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1190 related items for PubMed ID: 11223948

  • 1. Antisense targeting of c-fos transcripts inhibits serum- and TGF-beta 1-stimulated PAI-1 gene expression and directed motility in renal epithelial cells.
    Kutz SM, Providence KM, Higgins PJ.
    Cell Motil Cytoskeleton; 2001 Mar; 48(3):163-74. PubMed ID: 11223948
    [Abstract] [Full Text] [Related]

  • 2. Upstream stimulatory factor regulates E box-dependent PAI-1 transcription in human epidermal keratinocytes.
    Allen RR, Qi L, Higgins PJ.
    J Cell Physiol; 2005 Apr; 203(1):156-65. PubMed ID: 15372465
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  • 6. TGF-beta 1-induced PAI-1 expression is E box/USF-dependent and requires EGFR signaling.
    Kutz SM, Higgins CE, Samarakoon R, Higgins SP, Allen RR, Qi L, Higgins PJ.
    Exp Cell Res; 2006 Apr 15; 312(7):1093-105. PubMed ID: 16457817
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  • 8. Differential gene expression in migrating renal epithelial cells after wounding.
    Pawar S, Kartha S, Toback FG.
    J Cell Physiol; 1995 Dec 15; 165(3):556-65. PubMed ID: 7593235
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  • 10. Role of activator protein 1 transcriptional activity in the regulation of gene expression by transforming growth factor beta1 and bone morphogenetic protein 2 in ROS 17/2.8 osteoblast-like cells.
    Palcy S, Bolivar I, Goltzman D.
    J Bone Miner Res; 2000 Dec 15; 15(12):2352-61. PubMed ID: 11127200
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  • 11. Attenuation of plasminogen activator inhibitor type-1 promoter activity in serum-stimulated renal epithelial cells by a distal 5' flanking region.
    Slack JK, Higgins PJ.
    Cell Motil Cytoskeleton; 1999 Nov 15; 44(3):168-76. PubMed ID: 10542365
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  • 12. Induced PAI-1 mRNA expression and targeted protein accumulation are early G1 events in serum-stimulated rat kidney cells.
    Kutz SM, Nickey SA, White LA, Higgins PJ.
    J Cell Physiol; 1997 Jan 15; 170(1):8-18. PubMed ID: 9012780
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  • 13. A short region containing an AP-1 binding site is essential for transforming growth factor-beta-induced c-jun gene expression in osteoblastic cells.
    Hata S, Shimizu T, Fujimoto M.
    IUBMB Life; 2000 Mar 15; 49(3):229-34. PubMed ID: 10868915
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  • 14. PAI-1 gene expression is growth state-regulated in cultured human epidermal keratinocytes during progression to confluence and postwounding.
    Staiano-Coico L, Carano K, Allan VM, Steiner MG, Pagan-Charry I, Bailey BB, Babaar P, Rigas B, Higgins PJ.
    Exp Cell Res; 1996 Aug 25; 227(1):123-34. PubMed ID: 8806459
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  • 15. Expression of dominant negative Jun inhibits elevated AP-1 and NF-kappaB transactivation and suppresses anchorage independent growth of HPV immortalized human keratinocytes.
    Li JJ, Rhim JS, Schlegel R, Vousden KH, Colburn NH.
    Oncogene; 1998 May 28; 16(21):2711-21. PubMed ID: 9652737
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  • 16. Perturbation of the actin cytoskeleton induces PAI-1 gene expression in cultured epithelial cells independent of substrate anchorage.
    Providence KM, Kutz SM, Higgins PJ.
    Cell Motil Cytoskeleton; 1999 May 28; 42(3):218-29. PubMed ID: 10098935
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  • 17. Regulation of IL-1-induced gingival collagenase gene expression by activator protein-1 (c-Fos/c-Jun).
    Hamid QA, Reddy PJ, Tewari M, Uematsu S, Tuncay OC, Tewari DS.
    Cytokine; 2000 Nov 28; 12(11):1609-19. PubMed ID: 11052811
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  • 18. Transforming growth factor-beta negatively modulates proliferation and c-fos expression of the human endometrial adenocarcinoma cell line HEC-1-A.
    Bergman CA, Talavera F, Christman GM, Baker VV, Roberts JA, Menon KM.
    Gynecol Oncol; 1997 Apr 28; 65(1):63-8. PubMed ID: 9103392
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  • 19. Transcriptional regulation of plasminogen activator inhibitor-1 by transforming growth factor-beta, activin A and microphthalmia-associated transcription factor.
    Murakami M, Ikeda T, Saito T, Ogawa K, Nishino Y, Nakaya K, Funaba M.
    Cell Signal; 2006 Feb 28; 18(2):256-65. PubMed ID: 15961275
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  • 20. A c-fos/Estrogen receptor fusion protein promotes cell cycle progression and proliferation of human cancer cell lines.
    Crowe DL, Brown TN, Kim R, Smith SM, Lee MK.
    Mol Cell Biol Res Commun; 2000 Apr 28; 3(4):243-8. PubMed ID: 10891399
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