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754 related items for PubMed ID: 11163212

  • 1. The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues.
    Lindsten T, Ross AJ, King A, Zong WX, Rathmell JC, Shiels HA, Ulrich E, Waymire KG, Mahar P, Frauwirth K, Chen Y, Wei M, Eng VM, Adelman DM, Simon MC, Ma A, Golden JA, Evan G, Korsmeyer SJ, MacGregor GR, Thompson CB.
    Mol Cell; 2000 Dec; 6(6):1389-99. PubMed ID: 11163212
    [Abstract] [Full Text] [Related]

  • 2. Combined loss of proapoptotic genes Bak or Bax with Bim synergizes to cause defects in hematopoiesis and in thymocyte apoptosis.
    Hutcheson J, Scatizzi JC, Bickel E, Brown NJ, Bouillet P, Strasser A, Perlman H.
    J Exp Med; 2005 Jun 20; 201(12):1949-60. PubMed ID: 15967824
    [Abstract] [Full Text] [Related]

  • 3. Bcl-w forms complexes with Bax and Bak, and elevated ratios of Bax/Bcl-w and Bak/Bcl-w correspond to spermatogonial and spermatocyte apoptosis in the testis.
    Yan W, Samson M, Jégou B, Toppari J.
    Mol Endocrinol; 2000 May 20; 14(5):682-99. PubMed ID: 10809232
    [Abstract] [Full Text] [Related]

  • 4. Impact of the combined loss of BOK, BAX and BAK on the hematopoietic system is slightly more severe than compound loss of BAX and BAK.
    Ke F, Grabow S, Kelly GL, Lin A, O'Reilly LA, Strasser A.
    Cell Death Dis; 2015 Oct 22; 6(10):e1938. PubMed ID: 26492371
    [Abstract] [Full Text] [Related]

  • 5. Proapoptotic bcl-2 family members, Bax and Bak, are essential for developmental photoreceptor apoptosis.
    Hahn P, Lindsten T, Ying GS, Bennett J, Milam AH, Thompson CB, Dunaief JL.
    Invest Ophthalmol Vis Sci; 2003 Aug 22; 44(8):3598-605. PubMed ID: 12882813
    [Abstract] [Full Text] [Related]

  • 6. BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak.
    Zong WX, Lindsten T, Ross AJ, MacGregor GR, Thompson CB.
    Genes Dev; 2001 Jun 15; 15(12):1481-6. PubMed ID: 11410528
    [Abstract] [Full Text] [Related]

  • 7. An interconnected hierarchical model of cell death regulation by the BCL-2 family.
    Chen HC, Kanai M, Inoue-Yamauchi A, Tu HC, Huang Y, Ren D, Kim H, Takeda S, Reyna DE, Chan PM, Ganesan YT, Liao CP, Gavathiotis E, Hsieh JJ, Cheng EH.
    Nat Cell Biol; 2015 Oct 15; 17(10):1270-81. PubMed ID: 26344567
    [Abstract] [Full Text] [Related]

  • 8. Bax- and Bak-induced cell death in the fission yeast Schizosaccharomyces pombe.
    Jürgensmeier JM, Krajewski S, Armstrong RC, Wilson GM, Oltersdorf T, Fritz LC, Reed JC, Ottilie S.
    Mol Biol Cell; 1997 Feb 15; 8(2):325-39. PubMed ID: 9190211
    [Abstract] [Full Text] [Related]

  • 9. Bak but not Bax is essential for Bcl-xS-induced apoptosis.
    Lindenboim L, Kringel S, Braun T, Borner C, Stein R.
    Cell Death Differ; 2005 Jul 15; 12(7):713-23. PubMed ID: 15861188
    [Abstract] [Full Text] [Related]

  • 10. Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death.
    Wei MC, Zong WX, Cheng EH, Lindsten T, Panoutsakopoulou V, Ross AJ, Roth KA, MacGregor GR, Thompson CB, Korsmeyer SJ.
    Science; 2001 Apr 27; 292(5517):727-30. PubMed ID: 11326099
    [Abstract] [Full Text] [Related]

  • 11. Interferon-alpha-induced apoptosis in U266 cells is associated with activation of the proapoptotic Bcl-2 family members Bak and Bax.
    Panaretakis T, Pokrovskaja K, Shoshan MC, Grandér D.
    Oncogene; 2003 Jul 17; 22(29):4543-56. PubMed ID: 12881711
    [Abstract] [Full Text] [Related]

  • 12. Phenylarsine oxide induces apoptosis in Bax- and Bak-deficient cells through upregulation of Bim.
    Ni B, Ma Q, Li B, Zhao L, Liu Y, Zhu Y, Chen Q.
    Clin Cancer Res; 2012 Jan 01; 18(1):140-51. PubMed ID: 22048238
    [Abstract] [Full Text] [Related]

  • 13. Apoptosis initiated when BH3 ligands engage multiple Bcl-2 homologs, not Bax or Bak.
    Willis SN, Fletcher JI, Kaufmann T, van Delft MF, Chen L, Czabotar PE, Ierino H, Lee EF, Fairlie WD, Bouillet P, Strasser A, Kluck RM, Adams JM, Huang DC.
    Science; 2007 Feb 09; 315(5813):856-9. PubMed ID: 17289999
    [Abstract] [Full Text] [Related]

  • 14. Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release.
    Kandasamy K, Srinivasula SM, Alnemri ES, Thompson CB, Korsmeyer SJ, Bryant JL, Srivastava RK.
    Cancer Res; 2003 Apr 01; 63(7):1712-21. PubMed ID: 12670926
    [Abstract] [Full Text] [Related]

  • 15. Deficiency in Bak and Bax perturbs thymic selection and lymphoid homeostasis.
    Rathmell JC, Lindsten T, Zong WX, Cinalli RM, Thompson CB.
    Nat Immunol; 2002 Oct 01; 3(10):932-9. PubMed ID: 12244308
    [Abstract] [Full Text] [Related]

  • 16. BAX and BAK mediate p53-independent suppression of tumorigenesis.
    Degenhardt K, Chen G, Lindsten T, White E.
    Cancer Cell; 2002 Sep 01; 2(3):193-203. PubMed ID: 12242152
    [Abstract] [Full Text] [Related]

  • 17. Consequences of the combined loss of BOK and BAK or BOK and BAX.
    Ke F, Bouillet P, Kaufmann T, Strasser A, Kerr J, Voss AK.
    Cell Death Dis; 2013 Jun 06; 4(6):e650. PubMed ID: 23744350
    [Abstract] [Full Text] [Related]

  • 18. Mcl-1 promotes survival of thymocytes by inhibition of Bak in a pathway separate from Bcl-2.
    Dunkle A, Dzhagalov I, He YW.
    Cell Death Differ; 2010 Jun 06; 17(6):994-1002. PubMed ID: 20057504
    [Abstract] [Full Text] [Related]

  • 19. Embryogenesis and Adult Life in the Absence of Intrinsic Apoptosis Effectors BAX, BAK, and BOK.
    Ke FFS, Vanyai HK, Cowan AD, Delbridge ARD, Whitehead L, Grabow S, Czabotar PE, Voss AK, Strasser A.
    Cell; 2018 May 17; 173(5):1217-1230.e17. PubMed ID: 29775594
    [Abstract] [Full Text] [Related]

  • 20. The proapoptotic activities of Bax and Bak limit the size of the neural stem cell pool.
    Lindsten T, Golden JA, Zong WX, Minarcik J, Harris MH, Thompson CB.
    J Neurosci; 2003 Dec 03; 23(35):11112-9. PubMed ID: 14657169
    [Abstract] [Full Text] [Related]

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