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Journal Abstract Search
844 related items for PubMed ID: 11207304
1. Fas/Fas ligand pathway, apoptosis, and clonal anergy involved in systemic acetylcholine receptor T cell epitope tolerance. Deng C, Goluszko E, Christadoss P. J Immunol; 2001 Mar 01; 166(5):3458-67. PubMed ID: 11207304 [Abstract] [Full Text] [Related]
2. B7-1 costimulatory molecule is critical for the development of experimental autoimmune myasthenia gravis. Poussin MA, Tüzün E, Goluszko E, Scott BG, Yang H, Franco JU, Christadoss P. J Immunol; 2003 Apr 15; 170(8):4389-96. PubMed ID: 12682276 [Abstract] [Full Text] [Related]
3. Impaired Fas signaling pathway is involved in defective T cell apoptosis in autoimmune murine arthritis. Zhang J, Bárdos T, Mikecz K, Finnegan A, Glant TT. J Immunol; 2001 Apr 15; 166(8):4981-6. PubMed ID: 11290777 [Abstract] [Full Text] [Related]
4. Absence of IL-4 facilitates the development of chronic autoimmune myasthenia gravis in C57BL/6 mice. Ostlie N, Milani M, Wang W, Okita D, Conti-Fine BM. J Immunol; 2003 Jan 01; 170(1):604-12. PubMed ID: 12496449 [Abstract] [Full Text] [Related]
5. A dual altered peptide ligand down-regulates myasthenogenic T cell responses and reverses experimental autoimmune myasthenia gravis via up-regulation of Fas-FasL-mediated apoptosis. Aruna BV, Ben-David H, Sela M, Mozes E. Immunology; 2006 Jul 01; 118(3):413-24. PubMed ID: 16827902 [Abstract] [Full Text] [Related]
14. Costimulation-dependent modulation of experimental autoimmune encephalomyelitis by ligand stimulation of V alpha 14 NK T cells. Pál E, Tabira T, Kawano T, Taniguchi M, Miyake S, Yamamura T. J Immunol; 2001 Jan 01; 166(1):662-8. PubMed ID: 11123351 [Abstract] [Full Text] [Related]
15. Genetic evidence for involvement of classical complement pathway in induction of experimental autoimmune myasthenia gravis. Tüzün E, Scott BG, Goluszko E, Higgs S, Christadoss P. J Immunol; 2003 Oct 01; 171(7):3847-54. PubMed ID: 14500686 [Abstract] [Full Text] [Related]
16. The susceptibility to experimental myasthenia gravis of STAT6-/- and STAT4-/- BALB/c mice suggests a pathogenic role of Th1 cells. Wang W, Ostlie NS, Conti-Fine BM, Milani M. J Immunol; 2004 Jan 01; 172(1):97-103. PubMed ID: 14688314 [Abstract] [Full Text] [Related]
17. Apoptosis by neglect of CD4+ Th cells in granulomas: a novel effector mechanism involved in the control of egg-induced immunopathology in murine schistosomiasis. Rutitzky LI, Mirkin GA, Stadecker MJ. J Immunol; 2003 Aug 15; 171(4):1859-67. PubMed ID: 12902487 [Abstract] [Full Text] [Related]
20. Suppression of experimental autoimmune myasthenia gravis by epitope-specific neonatal tolerance to synthetic region alpha 146-162 of acetylcholine receptor. Shenoy M, Oshima M, Atassi MZ, Christadoss P. Clin Immunol Immunopathol; 1993 Mar 15; 66(3):230-8. PubMed ID: 7679342 [Abstract] [Full Text] [Related] Page: [Next] [New Search]