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Journal Abstract Search

239 related items for PubMed ID: 11252663

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  • 8. [Possible involvement of protein kinase C in the pathophysiology and treatment of bipolar disorder].
    Einat H, Chen G, Manji H.
    Harefuah; 2004 Jun; 143(6):420-5, 462. PubMed ID: 15524099
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  • 11. Chronic lithium treatment alters the excitatory/ inhibitory balance of synaptic networks and reduces mGluR5-PKC signalling in mouse cortical neurons.
    Khayachi A, Ase A, Liao C, Kamesh A, Kuhlmann N, Schorova L, Chaumette B, Dion P, Alda M, Séguéla P, Rouleau G, Milnerwood A.
    J Psychiatry Neurosci; 2021 Jun 02; 46(3):E402-E414. PubMed ID: 34077150
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  • 13. Glutamate receptors as targets of protein kinase C in the pathophysiology and treatment of animal models of mania.
    Szabo ST, Machado-Vieira R, Yuan P, Wang Y, Wei Y, Falke C, Cirelli C, Tononi G, Manji HK, Du J.
    Neuropharmacology; 2009 Jan 02; 56(1):47-55. PubMed ID: 18789340
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  • 15. Lithium and synaptic plasticity.
    Salinas PC, Hall AC.
    Bipolar Disord; 1999 Dec 02; 1(2):87-90. PubMed ID: 11252664
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  • 16. Platelet protein kinase C alpha levels in drug-free and lithium-treated subjects with bipolar disorder.
    Young LT, Wang JF, Woods CM, Robb JC.
    Neuropsychobiology; 1999 Dec 02; 40(2):63-6. PubMed ID: 10474058
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  • 18. Chronic lithium salt treatment reduces CRE/CREB-directed gene transcription and reverses its upregulation by chronic psychosocial stress in transgenic reporter gene mice.
    Böer U, Cierny I, Krause D, Heinrich A, Lin H, Mayr G, Hiemke C, Knepel W.
    Neuropsychopharmacology; 2008 Sep 02; 33(10):2407-15. PubMed ID: 18046304
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