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Journal Abstract Search

91 related items for PubMed ID: 11166710

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  • 2. Parvalbumin and calbindin D-28k immunoreactivity in transgenic mice with a G93A mutant SOD1 gene.
    Sasaki S, Warita H, Komori T, Murakami T, Abe K, Iwata M.
    Brain Res; 2006 Apr 14; 1083(1):196-203. PubMed ID: 16546142
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  • 3. Quantitative immunocytochemical analysis of the spinal cord in G86R superoxide dismutase transgenic mice: neurochemical correlates of selective vulnerability.
    Morrison BM, Gordon JW, Ripps ME, Morrison JH.
    J Comp Neurol; 1996 Sep 30; 373(4):619-31. PubMed ID: 8889947
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  • 6. Effects of the PPARgamma activator pioglitazone on p38 MAP kinase and IkappaBalpha in the spinal cord of a transgenic mouse model of amyotrophic lateral sclerosis.
    Shibata N, Kawaguchi-Niida M, Yamamoto T, Toi S, Hirano A, Kobayashi M.
    Neuropathology; 2008 Aug 30; 28(4):387-98. PubMed ID: 18312546
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  • 9. Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.
    Li QX, Mok SS, Laughton KM, McLean CA, Volitakis I, Cherny RA, Cheung NS, White AR, Masters CL.
    Aging Cell; 2006 Apr 30; 5(2):153-65. PubMed ID: 16626394
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  • 13. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.
    Neurobiol Dis; 2014 Apr 30; 64():48-59. PubMed ID: 24361555
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  • 16. Redox proteomics analysis of oxidatively modified proteins in G93A-SOD1 transgenic mice--a model of familial amyotrophic lateral sclerosis.
    Poon HF, Hensley K, Thongboonkerd V, Merchant ML, Lynn BC, Pierce WM, Klein JB, Calabrese V, Butterfield DA.
    Free Radic Biol Med; 2005 Aug 15; 39(4):453-62. PubMed ID: 16043017
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  • 17. Immunohistochemical study on the distribution of glycogen synthase kinase 3alpha in the central nervous system of SOD1(G93A) transgenic mice.
    Chung YH, Joo KM, Kim DJ, Kim SS, Kim KY, Lee WB, Cha CI.
    Neurol Res; 2008 Nov 15; 30(9):926-31. PubMed ID: 18671898
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  • 18. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 15; 7(6 Pt B):623-43. PubMed ID: 11114261
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  • 19. Progressive loss of a glial potassium channel (KCNJ10) in the spinal cord of the SOD1 (G93A) transgenic mouse model of amyotrophic lateral sclerosis.
    Kaiser M, Maletzki I, Hülsmann S, Holtmann B, Schulz-Schaeffer W, Kirchhoff F, Bähr M, Neusch C.
    J Neurochem; 2006 Nov 15; 99(3):900-12. PubMed ID: 16925593
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  • 20. Calbindin immunoreactivity in the neurons of the spinal cord and dorsal root ganglion of the domestic fowl.
    Lunam CA.
    Cell Tissue Res; 1989 Jul 15; 257(1):149-53. PubMed ID: 2752404
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