86(1):22-8. PubMed ID: 8616780
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